细胞发生

There are 12 cases were death. ConclusionsPersisting hypoxemia always results in hyperpotassemia, cellular acidosis, increasing of blood consistence, heart overexertion as well as hepatocyte edema,degeneration even cytoclasis and so on.

死亡12例。结论持续存在的低氧血症可导致高血钾，细胞内酸中毒，血液粘稠度增加，心脏负荷增加，肝细胞水肿、变性甚至坏死等，严重的可以出现多脏器功能衰竭、DIC发生甚至死亡。

Ozone easily sets off a chemical reaction with the lipoprotein of the cytoderm in bacteria or sets off a chemical reaction with phospholipid, protein in the epicyte , so that the cytoderm of the bacteria and the cells are destroyed, and the permeability of the epicyte improvesm, the matter in the cells flows out and loses the activity.

臭氧很容易同细菌的细胞壁中的脂蛋白或细胞膜中的磷脂质、蛋白质发生化学反应，从而使细菌的细胞壁和细胞受到破坏，细胞膜的通透性增加，细胞内物质外流，使其失去活性。

CML is cytogenetically marked by the philadelphia chromosome, which originates from a reciprocal translocation between chromosome 9 and 22 and is molecularly marked a chimeric bcr-abl gene, resulting from juxta-positive of the abl proto-oncogene on chromosome 9 with the bcr gene, which is normally located on chromosome 22. The chimeric bcr-abl gene expression an 8. 5kb hybrid mRNA transcript giving rise to a 210-KD fusion protein (P210〓) with increased tyrosine kinase activity. P210〓 plays a key role in the pathogenesis of CML. The continuous cell line K562 was established from the pleural effusion of a 53-year-old female with CML in terminal blast crisis, and was a human erythroleukemia line, contained Ph chromosome.

绝大多数慢粒患者白血病细胞中具有Ph染色体，是由9号染色体长臂3区4带和22号染色体长臂1区1带相互易位形成，即t（9；22），使位于9q〓的c-abl原癌基因在第二外显子的5'端断裂并易位到22 q〓的M-bcr基因第2或第3外显子的3'端，形成异常的bcr-abl嵌合基因，该基因转导出异常的mRNA，编码并翻译出P210蛋白，该蛋白具有很强的酪氨酸激酶活性，使粒细胞发生恶性增殖。K562细胞属于慢粒急变、红白血病细胞株，具有Ph染色体。

Previously study shows that Bcl-xL deamidation is induced in response to cisplatin treatment in the absence of Rb, and Bcl-xL deamidatioon alters its antiapoptosis function.

之前的研究更显示在无Rb的细胞中当Bcl-xL 受到抗癌药物 cisplatin 刺激发生去胺基化后，会失去其原有抗细胞凋亡的功能。

However, at the beginning of PVR process, stimulated and activated by the initiative factors, RPE begin to dislocate from Bruchs membrane , migrate, proliferate, dedifferentiate, transdifferentiate and secrete provisinal extracellular matrix proteins, followed by membrane formation and contraction on both surfaces of the neuroretina and in the vitreous body, resulted in tractional retinal detachment.

PVR发生后，RPE受到始动因素的刺激，开始活化，脱离原位，去分化，移行、增生、发生表型转化等，并分泌细胞外基质(extracellular matrix， ECM)，最终在视网膜前后表面和玻璃体内形成具有收缩能力的增生膜，造成牵拉性视网膜脱离。

Therefore, corresponding changes in metabolism level are occurred during the form transition of the dimorphic cell in T.

因此，伴随银耳二型态细胞两相形态的转变，细胞代谢水平发生了相应变化。

Disconnective operation could weaken gastric mucosal cellular proliferative ability,this could be one of the rebleeding factors.CGRP showed a considerable protection for the gastric mucosa against postoperative rebleeding.

断流术削弱了胃粘膜细胞增殖能力，可能是易发生术后PHG胃出血的因素之一，CGRP改善了粘膜细胞增殖受抑状态，可保护胃粘膜，减少出血。

LX2 cells were transfected at the adenovirus tite pfu100, and were harvested at different times (24,48,72h), then western blotting was executed to detected the expression of ectogenous ampk.

对凋亡信号通路的进一步研究发现，Bcl-2在LX2中是低表达的，而Bax在AMPK过表达之后，表达增加，而用RNAi方法敲低Bax之后，细胞再用携带AMPK基因的腺病毒感染，LX2细胞不再发生凋亡，说明Bax参与了AMPK诱导LX2凋亡的信号通路。

Thus, in the process of chronic eczematous dermatitis, the apoptosis of keratinocyte is not only a turning point, but also a new target of drug screening.

因此，角质形成细胞的凋亡为湿疹性皮炎慢性化的转折点，阻断角质形成细胞的凋亡可以减轻湿疹慢性化进程的发生，从而为湿疹性皮炎的治疗提供了一条新的途径，成为药物筛选的一个新靶点。

Among all adhesion molecules sereted by endotheliocyte, intercellular adhesion molecule-1(ICAM-1) and vascular cell adhesion molecule-1(VCAM-1) have considerable effect on the occurrence and development of diabetic retinopathy.

在内皮细胞分泌的粘附分子中，细胞间粘附分子-1(intercellular adhesion molecule -1，ICAM-1)对糖尿病视网膜病变的发生和发展有着重要的作用。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。