细胞原体

Objective: To explore the adhesiveness and pathogenicity of UU to rabbit epithelial cell of tubal mucosa.

目的：探讨解脲支原体对兔输卵管黏膜上皮细胞的粘附及其致病性。

Methods 90 patients suffered from M.pneumonia were administered and divi ded into three groups:mild group, severe group, and normal control group. T cell subset parameters, natural killer cell and the serumsoluble interleukin- 2 recepter of all of above were determined.

方 法对90例住院的支原体肺炎患儿，根据其X线肺部受侵程度，分为重症组和轻症组，并设立了对照组，分别检测了各组极期和恢复期的T细胞亚群、血清可溶性白细胞介素-2受体(SIL-2R)及天然杀伤细胞数。

Two anti-chicken, duck respiratory IgY (ND ++ Mycoplasma avian flu) and pathogens have unity, Conformational changes in the bacteria surface membrane electric field and launched lobulation of pathogenic cells and macrophage phagocytosis. the same time, pathogens in the body tissue adhesion, it is difficult to create lesions.

抗鸡、鸭呼吸道卵黄免疫球蛋白 IgY （禽流感+新城疫+支原体）与致病菌抱结后，改变了致病菌表面构象和细胞膜电场，发动分叶细胞和吞噬细胞对致病菌进行吞噬，同时组织致病菌黏附于肌体组织细胞，使其难以形成病灶。

Abstract］ Objective It is to observe the immunologic effect of spleen aminopeptide oral lyophilized powder on mycoplasma pneumoniae infection.Methods 78 case of MP infection were divided into two groups at random.The control group were treated with routine therapy.The treatment group were treated with spleen aminopeptide oral lyophilized powder based on routine therapy for 20 days.T cell subgroup,immunoglobulin and addiment were determined before and after treatment.

目的 观察脾氨肽口服冻干粉对小儿肺炎支原体感染的疗效及免疫学影响方法 78例MP感染患儿随机分两组，对照组38例予以常规治疗，观察组40例在常规治疗的基础上予以脾氨肽口服冻干粉治疗，治疗后分别测定患儿T细胞亚群、免疫球蛋白及补体等变化情况。

Many hES cell lines have been established under different conditions in the world. The firstly established hES cell lines were cultured on mouse embryonic fibroblast cells with medium containing many undefined animal components such as fetal bovine serum, which may cause cross-transfection with animal pathogen and mycoplasm.

目前，世界上已经建立了多株hES 细胞系，最早建立的hES 细胞系是生长在小鼠胚胎成纤维(mouse embryonic fibroblast， MEF)细胞上的，培养体系中含血清等动物源性成分，这些成分可能引起动物源性病原体或支原体的污染，从而限制了 hES 细胞的临床应用。

M. penetrans can stimulate the production of NO and inhibit mouse macrophages prolification, thus it may be an important etiological factor.

穿透支原体的脂质相关膜蛋白能诱导巨噬细胞产生一氧化氮，且能抑制巨噬细胞的增殖，因而可能与其致病性相关。

Results M. penetrans LAMPs stimulated mouse macrophages to produce nitric oxide in dose-and time-dependent manner. And M. penetrans LAMPs inhibited cell prolification in mouse macrophages.

结果 穿透支原体脂质相关膜蛋白能以时间和剂量依赖方式刺激小鼠巨噬细胞产生NO，并且对小鼠巨噬细胞的增殖有一定的抑制作用。

Methods M. penetrans was cultured and its LAMPs were extracted. Mouse macrophages were stimulated with M. penetrans LAMPs to analyze the production of nitric oxide. Cell prolification was detected in M. penetrans LAMPs-stimulated mouse macrophages by MTT assay.

用SP-4培养基培养穿透支原体并提取其脂质相关膜蛋白，用提取的脂质相关膜蛋白刺激小鼠巨噬细胞，以ELISA法检测NO的产生，并用MTT法检测其对小鼠巨噬细胞增殖的影响。

The embryogenic cell first divided into multicellular proembryos,then globular embryoid,pearshaped embryoid,long embryoid,scutiform embryoid and mature embryoid.

胚性细胞继续分裂形成多细胞原胚，经球形胚、梨形胚、长形胚、子叶分化期到成熟胚，其发生的胚状体与单子叶合子胚形成具有相似的过程。

Methods Th multi-epitope peptide was made up of 5 Th epitopes(heat shock protein 65 of Mycobacterium tuberculosis p1-20,E2 protein of Rubella virus p54-65,engineering epitope PADRE,heat shock protein 60 of Chlamydia trachomatis p35-48,and tetanus toxin p830-843),and its gene sequence including of 219bp.

方法复合T辅助细胞表位由5个T辅助细胞表位组成(结核杆菌热休克蛋白65p120、风疹蛋白E2-4p5465、人工合成T辅助细胞表位PADRE、沙眼衣原体热休克蛋白60p3548、破伤风类毒素p830843)，其基因序列由219个核苷酸组成。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。