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细胞核

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The LC flaps to concuss back track between the cell membrane and the cell nucleus is output source of the living creature memory.

神经元细胞膜与细胞核之间形成的 LC振荡回路是生物记忆产生的根源,神经元细胞群之间的电磁谐振是生物智能产生的基础。

The SPY gene is expressed constitutively in plants, and most of the SPY protein presents in nucleus.

SPY基因在植物中呈组成型表达,其蛋白主要出现在细胞核部位。

Stratum granulosum: outside the Spinous layer, made up of cells further differentiated to cuticle cells, so it would lose the nucleolus and turn to the enucleated startum lucidum and horny layer after completely cornified to horny layer.

颗粒层:位于棘层之外,是进一步向角质层细胞分化的细胞,因此它起着向角质层完全角化后,便失去细胞核,而转化成无核的透明层和角质层。

An amount increase ofPAI-3 andK10 was observed after theKCswere cultured for48 h in high calcium medium,butat72 h the exprssion ofPAI-3 decreased. The positive stain ofPAI-3 wasmainly localized in perinuclearmembrane in lowcalcium medium and in cytoplasm in high calcium medium. PAI-3 was incorporated into cornified envelope ofterminally differentiated keratinocytes.

结果 高Ca2+浓度下培养24 h后,分化标记物K10弱阳性表达, PAI-3呈阳性表达;培养48 h后, K10强阳性表达,而PAI-3的阳性表达明显增加, 72 h后,表达开始降低,在低Ca2+浓度下阳性染色主要位于细胞核膜周围而高Ca2+浓度下主要位于细胞质,同时, PAI-3存在终末分化KCs角质壳中。

At globular embryo stage, calcium concentrated on plasma membrane and certain areas of cytoplasm; at cotyledonary embryo stage, calcium presented on cell wall and intercellular space.

胚性愈伤组织细胞的ATP酶活性高,定位于细胞壁、胞间隙、质膜及细胞核,为物质的吸收和积累提供能量。

The type of cell death caused by ricin was related to the ricin concentration and time of incubation with HeLa cells. The typical apoptosis was induced by ricin 0.05μmol〓. The apoptotic cells mainly showed cell shrinkage, cytoplasmic membrane blebbing, the chromatin condensation and fragmentation, crescentic nuclear and membrane bound apoptotic bodies formation.

细胞死亡方式也与蓖麻毒素作用时间和浓度相关,0.05μmol.L〓的蓖麻毒素可引起HeLa细胞发生典型的细胞凋亡,主要特征是细胞体积缩小,胞浆起泡,细胞核染色质浓缩、凝集,新月状核和膜包裹核染色质的凋亡小体形成。

The most common architectural pattern was cribriform (73%), followed by fused/poorly defined glands (55%), cords/single cells (11%), and solid sheets (5%). Nuclear enlargement was observed in 45 of the 55 studied cases (82%). Prominent nucleoli were either absent or infrequent in 38 cases (69%).

癌细胞最常见的组织结构是筛状(73%),接下来是融合的/不成形的腺管(55%)、条索状/单个细胞状(11%)、实性巢团状(5%)。55例中有45例(82%)可见细胞核增大。38例(69%)见不到或罕见显著的核仁。

The levels of SOD and CAT significantly increased and MDA decreased in bifidobacterium group compared to radition damage group.Electronic microgragh of spine neurocyte and neurogliocyte showed that cytoplasm,cytoblast and plastiosome were significantly engorged ,and neurogliocyte apoptosised in earlier period. TLR2mRNA increased in C group compared to B group by RT-PCR. Cytochrome C in kidney increased in C group compared to B group, but bcl-2,NF-Кb decreased. Conclusions : Enteroclysm is better compared to intragastric administration.

双歧杆菌处理组肝组织中SOD和CAT水平明显高于放射损伤组(P.01),同时MDA明显低于放射损伤组(P.01);电镜结果显示:放射损伤组与双歧处理组的细胞浆、细胞核和线粒体均明显肿胀,双歧处理组同时具有早期凋亡现象;RT-PCR结果显示:双歧处理组TLR2mRNA的表达明显高于放射损伤组;免疫组化结果显示,双歧处理组Cytochrome C的表达明显优于放射损伤组,而NF-кBp65和bcl-2则相反。

Results: The expression of MDA-7 was positive in the cytochylema and cell membrane of tumor cells with the light microscopy.

结果:光镜下,MDA-7主要位于肿瘤细胞胞浆和胞膜上,为粗、细不一的棕黄色颗粒,细胞核无阳性反应着色。

It is well-known that for eucaryotic cells, DNA is located in the karyon and can not penetrate to the cytochylema and the tissue fluid because the cell membrane can act as barriers for DNA transportation. Therefore, the appearance of DNA in cell matrix and the serum can indicate that some diseases have taken place. A main reason for cancer occurrence is the mutation of DNA. It may cause the change of the sequence and expression of cancer suppressor genes.

真核细胞的DNA广泛的存在于细胞核内的染色体上,由于核膜的选择性通透作用,使DNA无法分散到其它细胞的亚器官,加之细胞膜的隔离效果,DNA更难扩散到细胞外基质中,因此细胞外基质中DNA含量的增加往往也会预示某些疾病的发生。

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。

There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.

双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。