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These results demonstrate that human third molar pad possesses neural crest-derived cells that represent multipotent stem/progenitor cells.

这些结果显示人第三磨牙根部组织垫保持有神经嵴源性细胞特点,表现出多能干细胞/祖细胞特性。

IL-1 is elevated in tissue homogenates of temporal lobe and in cerebrospinal fluid from patients with AD. In Alzheimer patients activated immunoreactive IL-1+microglia are increased sixfold over those presenting in age matched controls and the activated IL-1+ microglia are involved in the evolution of diffuse plaque to the diagnostic neuritic plaque. IL-1 is produced by astrocytes and microglia which are components of senile plaques and induced astroglia proliferation.

AD患者颞叶脑组织及脑脊液中发现IL-1含量增高,并且IL-1免疫反应阳性的小胶质细胞比同龄的对照组高6倍,IL-1免疫反应阳性的小胶质细胞与弥散性淀粉样沉淀斑块发展成为具有诊断意义的神经纤维斑块有关。

He angiogenesis and tumor growth of human neuroblastoma can also be effectively inhibited by TrkA gene.

rkA基因能降低NB细胞的致瘤性及抑制神经母细胞瘤的血管生成和肿瘤生长。

It is a Intracellular Signaling Mechanism as neurocirculatory activator, but also powerfully to activate Hypophysis including adenohypothysis and NeuroHypophysis,Neuroendocrine Cell as NeuroHormonal Regulator for rejuvenescence,and furthermore enable to prevent from Central Pain Syndrome,Psychiatric Disorder and Melancholia.http://neurosignals.

这是一个细胞内信号传递机制作为神经循环系统的启动,而且也有力地启动脑垂体包括腺垂体和神经垂体,神经内分泌细胞作为神经体液调节,返老还童,恢复活力rejuvenescence,而且还能够防止中枢性疼痛症候群、精神障碍和抑郁症

In this report, we analyzed the cellular and noncellular findings of bronchoalveolar lavage fluid in 18 cases with IPF, 12 patients with Sar and 9 healthy volunteers as conirols, using immunohistochemicat staining, we also have estimted collagen typeⅠ and type Ⅲ semiquantitatively in transbronchial lung biopsy specimens from 25 patients with IPF and 10 normal lung tissue. The aim of this investigation is to select the criteria for evaluating the disease actiity.

本文对18例IPF、12例Sar和9例正常对照者的支气管肺泡灌洗液中细胞成份和非细胞成份进行了检测和分析,同时对10例正常肺和25例IPF患者经支气管肺活检的肺组织标本,应用免疫组化方法、半定量剂检测其中Ⅰ型胶原和Ⅲ型胶原的含量结合临床治疗效果,对有助于疾病活动性判断的指标进行了筛选和进一步的评价。

The object of this study is to cancerous and normal cells intimacy of hematoporphyrin derivative for diagnosis and therapy cancer, fluorescence spectra of cancerous and normal cells are measured using a ultrashort pulses laser spectral technique and picosecond time-correlated single-photon counting system.

研究用于癌症诊断与治疗的光敏剂血卟啉(hematoporphyrin derivative, HPD)对人体癌细胞与正常细胞的亲和性,采用超短脉冲激光光谱技术和皮秒时间相关单光子计数系统,测量经血卟啉培养的活体癌细胞与正常细胞的荧光光谱,观测到癌细胞样品在创645nm处具有特有的光谱谱峰;测量不同浓度血卟啉荧光光谱,观测到随着血卟啉浓度的逐渐增大,在645nm处出现的光谱峰也随之显著增强。

But the normal reaction to ConA does not support this.

而对ConA的刺激反应正常的结果,并不支持所诱导产生的抑制性T细胞为非特异性的自然抑制细胞。

Will amino acid introduces in fighting tumor medicaments element, can raise its to be opposite of tumor cell alternative, thing of antidote of the fat dissolve sex that enhances medicaments, delay is right cellular noxiousness.

将氨基酸引入抗肿瘤药物分子中,可以提高其对肿瘤细胞的选择性,增强药物的脂溶性、缓解药物对细胞的毒性等。

Therefore, CRP could mediate E-LDL participating in the early stage of atherogenesis. CRP is an ancient conserved protein whose initial role as a pattern recognition molecule may have been to defend against bacterial infections, but whose present biological role appears quite complex. CRP is protective against a variety of bacterial infections and inflammatory stimuli as an opsonin. It is likely that the activity of CRP in humans is either pro- or anti-inflammatory is dependent on the context in which it is acting.

由于CRP的识别相可以结合被暴露出磷酸胆碱结构的E-LDL,形成E-LDL/CRP复合体,而内皮细胞表面能以高亲和力识别CRP的配体相,从而CRP-相性结构具有结合E-LDL,被内皮细胞捕获,引起脂质沉积和激活补体、触发炎症反应的可能性,这样可以解释早期动脉粥样硬化为何同时存在E-LDL、CRP、补体,而这三种物质在同时、同地出现绝非一个巧合。

On magnification, all tumors have sharp peripheral circumscription and all tumors were confined to the upper dermis,and tumor continuity with the epidermis was noted in some cases.Some basloid aggregations associated with a palisaded alignment of the cells at the periphery of the aggregations can be seen..

镜下可见肿瘤边界清楚,大多位于真皮浅层,部分与表皮相连,可见多数基底样细胞集合,周边细胞核成栅栏样排列,绕以明显的纤维性基质以及纤维母细胞,有时在结缔组织之间可见裂隙。

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