细胞生成

Surfactant is produced by alveolar type II cells, stored intracellularly in organelles known as lamellar bodies, and secreted by exocytosis.

肺表面活性物质由肺泡Ⅱ型细胞生成，储存于细胞内的一些细胞器中，由胞外排泌形式分泌。

It publishes original research reports, reviews in the following areas: cell cycle regulation, cytokines, erythropoiesis, gene therapy, general hematopoiesis, granulopoiesis, hematological malignancies, immunobiology, immunotherapy, lymphopoiesis, megakaryocytopoiesis, microenvironment, monocyte development, molecular genetics, signal transduction, stem cell biology, and experimental as well as clinical stem cell transplantation.

该杂志刊载细胞周期调控、细胞因子、红细胞生成、基因疗法、全身血细胞生成、粒细胞生成、血液肿瘤、免疫生物、免疫疗法、淋巴细胞生成、巨核细胞生成、微环境、单细胞生成、分子遗传学、信号传导、干细胞生物学、实验与临床干细胞移植方面的原始论文及综述等。

But the role of TPO on megakaryocyte growth and development and the expression of TPO mRNA in human bone marrow are not well known.

为进一步了解TPO在巨核细胞—血小板生长发育过程中所起的作用，本文就TPO在体外培养过程中对巨核细胞生成的影响，对基质细胞以及血小板减少症患者骨髓中TPO mRNA的表达进行了研究探索。

Flow cytometry analysis revealed that NIT transiently transfected cells were arrested in G2 phase.

在HeLa细胞中大量表现NIT蛋白可以抑制细胞的生长，同时也可以抑制细胞生成聚落的能力。

Imatinib, a BCR-ABL tyrosine kinase inhibitor, is able to induce a complete cytogenetic response in more than 85% of patients with CML. However, the researchers note that patients with a complete cytogenic response tend to relapse when imatinib is discontinued, and less than 10% of patients achieve a molecular remission, defined by an undetectable residual disease using real-time quantitative–polymerase chain reaction.

Imatinib是一种BCR-ABL酪胺酸激酶抑制物，可以在85%以上的CML病患诱导完整的细胞生成反应；不过，研究者指出，完整细胞生成反应的病患倾向在停用imatinib时复发，不到10%的病患达到分子生物学上的缓解，其定义为以即时定量聚合酶连锁反应侦测不出残余疾病。

Imatinib, a BCR-ABL tyrosine kinase inhibitor, is able to induce a complete cytogenetic response in more than 85% of patients with CML. However, the researchers note that patients with a complete cytogenic response tend to relapse when imatinib is discontinued, and less than 10% of patients achieve a molecular remission, defined by an undetectable residual disease using real-time quantitative–polymerase chain reaction.

Imatinib是一种BCR-ABL酪胺酸激酶抑制物，可以在85%以上的CML病患诱导完整的细胞生成反应；不过，研究者指出，完整细胞生成反应的病患倾向在停用imatinib时復发，不到10%的病患达到分子生物学上的缓解，其定义为以即时定量聚合酶连锁反应侦测不出残餘疾病。

Studies were conducted on cultured normal and hyperfunctioning human thyroid cells in order to compare the effects of thyrotropinon the secretion of cAMP.

以bTSH刺激体外培养的正常和甲亢甲状腺细胞，通过测定细胞生成环磷腺苷的量，探讨bTSH对两种甲状腺细胞功能的影响。

2TNF-α in the concentration of 10-3-10u/ml significantly inhibited TSH-induced cAMP release by both normal and hyperfunctioning thyroid cells.

2在10~(-3)～10U/ml的浓度区间，TNF-α显著抑制TSH对正常和甲亢甲状腺细胞生成cAMP的刺激效应，10~3U/ml的TNF-α仅抑制TSH对正常甲状腺细胞的刺激作用，对甲亢甲状腺细胞无明显影响。

Analysis of patient's primary leukemia blasts revealed that those carrying the t(8;21) generating AML1/ETO, the most common acute myeloid leukemia-associated fusion protein, display low levels of a microRNA-223 (miR-223), a regulator of myelopoiesis.

对原发性白血病的胚细胞进行分析发现具有 t （8；21）能够产生 AML1/ETO 蛋白，而这种蛋白是急性髓性白血病相关融合蛋白中最常见的一种，这样的白血病胚细胞具有微小 RNA-223（ miR-223）的低表达， miR-223是骨髓细胞生成的调节因子。

Extracellular calcium is required for normal progression through the initial recognition-alignment phase of myogenesis. A net calcium influx into fusion-competent myoblasts is a requisite step in membrane fusion.

细胞与细胞之间识别和并列的初期阶段，胞外钙离子的内流是正常肌细胞生成过程所必不可少的，钙离子进入具有融合潜能的成肌细胞内，从而使细胞膜发生融合。

Breath, muscle contraction of the buttocks; arch body, as far as possible to hold his head, right leg straight towards the ceiling (peg-leg knee in order to avoid muscle tension).

呼气，收缩臀部肌肉；拱起身体，尽量抬起头来，右腿伸直朝向天花板（膝微屈，以避免肌肉紧张）。

The cost of moving grain food products was unchanged from May, but year over year are up 8%.

粮食产品的运输费用与5月份相比没有变化，但却比去年同期高8％。