细胞坏死

Although many of the secondary processes can be injurious, they appear to be primarily epiphenomena of the 'execution' phase of cell death.

虽然许多二期过程是有害的，它们似乎表现为细胞坏死极期的主要副现象。

The overexpression of HO-1 induced by CoPP can inhibit death of the myocardial cells and subsequently alleviate myocardial ischemia/reperfusion injury,whose major mechanism may be related with anti-oxygen free radicals.

CoPP诱导的HO-1过表达可以抑制心肌缺血再灌注损伤后的细胞坏死，从而减轻心肌的再灌注损伤，其主要机制与抗氧自由基有关。

The upregulation of HO1 expression induced by CoPP can inhibit the necrosis of the myocardial cells and subsequently alleviate the myocardial ischemia/reperfusion injury. One of its main mechanisms may be related with antioxygen free radicals.

CoPP诱导的HO1过表达可以抑制心肌缺血-再灌注损伤后的细胞坏死，从而减轻心肌的再灌注损伤，其主要机制与抗氧自由基有关。

Result HO-1 expression was upregulated following pretreatment of CoPP before reperfusion,resulting in both an increase in SOD activity and a decrease in MDA content and thus in a reduction in death of the myocardial cells after ischemia/reperfusion injury.

结果：再灌注前使用CoPP进行预处理，可以诱导HO-1蛋白的表达上调；HO-1蛋白表达上调可以减少缺血再灌注后的心肌细胞坏死，提高心肌组织中SOD含量并降低MDA的含量。

Severe nuclear damage indicated by pyknosis and karyolysis are customarily taken as evidence of cell necrosis.

表现为核浓缩和核溶解的严重核损伤通常被认为是细胞坏死的证据。

If the toxic effects of viral gene therapy can be overcome, Melton's work may provide a basis for treating certain types of diabetes, in which cells in the pancreas that produce insulin die and are not replaced.

如果能使病毒基因疗法不产生毒性效果，那么梅尔顿从事的工作可为治疗因分泌胰岛素的胰腺细胞坏死后没有得到替代的糖尿病打下基础。

In anticipation of achieving the above objectives, marine-derived actinomycetes, H2003 and Aspergillus terreus Thom have been screened by using this model, the EtOAC extracts of strain H2003 and Aspergillus terreus Thom showed bioactivities of inhibiting the cell cycle progression and cytoclasis of t...

并采用活性追踪分离方法对一株放线菌和一株真菌的代谢产物活性成分进行了初步研究采用减压硅胶柱层析、凝胶柱层析(Sephadex LH-20)、反相硅胶柱层析和反相制备高效液相等分离手段，从具有细胞坏死活性的放线菌H2003分离得到22个单体化合物。

Both staphylococcus aureus exotoxins and neutrophils can damage the RPEC by inducing necrosis, and their function had synergetic effect.

金黄色葡萄球菌外毒素对RPE细胞具有毒性作用，其机制主要以细胞坏死的方式进行。

Ultrastucture of EOS from modeling group is different from eutopic EM, Three weeks treatment with Danazol can make organelle altered and cell necrotized.

模型组EOS超微结构与在位子宫内膜有区别，丹那哩治疗3周可促使 EOS细胞器改变，细胞坏死。

The major concern during laser irradiation of teeth is the high potential of tissue overheating resulting high temperatures that can spread in toward the dentin,causing irreversible damage to cells presented in the pulp chamber and leading to cell death,so the related laser irradiation parameters should be controlled strictly.

激光辐照牙齿时所产生的温升容易引起牙髓细胞不可逆的损伤，导致牙髓细胞坏死，因此必须对相关的激光参数加以控制。建立了激光与牙齿相互作用的热传输模型，利用Crank-N icolson有限差分法数值模拟了不同Nd∶YAG激光参数照射下牙齿表面及内部的温度场的分布。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。