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组织细胞

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Methods The rat model of AD were induced by Aβ25-35 injected into the bilateral amygdala.

在早期的研究中观察到,神经组织细胞在体外培养的存活有赖于NGF的存在。

First, it is an element of the cell.

其一,它是构成组织细胞的成分。

The results showed that with treatment of the fixative solution added with antimonite,the free Ca2+ in pear different organs and tissues could be changed into antimonite calcium and precipitated in situ,which,after being stained,appeared as black particles under electronic microscope,whereas such particles could not be observed in the treatment without antimonite.

结果表明:在固定液中加焦锑酸钾处理进行制样,梨树不同器官或组织细胞中的自由态Caa2+,都能生成焦锑酸钙在原部位沉淀,经染色后在电子显微镜下呈现出黑色颗粒,而固定液中不加焦锑酸钾处理的则没有这种颗粒。

A layer of cells derived from the venter of the archegonium that covers the developing sporophyte.

帽状体:1。根冠:位于根尖的最前端,像帽子一样套在分生区外面,保护其内幼嫩的分生组织细胞

The waste material accumulates and saturates the tissues, which causes autointoxication.

体内的物质垃圾积聚起来,渗透到机体的组织细胞中,而引发自体中毒现象。

For example, malignant histiocytosis has been shown to be genetic in Bernese Mountain Dogs.

例如,恶性组织细胞增生症已被证明是遗传的伯恩山犬。

Fungus cultured: yeast like cells were found in the culture at 37℃ or in tissues.

真菌培养:在37℃培养或组织中马尔尼菲青霉菌呈酵母型,组织细胞外真菌长,弯曲,有横膈的腊肠状;在25℃培养中呈菌丝型,并产生红色色素扩散入培养基中。

Patients developed head cervical lymph node swelling. The lymph node biopsy revealed a background of reactive lymphadenopathy and necrosis in some cases and apoptosis, nuclear debris in all cases, loss of lymph node structure, proliferation of lymphocytes and various types histiocytes with phagocytosis, no infiltration of neutrophils. Three histological types: proliferative, necrotizing and xanthomatous were classified.

形态特征为在活跃的反应性增生淋巴结病变的背景下,出现不同程度的凝固性坏死,淋巴细胞凋亡及核碎片几乎见于所有病例,伴有多种形态的组织细胞增生,吞噬现象明显,无中性杜白细胞浸润,组织学上可分为3型:增生型,坏死型及黄色瘤样型。

Results:bFGF is a biological factor,which can induce cellular proliferation of bone tissue and vascular tissue.

结果:碱性成纤维细胞生长因子是一种生物因子,其可以诱导骨组织及血管组织细胞的分裂。

It has been suggested that HSPs could iufluence cell proliferation and transformation by binding to oncogenes and tumor suppressors.

有研究表明,内源及外源性的活性氧基团均可诱导巨噬细胞HSPs合成,但有关矽尘作用下,组织细胞HSPs表达有何改变,迄今国内外文献尚未见报道。

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