细支气管

In their Clinical Therapeutics article on inhaled insulin for diabetes mellitus, McMahon and Arky (Feb. 1 issue)1 report that short-term studies in animals hae not shown a substantial effect on cell-proliferation indexes in the aleolar or bronchiolar areas of the lung.

他们发表的关于吸入胰岛素治疗糖尿病的临床治疗文章中，McMahon and Arky 1 报道说短期的动物实验显示对肺泡和肺细支气管的细胞增殖并没有显著影响。

Cardiac dyspnea results from edema in bronchiolar walls and stiffening of the lung due to parenchymal or alveolar edema, which interfere with airflow.

心源性呼吸困难是由细支气管壁水肿和肺实质或肺泡水肿所致的肺硬变引起，妨碍气流通行。

Cardiac dy ea results from edema in bronchiolar walls and stiffening of the lung due to parenchymal or alveolar edema, which interfere with airflow.

心源性呼吸困难是由细支气管壁水肿和肺实质或肺泡水肿所致的肺硬变引起，妨碍气流通行。

HRCT(high resolution computed tomography) at present has been the perfect technique of CT in the spatial and contrast resodution and the best imaging method of assessments of small sati solitary nodular lesions and bronchiolar diseases in the lungs.

高分辨率 CT目前是一种使空间和密度分辨率尽可能完善的 CT技术，是当今评价肺内小结节性病灶和细支气管疾病最理想的影像方法。

Dyspnea in the presence of bronchiolar edema is associated with wheezing due to airflow obstruction; frothy and sometimes blood-tinged sputum is expectorated.

细支气管水肿性呼吸困难与气流梗阻引起的喘息关，可咳出泡沫痰，有时则咳带血痰。

PS is mostly synthesized and secreted by alveolar typeⅡ cells and also by Clara cells in the peripheral airways to a lesser extent, and approximately 7% of alveolar surfactant reaches the bronchiolar tree via the mucociliary escalar.

PS主要由肺泡Ⅱ型上皮细胞产生，少部分由外周气道的Clara细胞产生，约7％的肺泡表面活性物质经纤毛运送到达细支气管。

Dy ea in the presence of bronchiolar edema is a ociated with wheezing due to airflow o tructio frothy and sometimes blood-tinged utum is expectorated.

细支气管水肿性呼吸困难与气流梗阻引起的喘息关，可咳出泡沫痰，有时则咳带血痰。

Whatever the situation, macrophages are elevated in the lungs of smokers and those patients with COPD, where they accumulate in the alveoli, bronchioli, and small airways.

无论什么情形，巨噬细胞在吸烟和COPD患者肺部都会增高，并会在肺泡，细支气管及小气道内积聚。

Eighteen (72%) chronic cases showed a pattern resembling usual interstitial pneumonia, but, in most cases, with more peribronchiolar fibrosis than one would expect in UIP.

慢性患者中有18例（72%）的组织结构类似寻常型间质性肺炎，但与UIP不同的是，绝大多数病例细支气管周纤维化病灶不只一处。

In others, the abnormal ground-glass opacities are centrilobular and peribronchiolar in location, resulting in the appearance of ill-defined centrilobular nodules.

在有的病人，整个小叶异常的密度增高，而邻近的小叶表现正常；而其他病人毛玻璃样阴影可位于小叶中心或细支气管血管周围，如同边界不清的小叶中心型结节。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。