细支气管的

The result showed that there was no expression in the normal control group and an apparent expression of ERK1/2 and Elk in the asthma group. A rather dense expressing of ERK1/2 was at bronchiole and mucous membrane, sub mucous membrane, smooth muscle, cytoplasm and nuclei of the out layer of the smooth muscle cell and an expression of positive fiber at submucous membrane.

结果发现正常肺内没有发现ERK1/2和Elk的表达，而哮喘时肺内有明显的ERK1/2和Elk表达，ERK1/2较密集表达在小支气管和细支气管的粘膜层、粘膜下层、平滑肌层和平滑肌外层细胞的胞浆和胞核中，也可见粘膜下层有阳性纤维表达。

Was presented in 12 cases (24%) by bacterial isolation. By in-situ hybridization, PCV2 signals were chiefly distributed in interstitial and necrotic lesions, and the positive signals could be found in macrophages-like cells and necrotic debris. PRRSV signals were mainly located at interstitial lesions and alveolar wall, and the positive signals could be found in macrophages-like cells and epithelial cells of alveoli. Swine influenza virus could be found in interstitial and necrotizing lesions, and the signals could be found in macrophages-like cells, and epithelial cells of terminal bronchiole and alveoli.

另运用原位杂交法对PCV2、PRRSV及猪流行性感冒病毒进行检测，结果发现PCV2主要分布於PNP的间质及坏死病灶区，并能於肺泡及终末细支气管上皮细胞、巨噬细胞与坏死细胞碎片皆可见有病毒核酸；PRRSV则多分布於间质病灶区及肺泡壁处，能於巨噬细胞与肺泡上皮细胞见到病毒核酸；SIV则存在於间质及坏死病灶区，且能於肺泡及终末细支气管上皮细胞与巨噬细胞见有病毒核酸。

Machine of concealed source sex turns a sex pneumonic is one kind a qualitative sex that names with clinical pathology is pneumonic, COP was 1985 Epler [the pathology performance that 1] offers is pulmonary alveolus inside, fine bronchus of sex of alveolar canal, breath and eventually the independent disease that end fine bronchus has granulation organization to form inside antrum is planted, say to send a gender especially again machine of fine bronchitic companion turns out-of-the-way sex the gender is pneumonic , will nearly 20 years report gradually grow in quantity, easy infection, n/med tuberculosis, tumour, as nodal as lung disease, other a disease of qualitative sex lung promiscuous, reached my courtyard to received vivid check of treated bronchus lens lung 2008 2007 now (8 case that TBLB) wins pathology to confirm are analysed as follows.

概要： 隐源性机化性肺炎是一类以临床病理命名的间质性肺炎，COP是1985年Epler等[1]提出的病理表现为肺泡内、肺泡管、呼吸性细支气管及终末细支气管腔内有肉芽组织形成的独立病种，又称特发性闭塞性细支气管炎伴机化性肺炎，近20年来报道逐渐增多，易和肺部感染、结核、肿瘤、结节病、其他间质性肺疾病等混淆，现将我院2007年至2008年收治的支气管镜肺活检获得病理证实的8例病例分析如下。

When the mice pre - sensitized were infected with RSV, the pulmonary inflammation, lymphocyte and eosinophils infiltration and cell - collar peribronchiles were more severe in lung organization and bronchiole than those in the single RSV infection group.

结果病理检查证实小鼠感染RSV后发展为典型的间质性肺炎；致敏小鼠感染RSV后病变更明显，细支气管周围淋巴细胞浸润形成管套增厚，嗜酸细胞增多，炎性细胞浸润的细支气管比例显著增高。

In the early stages of disease, the presence of peribronchiolar fibrosis can also result in an abnormal prominence of the centrilobular arteries, giving rise to an increased reticulation in the peripheral lung.

这里的peribrochiolar，首先是没有血管的字根，其次应该是细支气管，所以应该是"……细支气管周围……"

These findings indicate variable patterns of bronchiolar epithelial cytokine expression depending on the duration of CS exposure, and that complex mechanisms govern bronchiolar molecular dynamics in vivo.

此研究表明CS不同暴露时间导致细支气管上皮细胞因子表达的差异。这种复杂的机制支配着体内细支气管分子动力学。

In their Clinical Therapeutics article on inhaled insulin for diabetes mellitus, McMahon and Arky (Feb. 1 issue)1 report that short-term studies in animals have not shown a substantial effect on cell-proliferation indexes in the alveolar or bronchiolar areas of the lung.

他们发表的关于吸入胰岛素治疗糖尿病的临床治疗文章中，McMahon and Arky 1 报道说短期的动物实验显示对肺泡和肺细支气管的细胞增殖并没有显著影响。

On the other hand the inflammation of bronchiolar and block of the lung small vessels cause the disproportion of the ventilation and flow of blood and the disorder of the diffusion function,in turn the hyoxemia and the repiratory failure would occurrence.

此外，细支气管的炎变以及肺小血管的闭塞引起通气血流比例失调和弥散能力降低，最终发生低氧血症及呼吸衰竭。

In their Clinical Therapeutics article on inhaled insulin for diabetes mellitus, McMahon and Arky (Feb. 1 issue)1 report that short-term studies in animals hae not shown a substantial effect on cell-proliferation indexes in the aleolar or bronchiolar areas of the lung.

他们发表的关于吸入胰岛素治疗糖尿病的临床治疗文章中，McMahon and Arky 1 报道说短期的动物实验显示对肺泡和肺细支气管的细胞增殖并没有显著影响。

Inflammation per se may be responsible for mild airflow limitation.7 8 9 Indeed, it has been suggested that inflammation may lead to functional bronchiolar constriction by releasing mediators that may act directly on bronchiolar smooth muscle.10 The chronicity of inflammation would, in turn, produce other changes such as airways fibrosis, and may increase the smooth muscle mass either directly as a result of inflammation or indirectly as a result of chronically increased muscle tone.

炎症本身导致了气流限速轻微下降。而事实上它提示了由于释放的介质直接作用于细支气管平滑肌上，炎症引起了功能性细支气管的缢痕。慢性炎症反多来又会产生气道纤维化等其他改变，这样无论是炎症反应的直接影响结果，还是慢性肌紧张增高的直接影响结果，都会增加平滑肌质量。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。