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Methods KC and FE of tympanic membrane were cultured on the surface of CCM prepared by torrefaction, then the cells biological characteristics were detected by immunofluorescence stain of keratin and vimentin, and the cells proliferative activity were identified by immunofluorescence stain of PCNA.

方法热干法制备CCM,传代的大鼠鼓膜上皮KC和FB分别接种于培养板及CCM表面,通过角蛋白、细胞膜波形蛋白免疫荧光染色鉴定细胞特异性,通过细胞核增殖抗原(proliferating Cell nuclear antigen, PCNA)免疫荧光染色测定膜上细胞的增殖能力,通过细胞培养液上清的经脯氨酸测定评价对FB胶原分泌功能的影响。

During the wall thickening, the number of Golgi bodies increased apparently and cytoplasm was filled with Golgi vesicles. Polylamellate structure of the secondary wall appeard, Along with the further development of tracheary elements, the nucleous disappeared and organelles were decreased, while cortical microtubules were arranged neatly against the inner side of plasmalemma.

初生壁与质膜之间有间隔较规律的次生壁突起;中期,线粒体和高尔基体的数量明显增加,细胞质中几乎充满高尔基体及其囊泡,次生壁中微纤丝已成有序排列;晚期,细胞核消失,导管中央形成空腔,细胞器迅速减少,仅观察到少量高尔基体和线粒体,有大量周质微管沿细胞长轴分布,且在次生壁的周围有大量小泡附着,次生壁的分层结构清晰可见。

NF-κB and ERβare directly binding together in Jurkat nucleus.E_2 may affect the transcriptive activity of NF-κB via ERβand further affect the fate of Jurkat cell.

ERβ与NF-κB在Jurkat细胞核中存在直接相互作用,E_2可能通过ERβ影响了NF-κB的转录活性,进一步影响了Jurkat细胞的命运。

The changes of the structure of the spinotrapezius microvessel were observed by microscope under transillumination.

微泡空化介导外源大分子物质传输到组织间隙的外溢通道实质是血管内皮细胞的撕裂所致,撕裂发生在细胞核附近;这种外溢的通道可以是一过性产生,具有可修复性;红细胞可以通过小于自己直径的外溢通道。

Phosphorylated Stat5a and Stat5b form homo-or heterodimers and translocate to the nucleus where they activate genetic programs of cell proliferation and differentiation.

磷酸化的Stat5a和5b可形成同源和异源二聚体并进入细胞核激活下游基因引起细胞的增生和分化。

As we show in detail in Figure 29.31, the result of Wnt signalling is to increase the level of -catenin, which causes it to translocate to the nucleus.

另一类途径由激酶级联的活化构成。在级联中,一系列激酶连续地相互激活,最终导致细胞核里转录因子的磷酸化和活化。

We concluded that HA induces CD44 ICD release and the released ICD then translocate into nucleus to regulate gene expression in the breast tumor cells.

综合以上实验结果我们发现,在乳癌细胞MDA-MB-435s中,经由玻尿酸刺激之后,会促使CD44 ICD的产生,而被释放到细胞质的CD44 ICD会进而进入到细胞核内,调控基因的表现。

Once activated,these two transcription factors translocate into the nucleus for inducing expression of INF-β,thus resulting in initiation of innate immune response and subsequent regulation of acquired immune response.

激活转录因子IRF-3和NF-κB,使其进入细胞核内,诱导干扰素的表达,从而启动固有免疫反应和调节随后的获得性免疫反应,增强机体抵抗病毒的能力。

Otch can translocate into nucleus and regulates downstream genes transcription. So it is suggested that AICD may play a role like NICD.

ICD会进入细胞核并对细胞进行生理调控,因此一般推测AICD可能具有类似NICD之角色。

Immunocytochemical results showed that TrkC were located on the plasma membrane within 24 hours however, began to translocate into nucleus within 72 hours of the differentiation of NSCs.

神经干细胞分化24小时,TrkC定位于细胞膜表面;神经干细胞分化72小时,TrkC开始定位于细胞核

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Yang yinshu、Wang xiangsheng、Li decang,The first discovery of haemaphysalis conicinna.

1〕 杨银书,王祥生,李德昌。安徽省首次发现嗜群血蜱。

Chapter Three: Type classification of DE structure in Sino-Tibetan languages.

第三章汉藏语&的&字结构的类型划分。