组织细胞的

Research of ultramicro- structure of anal sphincter in the cases of patients with fecal incontinence shows : shortness of smooth muscle cells of anal sphincter , destruction of normal structure of the rest cells , elongation of elastic tissue and increase of collagen fibers. Thus the most familiar cause of copracrasia is Degeneration of finespun smooth muscle of anal sphincter which mantain anal sphincter closed .One of the pathomechanism of fecal incontinence is decrease of anal resting pressure .

对大便失禁者肛门内括约肌的超微结构研究发现：肛门内括约肌的平滑肌细胞缺失、剩余细胞的正常关系破坏、弹性组织拉伸、胶原纤维成分增加，因而大便溢漏最常见的原因是维持括约肌关闭的肌肉-肛门内括约肌的纤细平滑肌的退行性病变，肛管静息压降低是大便失禁发病的一个病理学机制。

With the help of Axioplan 2 imaging system, we quantified the change of expression of ICAM-1, MUC5AC, goblet cell and NF-κB. Analyze the results by SAS method. Results (1) Grouped by index of lung function, the expression of ICAM-1 at the level of gene and protein had increased significantly in the epithelial cells of patients with COPD, compared with group of normal lung function.

取病灶远端的正常叶段支气管组织制备石蜡切片，原位杂交检测气道上皮中ICAM-1的mRNA的表达量；免疫组化检测气道上皮中ICAM-1和MUC5AC的蛋白表达变化；AB/PAS特殊染色法检测患者气道上皮细胞中杯状细胞的含量；PCR扩增法检测原代培养的气道上皮中MUC5AC的基因表达量和NF-κB的变化。

Although their role in formation and deposition of cellulose microfibrils had been proposed, they may also performed a function in the lignification.

另外，在茎的部分基本薄壁组织细胞和韧皮部等未木质化的细胞壁中，过氧化物酶也同样表现出较强的活性，这说明在茎的不同组织中分布的这种酶，可能是几种不同功能的同工酶形式。

Rat AAA model was made by perfusion PPE elastase through the abdominal aorta, and the specimen was obtained on postoperativeday 3, 7, 14, 28, respectively. Specific elastic fiber staining was used to observe the disruption of elastic fiber in aortic wall. Immunohistochemistry staining of CD68 was applied to observe the microphage infiltration. The in tisu hybridization and western blot of MCP-1 and MMP-2 were used to study the expression of mRNA and protein. Thus we can analyze the facilitated function of MCP-1 gene to the microphage adherence and infiltration on the earlier AAA.

采用经腹主动脉腔内导管灌注的方法制成大鼠AAA模型，分别于术后3天、1周、2周、3周、4周获取各组大鼠腹主动脉标本，行弹力纤维特殊染色观察动脉壁弹力纤维的破坏情况；行CD68免疫组织化学染色观察动脉壁中巨噬细胞的浸润；行MCP-1及MMP-2的原位分子杂交、Western蛋白质印迹观察二者的mRNA表达和蛋白表达；分析MCP-1基因促进巨噬细胞的黏附、浸润在早期AAA发病中的作用。

With the development of physiology,immunohistochemistry and application of electron microscope,researchers want to know the ultramicrostructure,3D ultramicrostructure and physiologic function of ICCs,to think highly of the effect of ICC,and try to interpret morphosis and physiologic function about it.

随着电镜的应用及生理学、免疫组织化学的发展，人们对Cajal间质细胞的超微结构、三维网络结构及生理功能有了进一步的认识，开始重视Cajal间质细胞的作用，试图阐明它们的形态结构和生理功能。

METHODS: According to adherent + Thy1.1 antibody and complement-purification method, cranium was opened to expose olfactory bulb. Thereafter, two olfactory bulbs were obtained to remove cerebral pia mater, blood capillary, and peripheral tissues; additionally, olfactory nerve layer and olfactory bulb granular layer were sheared into 1-mm3 pieces for extract single-cell suspension. The cells were adjusted at the density of 1×107 /L and incubated with poly-l-lysine-coated culture bottle or culture plate in 5% CO2 incubator at 37 ℃. On the third day, cells were cultured with serum-free DMEM/F12 culture media.

在差速贴壁+Thy1.1抗体及补体纯化法的基础上，剪开大鼠颅骨，显露位于颅腔前方的嗅球，取出2只嗅球，在显微镜下去除嗅球表面的软脑膜和毛细血管及外周组织，保留富含嗅鞘细胞的嗅神经层和嗅球颗粒层，剪成1 mm3小块分离获取单细胞悬浮液，调整细胞密度至1×107 L-1，接种在用poly-l-lysine包被的培养瓶或培养板中，于37 ℃、体积分数为5%的CO2培养箱中培养，第3天用无血清DMEM/F12培养基换液培养。

In a chapter on cancer, for instance, his description of how the cells operate contains this starling analogy: In the community of living tissues, the uncontrolled mob of misfits that is cancer behaves like a gang of perpetually wilding adolescents.

比如说，在关于癌症的那一章里，他对细胞的活动作了这样惊人的类比：在活细胞组织中，癌症就像是那些与社会格格不入的不受控制的暴民，他们的行为就像是一帮永远叛逆不羁的青少年。

When a strong and slowly degrading scaffold is used, the starting position of alular tissue engineering is a stronger ale and seeded cells are allowed more time to create a strong extracellular matrix.

当使用一个强壮的降解缓慢的支架时，瓣膜组织工程的起始点是一个更加强壮的瓣膜，种植的细胞有更多的时间创造强壮的细胞外基质。

Capsule of result antler glossy ganoderma can restrain the tumour of small rat of tumour of Lewis carry on one's shoulder to grow apparently, the elevatory, kidney of the Scr of serum of small rat of tumour of carry on one's shoulder that suitable platinum of remarkable changeover causes and BUN is coriaceous those who organize MDA content is elevatory, and SOD and GSH-Px reduce capsule of; antler glossy ganoderma actively poisonous to the cell of cell of tumour of different type person active has bigger difference, its IC50 sort is: HT-29

结果鹿角灵芝胶囊能明显抑制Lewis荷瘤小鼠的肿瘤生长，并显著逆转顺铂造成的荷瘤小鼠血清Scr和BUN的升高、肾皮质组织MDA含量的升高，以及SOD和GSH-Px活性的降低；鹿角灵芝胶囊对不同类型人肿瘤细胞的细胞毒活性有较大差异，其IC50排序为：HT-29

As mentioned earlier, elastinolysiss thought to be an essential component of emphysema.3 This is supported by both pathophysiologic and biochemical investigations, which show reduced lung elastic recoil, disrupted elastic tissue structure, higher circulating levels of elastin peptide breakdown products, and higher levels of desmosine in urine and lung secretions.3 Of the macrophage-derived MMPs identified in emphysematous and COPD tissue, the gelatinases MMP2 and MMP9 are elastinolytic.

正如早些时候提及的，elastinolysiss被认为是肺气肿的主要成份之一。这可以由病理生理研究与生化研究所证实，这两项研究同时提示肺弹性活力的降低，弹性组织结构的断裂，弹力蛋白肽分解产物循环水平的增高，以及尿与肺脏分泌物中锁链素水平的增高。在气肿型或COPD组织中，经确定的巨噬细胞源性的金属蛋白酶中，elastinoly的MMP2与 MMP9是elastinolytic。

Plunder melds and run with this jewel!

掠夺melds和运行与此宝石！

My dream is to be a crazy growing tree and extend at the edge between the city and the forest.

此刻，也许正是在通往天国的路上，我体验着这白色的晕旋。