组织细胞

M^2, p=0.46. The study showed no difference between the juvenile macromastic breast and non-tumor part of cancer breast in mammary epithelial cells, suggesting that the cause of juvenile macromastia may be from factors other than abnormal mammary epithelial cell size.

69.5±10.0μm^2， p=0.46。本研究发现青春期巨乳症的乳房组织与乳癌病患未受癌症感染之乳房组织，在上皮细胞之尺寸方面并没有差异，青春期巨乳症的成因可能是与乳房上皮细胞之生长较为相关。

The leaf in the Karst area was isolateral and had closely arranged mesophyllous cells.

在显微结构方面：岩溶区生长的扇叶铁线蕨的叶片具有旱生植物叶片特点，即叶片为等面叶，叶肉细胞排列较为紧密以及叶片维管组织发达等：而非岩溶区生长的扇叶铁线蕨的叶片为异面叶，特点为叶肉细胞排列相对疏松，维管组织不发达。

Under imitated daily field microclimatic conditions for heat stress, the rice cultivar "Jinyou63"(which is a hybrid rice combination) was treated with a consecutive 3-day heat stress during meiosis in phytotron. Anther and pollen tissues at different development stages after subjected to heat stress were sampled and analyzed for transverse abnormalities.

本研究模拟田间典型灾害天气条件，在人工气候箱内对杂交水稻&金优63&花粉母细胞减数分裂期进行连续3 d的热害胁迫处理，对处理后不同发育时期的花药组织取样切片观察，从组织、细胞水平探讨水稻受高温胁迫后花药可能出现的早期异常特征。

Methods Immunohistochemical and morphometrical analysis methods were performed on vital skin wounds. Results In normal and injury mouse skin IL-10 immunoreactivity was observed in epidermal cells.

方法应用免疫组织化学技术和形态计量法，对小鼠不同时程皮肤切创组织中IL-10不同表达部位（表皮细胞及表皮下组织的浸润细胞）的变化进行研究。

Prenatal EGF can improve pulmonary hypoplasia in the Nitrofen-induced CDH rat model, which was manifested as increased Lw/Bw and more mature histological and morphometrical pulmonary appearances. Moreover, EGF can promote type Ⅱ pneumocytes differentiation and increase their amounts, thus increase the production of surfactant.

EGF对大鼠先天性膈疝模型肺发育不良具有明显的改善作用，不仅可以促进肺组织生长，表现在肺重量／体重增加，肺组织形态得到明显改善，而且对Ⅱ型细胞有明显的促分化作用，Ⅱ型细胞密度增加，功能增强；而且随着EGF剂量的增加作用加强，当剂量为150ug／只（600ug／kg）时，作用较为明显。

Results:(1) FSN can inhibit secondary joints swelling and multi-arthritis evidently, improve the whole condition of rats; at the same time, it can also lighten the synovial inflammation and hyperplasia of lesion joints distinctly, and prevent the joint cartilage and bone from destruction; the collective efficiency of FSN is better than TWP.(2) FSN can raise AA rats low LTT of spleen to normal nearly, remedy the disorder of Th / Ts and Thl / Th2 cells balance in peripheral blood, restrain exorbitant TNF- a ? IL-1 produced by PM O , thereof exert anti-inflammatory and immunoregulation effect.(3) Inside the lesion joints, FSN can depress abnormal hyper-expression of TNF- a mRNA and NF-kB in synovial tissues, as well as advance the expression of Caspase-3 (a proteolytic enzyme of apoptosis), reduce synovial immflamination and proliferation.(4) FSN can lower the expression of VEGF in synovial tissue,reduce neogenetic veins, so inhibit the growth of pannus tissue and the damage of cartilage and bone by that.(5) The above effect of FSN are in proportion to its dosage.Conclusions: FSN has inhibitory effects on symptom and condition of experimental RA, which is better than TWP as a whole.

结果：(1)FSN有明显的抗炎作用，可显著抑制AA大鼠足跖肿胀与多关节炎，改善大鼠的全身情况，同时明显减轻病变关节滑膜炎症与增生，防止关节软骨及骨质的破坏，其综合作用优于TWP；(2)FSN还能使AA大鼠低下的脾LTT恢复至接近正常，纠正外周血中T细胞亚群Th／Ts及Th1／Th2平衡紊乱，抑制大鼠PHφ过高的TNF-α、IL-1分泌，而发挥抗炎和免疫调节作用；(3)在病变关节局部，FSN能显著抑制AA大鼠滑膜细胞异常增高的TNF-αmRNA的表达，降低滑膜组织中NF-kB表达，并增加凋亡蛋白酶Caspase-3表达，从而抑制滑膜的炎症与增殖；(4)FSN还可下调滑膜组织中VEGF表达，减少血管新生，由此抑制血管翳的形成及其对软骨和软骨下骨的侵蚀；(5)FSN的上述作用皆呈现一定的量效依赖关系，高剂量组作用优于低剂量组。

FSN can raise AA rats low LTT of spleen to normal nearly, remedy the disorder of Th/Ts and Th1/Th2 cells balance in peripheral blood, restrain exorbitant TNF-α、IL-1 produced by PMφ, thereof exert anti-inflammatory and immunoregulation effect.(3) Inside the lesion joints, FSN can depress abnormal hyper-expression of TNF-αmRNA and NF-kB in synovial tissues, as well as advance the expression of Caspase-3 (a proteolytic enzyme of apoptosis), reduce synovial immflammation and proliferation.(4) FSN can lower the expression of VEGF in synovial tissue, reduce neogenetic veins, so inhibit the growth of pannus tissue and the damage of cartilage and bone by that.(5) The above effect of FSN are in proportion to its dosage. Conclusions: FSN has inhibitory effects on symptom and condition of experimental RA, which is better than TWP as a whole.

结果：(1)FSN有明显的抗炎作用，可显著抑制AA大鼠足跖肿胀与多关节炎，改善大鼠的全身情况，同时明显减轻病变关节滑膜炎症与增生，防止关节软骨及骨质的破坏，其综合作用优于TWP；(2)FSN还能使AA大鼠低下的脾LTT恢复至接近正常，纠正外周血中T细胞亚群Th/Ts及Th1/Th2平衡紊乱，抑制大鼠PMφ过高的TNF-α、IL-1分泌，而发挥抗炎和免疫调节作用；(3)在病变关节局部，FSN能显著抑制AA大鼠滑膜细胞异常增高的TNF-αmRNA的表达，降低滑膜组织中NF-kB表达，并增加凋亡蛋白酶Caspase-3表达，从而抑制滑膜的炎症与增殖；(4)FSN还可下调滑膜组织中VEGF表达，减少血管新生，由此抑制血管翳的形成及其对软骨和软骨下骨的侵蚀；(5)FSN的上述作用皆呈现一定的量效依赖关系，高剂量组作用优于低剂量组。

Nonadherent cells were removed on day 1 at the time of the first medium change, and fresh medium was changed every 3 days thereafter.

能够作为组织工程中不同细胞的来源；在体内正常循环中能够分布于多种组织和器官，可以用于细胞和基因治疗。

The Western blot results were consistent with those of immunohistochemical method. CONCLUSION: p38 MAPK expression increases in alveolar and airway epithelial cells in LPS induced acute lung injury rat models. The activation of p38 MAPK is found in most lung tissues, suggesting that p38 MAPK participates in the signal transduction in inflammatory and noninflammatory cells.

LPS诱发的大鼠急性肺损伤模型中，磷酸化p38 MAPK在气道和肺组织内表达增加，p38 MAPK的激活见于肺组织内多数细胞，提示肺内炎性和非炎性细胞均有p38 MAPK信号分子的激活。

When they are released into the extracellular environment, or when they are membrane-bound and in contact with the pericellular zone, these enzymes degrade a broad range of connective tissue proteins and are important during the development of healthy tissue as well as in remodeling damaged tissue.

当它们被释放入细胞外环境，或者是与膜结合后与细胞外环境保持接触时，这些酶能够使大范围面积的结缔组织蛋白酶降级，这样对健康组织的发育以及对受损组织的重塑相当重要。

According to the clear water experiment, aeration performance of the new equipment is good with high total oxygen transfer coefficient and oxygen utilization ratio.

曝气设备的动力效率在叶轮转速为120rpm～150rpm时取得最大值，此时氧利用率和充氧能力也具有较高值。

The environmental stability of that world - including its crushing pressures and icy darkness - means that some of its most famous inhabitants have survived for eons as evolutionary throwbacks, their bodies undergoing little change.

稳定的海底环境─包括能把人压扁的压力和冰冷的黑暗─意谓海底某些最知名的栖居生物已以演化返祖的样态活了万世，形体几无变化。