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The general rule I learned from my cytopathology fellowship is that if a thyroid neoplasm looks not perfect for papillary carcinoma, follicular neoplasm, or Hurthle cell neoplasm, think about medullary carcinoma.

我从细胞同行那学习的经验是这样的,如果一个甲状腺肿瘤不完全象乳头癌,不完全象滤泡性肿瘤,也不完全象嗜酸细胞性肿瘤,这个时候,可能就是髓样癌。

Anthor factor affecting the cellular immunity is the presence of soluble interleukin-2 receptor (sIL-2R) that will compete with the membrane-bound IL-2R and desensitize the immune response.

因为介白素十号(IL-10)有压抑细胞性免疫反应(例如杀手细胞及吞噬细胞),却提升体液性免疫反应的作用,而EB病毒晚期基因BCRF 1与介白素十号有相似的基因序列(又称vIL-10),也可能具有相似的功能。

Cases of the abnormal sex differentiated diseases in children were presented. Among them, there were 4 cases of Turner's syndrome, 1 Klinefelter's syndrome, 9 female pseudohermaphroditism, 2 male pseudohermaphroditism, and 4 true hermaphroditism.

本文报道小儿性分化异常疾病20例(包括先天性卵巢发育不全综合征4例、先天性睾丸发育不全综合征1例、女性假两性畸型9例、男性假两性畸型2例、真两性畸型4例),对上述病例进行了细胞染色体核型分析,同时作了口腔上皮细胞性染色质、中性粒细胞核鼓植体及尿17酮类固醇、17羟类固醇的检查。

ALCL was not uncommon in children. The differential diagnoses included malgnant histiocytosis, T zone or pleomorphic T-cell lymphoma, Hodgkins lymphoma, mycosis fungoides, and metastatic carcinoma.

小儿间变性大细胞性淋巴瘤并不少见,需与恶性组织细胞增生症、T区或多形T淋巴瘤、霍奇金淋巴瘤、蕈样霉菌病和转移性癌等鉴别。

alcl was not uncommon in children. the differential diagnoses included malgnant histiocytosis, t zone or pleomorphic t-cell lymphoma, hodgkins lymphoma, mycosis fungoides, and metastatic carcinoma. immunohistochemical study cd30, cd15, lca and ema were very helpful for diagnosis and differential diagnosis.

小儿间变性大细胞性淋巴瘤并不少见,需与恶性组织细胞增生症、t区或多形t淋巴瘤、霍奇金淋巴瘤、蕈样霉菌病和转移性癌等鉴别。cd30、cd15、lca和ema免疫酶标检查对诊断及鉴别诊断十分有用。

There was high similarity in Sphericity, elips degree and regular form factor among the nucleus of mantle cell-centrocyte-centroblastic-centroblastic lymphoma.

套细胞-中心母细胞-中心细胞-中心母细胞性淋巴瘤细胞的细胞核在圆球度、规化形状因子和椭圆度等参数上具有较大的相似性。

To learn more about treatments for sickle cell anemia and what causes sickle cell anemia , visit us.

若要了解更多关于治疗镰状细胞性贫血及是什么原因导致镰状细胞性贫血,请访问我们。

C/EBPβnuclear translocation was observed clearly in the 1-15 days silicotic tissue. There was a significant over-expression of C/EBPβin lung tissue of silica exposed rats as compared with that in the normal ones.

在染尘1-29天(以尘细胞灶和细胞性结节为主)和36-64天(以结节中成纤维细胞明显增生为主)的大鼠矽肺组织中,C/EBPβ表达的程度明显不同,差异有显著性。

That means Pho85 kinaseand calcineurin were involved in salt tolerance with an identical target protein or in 中科院上海生化与细胞所博士学位论文摘要 the same pathway. Inhibition of calcineurin decrease the YPH499, pho80? mutant,and pap1 (pcl7)? mutant Mn2+ tolerance but not that of pho85? mutant and thepho85? mutant was more sensitive to Mn2+ than YPH499, pho80? mutant, and pap1(pcl7)? mutant even with the addition of cyclosporin A. Therefore, the conclusioncould be drawn that PHO85 gene played a dominant role in Mn2+ homeostasisregulation in compare with calcineurin. As for Ca2+ tolerance, cyclosporin A canincrease the tolerance to Ca2+ of all the mutant mention above, that means Pho85kinase and calcineurin function antagonistically in regulation of Ca2+ homeostasis.In Bioinformatics, BRI3 gene is a novel gene without any function clue but givehigh conservation in mammalian.

我们通过钙调磷酸酶的特异抑制剂环孢菌素A研究了YPH499、+2+pho85缺失株、pho80缺失株、pap1缺失株在钙调磷酸酶失活后对Na、Mn、2+Ca金属离子敏感性的变化,结果显示Pho85蛋白激酶和钙调磷酸酶通过直接+或间接激活同一个靶蛋白或途径来增强细胞对Na的耐受;和钙调磷酸酶相比,2+PHO85的缺失对酵母细胞Mn耐受性的破坏是相对控制性的,钙调磷酸酶的失2+活不能进一步降低pho85缺失株对Mn的耐受能力;Pho85和钙调磷酸酶在对2+Ca的耐受调节中是相互拮抗的,钙调磷酸酶的失活能增加pho85缺失株和2+pho80缺失株的Ca耐受。i中科院上海生化与细胞所博士学位论文摘要对本实验室克隆的人新基因BRI3进行系统的生物信息学分析,发现它是一个在哺乳动物中保守的,但功能未知的基因。

This study is to further investigate the mechanisms of osteoclastic bone resorption induced by IL-1 with established osteoclasts culture and osteoclasts, bone marrow stromal cells co-culture system.

本文利用建立的破骨细胞培养和破骨细胞、骨髓基质细胞共培养体系,在体外进一步研究IL-1诱导破骨细胞性骨吸收的机制,旨在探讨细胞因子在骨质疏松症骨丢失发生中的作用。

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