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Results Compared to those before treatment the volume of the implanted keloid of Group D began to decrease since 14 days after treatment time-dependently (all P 《 0.05), and the volumes of the other 3 groups continued to increase and peaked on days 21, 14, or 7 respectively (all P 《 0.05).Microscopy showed infiltration of a larger quantity of histiocyte in the keloid tissue, and more obvious collagen disorganization and apoptosis of fibroblasts in Group D than in the other 3 groups.The protein expression of Bcl-2 was more remarkable and the protein expression of BAX was less remarkable in Group D than iu the other 3 groups.

结果 用药前和用药后7、14、21、28、35 d,D组瘢痕疙瘩组织块体积(mm3)分别为173±5、172±5、147±5、125±6、112±7和84±9,从用药后14 d开始明显缩小(均P<0.05);而其他3组瘢痕疙瘩组织块体积均明显增大,从用约后7 d开始各时点测得的体积均明显大于D组(均P<0.05)。D组瘢痕疙瘩组织中有大量小鼠组织细胞浸润,胶原结构破坏和成纤维细胞凋亡明显重于其他3组,Bcl-2蛋门质表达明显弱于而Bax蛋白质表达明显强于其他3组。

Objective The origin and the nature of the Hodgkin and Reed-Sternberg cell of Hodgkins lymphoma has been attracting a lot of medical researchers engaged in studying it,for its character by scattered large atypical cells residing in a complex admixture of inflammatory cells.Great improvement has been made since a new method of isolation of single H/RS cells from a frozen section had been set up by KUppers in 1994,and many studies have approved of the B-cell derivation of H/RS cell.lt has been reported that H/R-S cells might partly be originated from B-cell in our research before,but at the same time,we also found that only 18.8% of H/RS cells express CD20,31.3% of immunoglobulin heavy chain rearrangement have been revealed.

目的 霍奇金淋巴瘤(Hodgkins lymphoma,HL)由于它的恶性肿瘤细胞—H/RS细胞一般只占肿瘤组织的极少部分(不到1%),且散在分布在背景细胞间,因此对于H/RS细胞的来源和性质研究一直是人们探索的目标。1994年德国科隆大学Kūppers等发展了一种从冰冻组织切片上用显微操作仪挑取单个H/RS细胞的显微切割方法进行H/RS细胞基因分析后,人们对H/RS细胞来源的研究有了突破性的进展,多数支持B细胞来源。

They reported they were able to extract the stem cells without harm to mother or fetus and turn their discoery into seeral different tissue cell types, including brain, lier and bone.

此外,研究人员已成功利用干细胞培育出多种类型的组织细胞,包括脑组织细胞、肝组织细胞和骨组织细胞。

CT displayed the structure of femoral head was vague. A piece of coloboma could be observed clearly on the cartilage of a femoral head. Medullary canal was almost filled with white adipose tissue. Osteoblasts and osteocytes decreased. Empty bone lacuna increased. Thrombus distributed widely in the bone tissue microcirculation.

MA组病理检查肉眼可见股骨头软骨面局部缺损,坏死面暴露,髓腔内大量脂肪组织堆积;光镜下成骨细胞、骨细胞均减少,空骨陷窝增多,微小血栓广泛分布,变形扩大的脂肪细胞挤压正常骨髓组织细胞,骨小梁变细破裂明显。

Therefore, this study would calculate and survey the following indexes as the quantitative indexes for evaluating the degrees of cell senescence in hepatic and brain tissues, including numbers of neurons and dendrites in brain tissues; numbers of normal hepatocytes, dikaryotic hepatocytes, swollen hepatocytes as well as flake necrosis.

为此,本研究将计测脑组织神经元与树突数量;正常肝细胞,双核肝细胞、肿胀肝细胞数目、肝组织小片坏死个数作为评价脑神经及肝组织细胞衰老程度的量化指标。

The list of possible causes of diabetes insipidus in this case and many others is extensive and includes infection (tuberculous pachymeningitis and Whipple's disease), malignant neoplasms (germ-cell tumor and lymphoma), infiltrative processes (hemochromatosis and amyloidosis), autoimmune diseases (Wegener's granulomatosis, lymphocytic infundibulitis, and sarcoidosis), and histiocytosis (Langerhans'-cell histiocytosis and non-Langerhans'-cell histiocytosis).

这例病例其他许多病例尿崩症的可能病因广泛,包括感染(结核性脑脊髓膜炎和Whipple病),恶性肿瘤,变性疾病,自身免疫病(Wegener肉芽肿,垂体茎淋巴细胞瘤和粒细胞肉瘤)和组织细胞增生症(Langerhans细胞和非Langerhans细胞组织细胞增生症

In both inflammatory and healthy gingival epithelium, CD14 mRNA positive cells, which are mainly located at thornlike cell layer, were found.

另一方面,牙髓组织除树突状细胞外CD14表达细胞很少,牙龈组织皮下的成纤维细胞和未分化细胞也不表达CD14。

One hand mechanical obstruct led to the increase of veinous resistance and the obstacle of microcirculation, the other hand the adhesive PMN was activated in excess, the white blood cells released a lot of enzymes, in which PMN-elastase can decompose the components of cell and many albumens, inclusive of immunoglobulin、alexin and fibrication. These components induced the injury of the pancreatic capillary vessels and cell and lysosome enzy made the tissue protein hydrolyze and produced unsaturated fatty acids, which destroyed the structure and function of cellar membrane. The inflammatory cellar factors activate other immunocytes to produce the injury and necrosis of tissue, which aggravated the pathological injury and led to shock、pyaemia and MODS. So ICAM-1 and LFA-1 played an important role in SAP. Frossard found that the expression of ICAM-1 in the rat model, especially in serum、pancreas and lung. All these showed ICAM-1 is an important factor in AP and concomitant lung injury.

胰腺小叶组织局部血管EC首先被激活,ICAM-1表达升高,与被激活的PMN表面表达的LFA-1相结合,&PMN-EC&相互作用加剧,一方面机械性阻塞毛细血管导致静脉阻力增加、微循环障碍;另一方面粘附的PMN过度吞噬或激活,当白细胞吞噬的颗粒不能被封闭隔离,连同细胞内的酶被释放出来,其中的PMN-elastase能够降解细胞基质中各种成分,水解多种蛋白,加重胰腺的毛细血管内皮细胞和腺泡的损伤;释放的溶酶体酶使组织蛋白水解,产生的不饱和脂肪酸引发脂质过氧化方应,破坏细胞膜的结构和功能;释放的炎性细胞因子,激发其他的免疫细胞的功能,导致进一步的组织损伤和坏死,加重SAP的病理损伤,最终导致休克、脓毒血症及多器官功能障碍等严重后果。

Two different signals, whichwere brown and blue, reacted with the substrate (DAB and NBT/BCIP) separately, could be observed in the WSSV infected haemocyte. The result showed that in the lightly infected tissues infected haemocytes predominated in all infected cells. However in the heavily infected tissues the number of normal haemocyte decreased obviously and infected cell with strong positive intensity increased, and cell debris and tissue necrosis were observed.

应用这种双重免疫组化方法观察了被WSSV感染的血细胞在几种组织中的分布情况,发现在轻度感染组织中被侵染的血细胞在所有被感染细胞中占绝大多数,而在高度感染组中血细胞数目明显减少,细胞变形,出现许多的细胞碎片,同时被病毒感染的细胞数目增多,染色强度增强,组织坏死严重。

As for the transverse section of leaf,oil cells distribute in all mesophyll in some species, in the spongy parenchyma between palisade and spongy parenchyma in some species, and only in palisade parenchyma in some other species.

根据透明叶片观察,不同植物油细胞的分布密度不同;从叶片横切面观察,有些种油细胞分布于整个叶肉中,有的分布于栅栏组织与海绵组织之间及海绵组织中,有的仅分布于栅栏组织内。

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随着死亡的吉他手Schuldiner接受主唱的职务,乐队在现实中树立了重要的影响。

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