组织原

Objectives To investigate the effect of fibrinogen on tissue-type plasminogen activator and plasminogen activator inhibitor-1(PAI-1) mRNA expression in human umbilical vein endothelial cells.

目的 探讨纤维蛋白原对内皮细胞组织型纤溶酶原激活物和纤溶酶原灭活剂（PAI-1 mRNA表达的影响。

In order to study the ability to the thrombus formation,41 patients with essential hypertension and 34 patients with normotension were investigated.The plasma level of fibrinogen and tissue-type plasminogen activator antigen and plasminogen activator inhibitor 1(PAI-1)antigen were determined.

为了解高血压病人的血栓形成倾向，我们随机选取了41例高血压病人及34例正常血压者，分别观察了他们的纤维蛋白原及血浆组织型纤溶酶原激活物抗原及纤溶酶原激活物抑制物-1(PAI-1)的含量。

Plasminogen activator inhibitor-1 (PAI-1) is a physiological inhibitor of tissue-type plasminogen activator and urokinase-type plasminogen activator .

纤溶酶原激活物抑制因子-1（PAI-1）是组织型纤溶酶原激活物和尿型溶酶原激活物的生理性抑制因子。

Plasminogen activator inhibitor-1 (PAI-1) is a physiological inhibitor of tissue-type plasminogen activator t-PA...

纤溶酶原激活物抑制因子-1（PAI-1）是组织型纤溶酶原激活物和尿型溶酶原激活物的生理性抑制因子。

Methods S-P immunohistochemical staining was used to detect the expression of VEGF-C and its reˉceptor Flt-4in10neighboring noncancerous tissue and60NSCLC cases of non-small cell lung cancer and32lymph nodes cases with lymph nodes metastasis of non-small cell lung cancer.

目的 探讨血管内皮生长因子C及其受体Flt-4在人非小细胞肺癌组织中的表达及其与淋巴结转移等临床病理特征的关系方法采用免疫组化S-P法检测60例NSCLC原发灶组织、10例癌旁组织、32例伴有肺癌转移的淋巴结组织中VEGF-C、Flt-4的表达，并将结果与肺癌的临床病理特征进行分析。

The healing rate of wound of control group is obviously higher than the model group.(3) Observe under the microscope: When 3 days after we practice the surgery, HE and observe control group wound have a large number of inflammation nature cell soak, model group wound surface inflammatory cell relatively sparse in Balb/c mouse;The control group can seeⅠtype collagenic and green type collagenic day, model group type be obviously less than the control group; When 7 days after we practice the surgery,HE and bitter acid-staining observe control group peripheral inflammation cell soak, have appeared granulation organize.

3镜下观察：创伤后第3d，HE染色观察对照组伤口有大量炎性细胞浸润，模型组小鼠创面炎症细胞较稀疏，天狼猩红染色对照组可见少量呈黄色的Ⅰ型胶原和呈绿色的Ⅲ型胶原，模型组Ⅲ型胶原要明显少于对照组；创伤后第7d， HE染色观察对照组周边炎性细胞浸润，有肉芽组织出现。

In the normal uterus, Cytokeratins immunolabelling were detected in glandular cell, luminal epithelial cell, Vimentin immunolabelling were detected in stromal cell and endoblastic cell; CK7 immunolabelling were not detected in any tissue of the yak utenus.

研究结果显示：未妊娠时，泛角蛋白在子宫内膜腺上皮细胞、腔上皮细胞内表达，波形蛋白在子宫内膜基质细胞内表达，平滑肌肌动蛋白在子宫平滑肌和血管平滑肌内表达，牦牛子宫任何部位均不表达角蛋白7；妊娠30天左右时，泛角蛋白在子宫内膜腺上皮细胞、子宫内膜腔上皮细胞、滋养层细胞、内胚层细胞和尿囊细胞内表达，波形蛋白在子宫内膜基质细胞和内胚层细胞内表达，平滑肌肌动蛋白在子宫平滑肌和血管平滑肌内表达，角蛋白7在尿囊细胞内表达，偶尔在腔上皮细胞的细胞核边缘表达；消化法进行原代培养时，组织经胶原酶消化并通过100目和400目筛网组合可以有效地分离原代子宫内膜基质细胞和子宫内膜腺上皮细胞；分离得到的子宫内膜基质细胞活率达90%以上，并可在体外传代7次以上；分离得到的子宫内膜腺上皮细胞活率可达85%以上，并可在体外传代5次以上；RPMI1640培养基最适合子宫内膜基质细胞和子宫内膜腺上皮细胞的生长，维持子宫内膜基质细胞正常生长的FBS添加量为20%，维持子宫内膜腺上皮细胞正常生长的胎牛血清添加量为30%。

The method uses first phase law to determine 63 DVT patient contrasts normally with 30 the F Ⅻ C of the group, use law of thing of the heart that deliver quality to detect at the same time zymogen of serous fine dissolve is active , law of enzymatic couplet immunity measures sex of antigen of content of activation of zymogen of methodize fine dissolve (depressor of content of activation of zymogen of dissolve of T-PAAg), fine - 1 antigen sex (PAI-1Ag) and immunity compare chaotic law to determine serous D-2 gets together body content.

方法采用一期法测定63例DVT患者和30例正常对照组的FⅫC，同时采用发色底物法检测血浆纤溶酶原活性，酶联免疫法测定组织型纤溶酶原激活物抗原性、纤溶酶原激活物抑制剂-1抗原性(PAI-1Ag)及免疫比浊法测定血浆D-二聚体含量。

Results (1) the laminin synthesis was significantly improved by PBMC from nontreatment group (P＜0.05);(2) the 3 H-prolin incorporation was significantl decrease in the GEC treated by PBMC supernatant from nontreatment group(P＜0.05);(3) the supernatant of PBMC from nontreatment group stimulated the collagen Ⅲ synthesis(P＜0.05), while, there was no significant difference of collagen Ⅳ synthesis between 4 groups;(4) the activity of PBMC supernatant have no close relation with urinary protein excretion, pathological type and clinical type.

结果 (1)肾病极期未经激素治疗组PBMC上清明显促进了GEC合成层粘连蛋白；(2)未治PBMC上清抑制了GEC合成总胶原；(3)未治组PBMC上清促进了Ⅲ型前胶原的合成，而对Ⅳ型胶原的合成无明显影响；(4)肾病患儿PBMC的上述作用与是否足量激素治疗有关，而与尿蛋白能否阴转、肾组织病理类型、肾病临床类型等无直线相关关系。

Results Histologically, the tumor was composed of mildly cellular benign-appearing spindle-shaped cells and bands of hyalinized collagen in varying proportions from area to area, and a typical patternless archite...

结果镜下见肿瘤组织由不同比例的纤维母细胞和胶原构成，纤维母细胞分化良好，形态多样，散乱穿梭于胶原蛋白束之间或环绕在玻璃样变的血管周围，伴有局灶性致密区域；胶原成分发生玻璃样变，局部呈轮辐状或瘢痕状排列。

And Pharaoh spoke to Joseph, saying, Your father and your brothers have come to you.

47：5 法老对约瑟说，你父亲和你弟兄们到你这里来了。

Additionally, the approximate flattening of surface strip using lines linking midpoints on perpendicular lines between geodesic curves and the unconditional extreme value method are discussed.

提出了用测地线方程、曲面上两点间短程线来计算膜结构曲面测地线的方法，同时，采用测地线间垂线的中点连线和用无约束极值法进行空间条状曲面近似展开的分析。