细胞坏死

A virus strain was isolated from the arthrosis of broiler chickens.The virus was shown to have spherical particles with isometric symmetry,no envelope,double deck capsids,with outer capsid of 75nm,inner one of 50nm in diameter by electron microscopy.The virus was not sensitive to aether,acid and heat.The virus could multiply on the chick embryo fibroblastcell,and could cause syncytial pathologic change with the appearance of cytoplasm inclusion bodies.The challenged chickens of one-day old by nasal,oral,intramuscular and pulvillus injection displayed obvious clinical symptom of severe arthritis and accompanied with pathological changes of malabsorption and necrosis.Meanwhile,four avian reovirus isolates CL,S1133,YH and YB were examined in the cross-neutralization test.

从临床患病鸡关节病料中分离到一株病毒，在电镜下观察到病毒粒子无囊膜，有双层衣壳，外衣壳直径约75nm，内核约50nm；分离病毒株对酸、热有较强的抵抗力，对乙醚不敏感；病毒感染鸡胚成纤维细胞上能产生以融合细胞为特征的CPE；爪垫接种1日龄雏鸡表现出明显的病毒性关节炎临床症状，并伴有吸收障碍型和坏死型的病理变化；血清中和交叉试验表明，该病毒分离株与国内的2株番鸭源呼肠孤病毒分离株和国外S1133呈不同程度的交叉反应。

At 8 weeks, the liver in the experimental group developed purple, seems wrapping with millets. Pathological sections showed punctiform or lamellar cellular necrosis, inflammatory cell infiltration around liver canaliculus, displaying early symptoms of liver fibrosis. At 12 weeks, the liver looks gray, seems wrapping with many types of millet.

实验组8周时，肝脏呈暗紫色，表面有轻微粟粒样改变，病理切片示：肝细胞点状及点灶状坏死，汇管区炎症细胞浸润，呈早期纤维化症状。12周时，肝脏呈灰褐色，有较明显粟粒样改变。

The liver cell and renal tubular cell showed to dissolve putrescence.

肝细胞、肾小管细胞呈现溶解性坏死。

Reduce the glutamic-pyruvic transaminase in serum, lessen denaturalization and putrescence of liver cell and renew them, increase the contents of liver glycogen and r globin, and accelerate growth of liver cells, which has well protective effect to liver cells.

降低血清中谷丙转氨酶、减少和恢复肝细胞的变性和坏死，增加肝糖元、r球蛋白的含量，以及促进肝细胞的再生，对肝脏具有良好保护作用。

In addition intracellular potassium ions released through the incompetent sarcolemma of the dead muscle cells surge into the general circulation at this reperfusion stage and set the stage for sudden hyperkalaemic cardiac arrest.

另外，在再灌注时细胞内的钾离子从坏死肌细胞中经遭破坏的肌纤维膜进入体循环，导致这个阶段的心脏骤停。

Iron in the form of ferritin is highly toxic and may cause neural death and reactive gliosis. Astrocytes and microglia are responsible for uptake of excessive CSF iron in superficial siderosis.

星形胶质细胞和小胶质细胞可摄取脑脊液中过多的铁，而铁蛋白中铁的毒性又可导致神经元坏死和反应性神经胶质增生。

Results Microscopically,the tumor was composed of spindle-shaped cells and the cells were obviously atypical.M...

结果肿瘤主要由梭形细胞构成，细胞排列紧密，异型性明显，分裂相易见伴大片坏死及玻璃样变。

Results ZYC could increase the thickness of granular cell layer and decrease the thickness of theca cell layer, greatly decrease the level of T and LH in serum and the expression of TNF-α in rat ovary.

结果 助孕胶囊可使PCO大鼠的颗粒细胞层增厚，卵泡膜细胞层变薄，血清T、LH水平均明显下降，并下调卵巢组织肿瘤坏死因子α的表达。

We dyed 5 lung cancer specimens by TTC after they were treated by HIU, observing the pathological changes of dyed and undyed areas.

病理显示肿瘤细胞呈不可逆坏死，未着色区与治疗靶区一致，说明靶区的肿瘤细胞已丧失活性。

The light microscope observed four-layer structures of arthrodial cartilage, regularly ordered chondrocytes, even chromatin and clear nucleus in normal group. In the model group, the layer of arthrodial cartilage was thinning; chondrocytes in some regions showed karyopyknosis or necrosis with irregular order.

光镜下正常对照组关节软骨四层结构清晰可辨，软骨细胞排列整齐呈柱状，染色质分布均匀，细胞核清晰；模型对照组关节软骨层变薄，部分区域软骨细胞核固缩、坏死，软骨细胞排列紊乱，四层结构不易分辨。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。