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Surfactant is produced by alveolar type II cells, stored intracellularly in organelles known as lamellar bodies, and secreted by exocytosis.

肺表面活性物质由肺泡Ⅱ型细胞生成,储存于细胞内的一些细胞器中,由胞外排泌形式分泌。

PartⅠ/chapter 3 The viability of cultured cerebral endothelial cells was determined by measuring the release of intracellular enzyme lactate dehydrogenase by nonviable cells with increased permeability.

第一部分第三章乳酸脱氢酶(1actate dehydrogenase,LDH)是一种细胞内酶,当细胞失去活力膜通透性增加时即释放到细胞外。本文通过测定释放到培养液中的LDH的量来测定牛脑微血管内皮细胞的活力。

There are two types of maturation according to our observation.One is nucleocapsids obtained its tegument in the nucleus and enveloped from the inner nuclear membrane.Another is nucleocapsids entered cytoplasm through nuclear membrane,then obtained their tegument in the cytoplasm,enveloped from the plasma membrane,finally released by necrosis,exocytosis or other ways.

病毒成熟有两种方式:一为细胞核内核衣壳在核内获得皮层,通过核内膜获得囊膜成为成熟病毒;二为核内核衣壳通过内外核膜进入胞浆,核内和胞浆内的核衣壳在细胞浆中获得皮层,然后在各种质膜上获得囊膜,最后成熟病毒通过细胞破裂或其他方式释放到细胞外。

CD34 hematopoietic stem/progenitor cells reside in the bone marrow in close proximity to the endosteal bone surface, surrounded by osteoblasts, stromal cells, and various extracellular matrix molecules. We used a bioartificial matrix of fibrillar collagen I, the major matrix component of bone, as a scaffold for ex vivo expansion of HSCs.

CD34 的造血干细胞/祖细胞存在于接近骨内膜表面的骨髓中,由成骨细胞、间充质细胞以及各种细胞外基质分子所包绕,我们采用生物人工合成的纤维胶原I

CD34+ hematopoietic stem/progenitor cells reside in the bone marrow in close proximity to the endosteal bone surface, surrounded by osteoblasts, stromal cells, and arious extracellular matrix molecules. We used a bioartificial matrix of fibrillar collagen I, the major matrix component of bone, as a scaffold for ex io expansion of HSCs.

CD34+的造血干细胞/祖细胞存在于接近骨内膜表面的骨髓中,由成骨细胞、间充质细胞以及各种细胞外基质分子所包绕,我们采用生物人工合成的纤维胶原I

Multiple studies have confirmed that the apoptotic process of GCs is not only related to apoptotic genes, but triggered by cytokines related to reproduction and Caspases family. Cell adhesion and extra cellular matrix in follicles also play an important intraovarian role. Importantly, the reproductive hormones have strongly related to control ovarian GCs apoptosis.

现已证实,颗粒细胞凋亡不仅与凋亡相关基因有关,而且受许多细胞因子及Caspases家族的调控,也受细胞连接及细胞外基质的影响,更重要的是受到生殖激素首要地网络化控制。

Time constants do not increase but the weights of the fittings concentration de pendently enhance as the external Ba2+ concentration augments. So the inac tivati on becomes faster and faster as the external Ba2+ concentration increases. Time constants decrease and the weights of the fittings concentration dependently enh ance as the external Ba2+ concentration augments when external Ba2+ concentratio n (10 or 100 μmol/L) is higher. So the inactivation becomes faster and faster. It is showed that Ba2+ can go deeper in IRK1-T from the surface of the ch annel a s external Ba2+ concentration increases.

三级指数拟合表明:细胞外Ba2+低浓度(1和3 μmol/L)时,Ba2+与K+相互竞争同一结合位点,随着Ba2+浓度的增加,时间常数不增加但拟合的权数却浓度依赖性增加,所以失活过程随Ba2+浓度的增加越来越快;细胞外Ba 2+高浓度(10和100 μmol/L)时,时间常数随Ba2+浓度的增加而减少,拟合的权数却浓度依赖性减少,失活过程也越来越快,说明Ba2+作用位点由通道的表面部位进入通道更深的地方。

Time constants do not increase but the weights of the fittings concentration de pendently enhance as the external Ba2+ concentration augments. So the inac tivati on becomes faster and faster as the external Ba2+ concentration increases. Time constants decrease and the weights of the fittings concentration dependently enh ance as the external Ba2+ concentration augments when external Ba2+ concentratio n (10 or 100 μmol/L) is higher. So the inactivation becomes faster and faster. It is showed that Ba2+ can go deeper in IRK1-T from the surface of the ch annel a s external Ba2+ concentration increases. CONCLUSION: Two diffe rent mechanisms for external Ba2+ blocking exist.

三级指数拟合表明:细胞外Ba2+低浓度(1和3 μmol/L)时,Ba2+与K+相互竞争同一结合位点,随着Ba2+浓度的增加,时间常数不增加但拟合的权数却浓度依赖性增加,所以失活过程随Ba2+浓度的增加越来越快;细胞外Ba2+高浓度(10和100 μmol/L)时,时间常数随Ba2+浓度的增加而减少,拟合的权数却浓度依赖性减少,失活过程也越来越快,说明Ba2+作用位点由通道的表面部位进入通道更深的地方。

Time constants do not increase but the weights of the fittings concentration de pendently enhance as the external Ba2+ concentration augments. So the inac tivati on becomes faster and faster as the external Ba2+ concentration increases. Time constants decrease and the weights of the fittings concentration dependently enh ance as the external Ba2+ concentration augments when external Ba2+ concentratio n (10 or 100 μmol/L) is higher. So the inactivation becomes faster and faster. It is showed that Ba2+ can go deeper in IRK1-T from the surface of the ch annel a s external Ba2+ concentration increases. CONCLUSION: Two diffe rent mechanisms for external Ba2+ blocking exist.

三级指数拟合表明:情绪细胞外Ba2+低浓度(1和3 μmol/L)时,Ba2+与K+相互竞争同一结合位点,随着Ba2+浓度的增加,时间常数不增加但拟合的权数却浓度依赖性增加,所以失活过程随Ba2+浓度的增加越来越快;情绪细胞外Ba2+高浓度(10和100 μmol/L)时,时间常数随Ba2+浓度的增加而减少,拟合的权数却浓度依赖性减少,失活过程也越来越快,说明Ba2+作用位点由通道的表面部位进入通道更深的地方。

The therapeutic mechanism of the method of nourishing qi, activating blood, dispersing phlegm and promoting the collaterals was supposed as that it intervened the airway remodeling and alleviated the progress of airflow obstruction of COPD by suppressing the chemo taxis of cytokines, regulating the proliferation of fibroblast cell and smooth muscle cell and preventing the proliferation and plerosis of extracellular matrix.

推测益气活血化痰通络法防治COPD的机制可能为:通过抑制炎症细胞的趋化、影响细胞因子的活性、调节成纤维细胞和平滑肌细胞的增殖、阻止细胞外基质的增生修复,从而达到干预COPD患者气道结构重塑,减缓COPD气流受限进展的作用。

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However, as the name(read-only memory)implies, CD disks cannot be written onorchanged in any way.

然而,正如其名字所指出的那样,CD盘不能写,也不能用任何方式改变其内容。

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