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细胞坏死

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RESULTS The TUNEL-positive cells were located in the transitional zone between the necrotic core and normal cortex.

结果 TUNEL阳性细胞位于坏死中心与正常皮层之间的半影区。

This type of infarction is marked by loss of neurons and neuroglial cells and the formation of a clear space at the center left.

左侧中央的明亮的坏死区神经元和神经胶质细胞消失。

If want to changed the stat, must be operatived decrease secondary injury, limited proliferation rate of neuroglial cell, promoted neuron regeneration,after spinal cord injury.

只有有效的降低脊髓损伤后的继发性损害、限制神经胶质细胞的增生速度、促经神经元的再生、才能防止脊髓坏死空洞和胶质瘢痕的形成。

Treatment temperatures are representative of those required for thermal necrosis of ingrowing nociceptor nerve fibers and disc cellularity alone, or with coagulation and restructuring of anular collagen in the high temperature case.

治疗温度代表那些需要热坏死处理的内生的伤害感受器神经纤维以及间盘细胞构成,或者在高温下造成胶原环的凝固和重构。

Treatment temperatures are representatie of those required for thermal necrosis of ingrowing nociceptor nere fibers and disc cellularity alone, or with coagulation and restructuring of anular collagen in the high temperature case.

治疗温度代表那些需要热坏死处理的内生的伤害感受器神经纤维以及间盘细胞构成,或者在高温下造成胶原环的凝固和重构。

OBJECTIVE: To study the interaction between tumor necrosis factor-α and osteoprotegerin on the apoptosis of osteoblastic cells.

目的:探讨肿瘤坏死因子α能否诱导成骨类细胞凋亡及骨保护素能否抑制其凋亡。

They were cultured for 48 hours,and the a mount of osteolytic factors such as TNF-a and IL-6 of each group was determined.

培养48h后,用ELISA测定细胞上清液中肿瘤坏死因子-a、白细胞介素-6含量。

Osteonecrosis is defined as cell death of the components of bone.

摘要骨头坏死的定义为骨头内的成份细胞死亡。

It is evident that the plerocercoids parasitize mainly in the subcutaneous tissues and muscles of mice, and mice experimentally infected with plerocercoids show different degree of histopathological changes.

小鼠感染裂头蚴后,多组织器官有裂头蚴寄生,其中以皮下肌肉处多见。裂头蚴寄生可引起组织不同程度的增生、变性、坏死,组织结构破坏及大量炎症细胞浸润等病理变化。

Many researches have shown that penumbral area is a valuable target of therapy in clinic. The cell death ocurred in penumbral area are is mainly due to apoptosis.

近年来,随着医学和分子生物学技术的迅猛发展,对缺血型脑损伤的病理生理过程的研究越来越深入,研究表明缺血性脑损伤后最具有临床治疗价值的是处于低灌注状态的半暗带,半暗带神经元的坏死以细胞凋亡为主。

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