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线粒体的

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The mitochondrial gene components and gene arrangement of tokay are typical for vertebrates.

大壁虎线粒体基因组成和排列顺序为典型的脊椎动物线粒体组成。

Yangtsekiense populations. 1. The mitochondrial genome S. yangtsekiense and phylogentic studies of Decapoda We determined the complete 11, 405 bp mitochondrial DNA sequence of S.

长江华溪蟹线粒体基因组及十足目系统发生研究测定了长江华溪蟹线粒体基因组11, 405 bp的核苷酸序列,包括11条蛋白编码基因和17条tRNA基因。

However, Ooplasmic transfer can lead to mtDNA become heteroplastic, which is the mtDNA of donor and recipient coexist in offspring.

但是,卵胞质转移也可能会导致线粒体DNA异质,即供体和受体的线粒体DNA同时存在于后代体内。

The gene order data indicates that Hexapoda and Crussacea are more closely related with each other, and Chelicerata is closely related with Myriopoda. The results have conflicted with those of Boore (1998) results, but congruence with the results based on nuclear rRNA data.

线粒体基因组基因排列顺序数据显示六足动物与甲壳动物之间相似,螯肢动物与多足动物相似,这个结果和以前Boore(1998)对节肢动物线粒体基因组顺序分析结果不同,却和核rRNA数据的分析结果一致。

Objective To analyze clinical characteristics of 8 cases of MELAS syndrome that misdiagnosed as herpes simplex virus encephalitis.

线粒体脑肌病伴乳酸血症和卒中样发作综合征具有线粒体脑病、高乳酸血症和卒中样发作的特征,确诊主要依赖临床表现和肌肉病理检查。

MRS2 encode a magnesium transporter protein in Saccharomyces cerevisiae and deletion of MRS2 gene results in decrease in mitochondrial magnesium concentration, Group II RNA splicing defect and growth defect on nonfermentable carbon source.

MRS2 基因编码线粒体镁离子转运蛋白,MRS2缺失会导致酵母线粒体镁离子浓度下降、线粒体内II型内含子剪接缺陷和非发酵碳源上的生长缺陷。

ResultsCompared with group A and group C, significant increase in the levels of IL 8 and MDA and decrease in the activities of SOD and GSH Px were noted in group B (F=5.86-18.41,q=3.15-7.17, P.01); compared with group A, the activity of SOD and GSH Px was not changed markedly, the levels of IL 8 and MDA rised slightly but with no statistically significant. Mitochondria became vacuolated and deformed in group B, slightly swollen in group C.

结果 与A组、C组相比较,B组SOD、GSH Px的活性显著下降,MDA含量、IL 8浓度显著上升(F=5.86~18.41,q=3.15~7.17,P.01);与A组比较,C组SOD、GSH Px活性无明显变化,MDA含量及IL 8浓度轻微上升,但无统计学差异。B组肺组织线粒体破坏,呈空泡样变,C组仅见线粒体轻微肿胀。

This study demonstrated that cytotoxicity of adrenoco rtical cells induced by lead acetate is probably attributed to mitochondria oxid ative stress. Mitochondria damage might be the early cellular and molecular even t in toxic effect of lead on adrenal cortex.

线粒体氧化应激介导肾上腺皮质细胞毒性可能是PbAc毒作用机制之一,线粒体损伤是铅致肾上腺皮质毒作用的早期细胞和分子事件。

By electron microscope we can find the Shenqi group's cancer nuclear chromatin is partly condensing and a few with screen opening changing, the membrane is invaginating , the mitochondrias are mostly swelling and the number is decreased, with the cristae'number decreased or breakon even diminished and destructured in different degree, parts of the mitochondrias are bubbling changing.

细胞膜内陷,线粒体数量减少,多数肿胀,嵴减少或断裂,甚至消失,有不同程度的变性,部分线粒体呈空泡状。

In heathlty cells, AIF protein is normally confined to mitochondrial intermembrane space. However, after apoptosis is induced, AIF protein can be released from mitochondria and translocates into nucleus leading by its nuclear localization signal.

正常情况下,AIF蛋白定位于细胞线粒体膜间隙,当有凋亡信号刺激时,AIF分子从线粒体释放到细胞浆,再通过其核定位信号转位到细胞核中,直接引起染色体凝集和DNA的大片段(~50kb)断裂,导致细胞凋亡。

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