细胞发生

The expression absence of LRRC4 was ascribed to the loss of homozygosity of 7q32-ter in U251 cell lines. Conclusion The expression of LRRC4 gene is absent in glioblastoma cell lines, and it offers the important experiment proof for LRRC4 to act as a new candidate of brain tumor suppressor gene from glioma.

通过对胶质瘤细胞系基因组DNA中LRRC4的ORF序列的PCR扩增和测序分析，发现SF126，SF767，M17细胞中，ORF序列第279位氨基酸的第3个密码子存在同义点突变(3/5)，而U251和U87细胞基因组DNA中发生了LRRC4基因的缺失突变(2/5)。

GFP expression started during gastrulation in the axial hypoblast layer. During segmentation, GFP was detected in the notochord but not in the foor plate.

GFP在原肠作用期就开始表达，主要发生在中轴下胚层中；在体节形成期，GFP在脊索细胞中表达，但未发现在神经底板细胞中表达。

These results revealed that overexpression of c-Myc upregulated telomerase, which in turn resulted in immortalization of laryngeal squamous cells, and this mechanism existed not only in the initiation of laryngeal carcinogenesis but also in the whole process of cancer development.

这些研究表明，喉癌的发生可能是由于c-Myc的过度表达使端粒酶表达上调，从而使喉鳞状上皮细胞达到永生化，这一机制不仅存在于喉癌发生的早期，而且贯穿于喉癌的发展过程。

The liver oval cell line WB-F344 derives from adult normal Fisher 344 male rats. It's a kind of liver infantine epithelial cells and has characteristics of liver stem cells. Many studies in vitro and in vivo imply that it has the role as precursor cells of hepatocacinoma, which indicates its function in hepatocarcinogenesis.

肝卵圆细胞株WB-F344来自正常成年同基因Fisher344雄性大鼠，是一种肝脏幼稚上皮细胞，具有肝脏干细胞的性质，体内、外的许多研究表明，它与肝癌的发生有一定的关系，具有肝癌前体细胞的作用。

The liver oval cell line WB-F344 derives from adult normal Fisher 344 malerats.It's a kind of liver infantine epithelial cells and has characteristics of liver stemcells.Many studies in vitro and in vivo imply that it has the role as precursor cells ofhepatocacinoma,which indicates its function in hepatocarcinogenesis.

肝卵圆细胞株WB-F344来自正常成年同基因Fisher344雄性大鼠，是一种肝脏幼稚上皮细胞，具有肝脏干细胞的性质，体内、外的许多研究表明，它与肝癌的发生有一定的关系，具有肝癌前体细胞的作用。

Membrane alterations (membrane vacuoles, invagination into adjacent organelles, and myelin figure-like changes) occur in a subpopulation of mitochondria in sepsis, but other hepatocyte organelles showed no consistent ultrastructural injury.

败血症中膜的改变（膜泡，向邻近的细胞内陷和髓磷脂形态样的变化）发生在线粒体的亚群，但是其他肝细胞器官未见一致的超微结构损伤。

These cell factors will be released to cytosol under stimulating factor by involucrum penetrability changes or involucrum breaking. This will render amplifying of apoptosis signals.

在凋亡刺激因素作用下，线粒体渗透性发生变化或外膜破裂，这些细胞因子将释放入胞浆内使凋亡信号得以放大，最终导致细胞凋亡。

Histopathological observation was characterized by lacrimal glands atrophy,cornea keratinization of the corneal epithelium,less of goblet cells from the conjunctival epithelium and so on.

VitA缺乏兔泪腺上皮细胞萎缩，角膜上皮角化，结膜杯状细胞减少，导致泪液分泌的质和量改变及泪膜稳定性降低而发生干眼症。

It reveals that the lignification of cells around growing points is an important reason of roots elongation inhibition.

结果显示小麦根尖生长点及侧根发生点均被高度木质化的细胞所包围，说明生长点周围细胞细胞壁木质化是铝抑制小麦幼苗根系伸长的一个重要原因。

Because the signaling molecules RAS and ERK1/2 (extracellular signal–regulated kinases 1 and 2) are activated by an LH surge in granulosa cells of preovulatory follicles, we disrupted Erk1/2 in mouse granulosa cells and provide in vivo evidence that these kinases are necessary for LH-induced oocyte resumption of meiosis, ovulation, and luteinization.

研究人员仍然在试图解析接着发生的那些导致排卵（或在某些情况下没有排卵）的分子信号级联反应。研究人员现在证明，激酶ERK1和ERK2是该激素信号的至关重要的下游标靶。在那些颗粒细胞中缺乏这些酶的雌性小鼠中，其卵母细胞不会成熟，因而不会从它们的滤泡中释放出来。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。