细胞坏死

Necrosis of hepatocytes and focal inflammation in the liver parenchyma.

坏死的肝细胞和炎症的协调肝实质。

Objective To elucidate the role of interleukin 6 (IL 6) and tumor necrosis factor α in the pathogenesis of pemphigus, and to find out the relationship between the two cytokines and the activity of the disease.

目的探讨白介素-6(IL 6)、肿瘤坏死因子-α在天疱疮发病机理中的作用以及患者血清中这2种细胞因子水平与天疱疮病情的关系。

Objective To elucidate the role of interleukin-6 (IL-6) and tumor necrosis factor-α in the pathogenesis of pemphigus, and to find out the relationship between the two cytokines and the activity of the disease.

目的 探讨白介素－6(IL-6)、肿瘤坏死因子－α在天疱疮发病机理中的作用以及患者血清中这2种细胞因子水平与天疱疮病情的关系。

The data obtained in both groups were analyzed by using Student t test, product moment correlation, and multiple regression analysis.

结果：（1）HIFU组原发灶肿瘤组织出现凝固性坏死，未检测到凋亡的肿瘤细胞，而对照组为1.10±0.76/HPF。

So EA therapy could decrease the loss of neurons by restraining apoptosis and putrescence of brain neurons, accelerate the functional recover of damaging neurons and improve studying and memory ability.

电针治疗可以通过抑制神经元细胞凋亡和坏死两个途径减少皮质和海马神经元的丢失，促进受损神经元功能的恢复，改善VD模型大鼠的学习记忆障碍。

It can reduce the activity of amino transferase in blood serum alleviate the denaturalization of fat in liver cell , putrescence as well as inflammatory reaction .

，降低血清转氨酶活性，减轻肝细胞脂肪变性，坏死和炎性反应，促进白蛋白合

From occurrence and development mechanism, this article considers that the prevention and treatment for pyemia after severe empyrosis includes full body fluid anabiosis, early gastro-intestinal tract feeds, preventing intestinal tract endotoxin shifting, cutting the scab as soon as possible, eliminating necrosis, applying the antibiotic reasonably, immunity recuperation, maintaining the cellular immunity function, enhancing organism nutrition support, multi-organ function protection and improving organism prognosis.

本文通过对脓毒症的发生发展机制的阐述，认预防和治疗严重烧伤脓毒症全面的临床措施包括：快速、充分的体液复苏治疗和预防烧伤休克；早期胃肠道营养，以防止肠道内毒素移位；尽早切痂，清除坏死组织，封闭创面，合理应用抗生素，预防感染；免疫调理提高机体免疫能力，维护细胞免疫功能；加强机体营养支持治疗和多器官功能的保护，改善机体预后。

Objective To investigate the effect of tumor necrosis factor α on radiosensitivity of nasopharyngeal carcinoma in relation to TNFa-induced cell cycle synchronization.

目的探讨肿瘤坏死因子使细胞周期同步化对增加鼻咽癌的放射敏感性的影响。

AIM: To study the synergistic induction of apoptosis by TRAIL and cisplatin in rhabdomyosarcoma cells and investigate the role of mitochondrial membrane potential and caspase-3 in this process.

目的：探讨肿瘤坏死因子相关诱导凋亡配体蛋白和顺铂协同抑制横纹肌肉瘤细胞生长和诱导凋亡作用及其机制。

Our results indicate that the deficiency of dystrophin would lead to the reduction or deficiency of SG proteins, which would cause disruption/dysfunction of dystrophin-glycoprotein complex , loss of linkage between subsarcolemmal cytoskeleton and extracellular matrix which, in turn, would lead to sarcolemma instability and eventually to muscle cell necrosis.

表明dystrophin缺乏将导致包括SG在内的dystrophin相关蛋白的显著减少，DGC复合体结构破坏或功能障碍，细胞膜下骨架与细胞外基质连系的丧失，致使肌细胞膜不稳定，在肌肉收缩时容易受损，最终肌纤维变性、坏死。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。