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Induction of apoptosis by Etoposide was associated with upregulation of mRNAs for the BH3 only proteins Noxa, and to a lesser extent, Bim. In contrast, the expression levels of mRNAs for PUMA remained unchanged before and after treatment with Etoposide.

Noxa和PUMA主要受p53调节,细胞DNA损伤后通过p53可以上调Noxa和PUMA的表达[1]; Bim主要结合在微管动力蛋白上,受到细胞因子去除、Ca2+流出、紫外线照射、微管动摇和紫杉醇等刺激后可以转移到线粒体膜上[2],从而发挥促凋亡活性。

The results showed that ACP positive reactions were mainly deposited in lysosome, nucellar cell, cellular endomembrane, mitochondriurn, and endoplasmic reticulum in four tissues of the healthy shrimp. Furthermore, ACP activity was located in microvilli, and around partly lipid droplet in liver. AKP positive reactions were mainly deposited in cell membrane, nucellar cell, lysosome. endoplasmic reticulum and cellular endomembrane in four tissues.

结果显示,在健康的对虾体内,ACP依次出现在各组织中的溶酶体、细胞核、细胞内膜、线粒体、内质网以及肝脏组织中的部分脂滴周围及微绒毛中AKP依次出现在各组织中的细胞膜、细胞核、溶酶体、内质网以及肝脏组织中的脂滴周围及微绒毛中。

It was observed by means of electronic microscope that toxinresulted in the apparent change in ultrastructure of rapeseed leafincluding the damage of chloroplast membrane,dilated,disorderedgrana lamellae;the damage of mitochondria membrance andeventually vacuolated mitochondria;the contract of cell nuclein.

电镜观察发现:菌核菌产生的毒素引起油菜叶片超显微结构显著变化,主要包括叶绿体膜破坏,基质片层肿胀,排列紊乱,形成空腔,最终叶绿体解体;线粒体膜破坏,最后空泡化;细胞核质凝缩。

Two independent molecular sources were used to reconstruct phylogeny: the 16S rRNA gene on the mitochondrial genome and the 28S rRNA gene on the nuclear genome. A comparison of the sequences showed that the obtained 28S rDNA sequences have evolved at a much slower rate than the 16S rDNA, and that the former is better than the latter for resolving deep branching in the Odonata.

两种独立的分子资源用于重建系统发育发展史:线粒体基因组的16s rRNA基因和核基因组的28s rRNA序列比对表明:获得的28s rDNA序列以远低于16s rDNA的速率进化,并且28s rDNA比16s rDNA更适于分析蜻蜓目的深度分支。

This paper reviews the ooplasmic transfer in summary, the relation between ooplasm and fertilization and embryo development, the hereditary behavior of heterogenous mitochondria DNA and it's examination after ooplasmic transfer.

通过卵胞质转移研究概况、卵胞质与受精和胚胎发育、异种线粒体DNA遗传方式和卵胞质转移遗传物质的检测4个方面对卵胞质转移技术进行讨论。

Objective To investigate the clinical, genetic and pathological features of familial chronic progressive external ophthalmoplegia type of mitochondrial myopathy.

目的 探讨家族性慢性进行性眼外肌瘫痪型线粒体肌病的临床、遗传和病理特点。

To study the mitochondrial deletion DNAs of skeletal muscle from patients with chronic progressive external ophthalmoplegia and KearnsSayre syndrome.

目的:检测慢性进行性眼外肌麻痹(chronic progressive external ophthalmoplegia, CPEO)及KearnsSayre综合征患者骨骼肌细胞线粒体DNA的缺失情况。

At mononucleus microspore stage,cytoplasm were thick and organella,such as mitochondria,endoplasmic reticulum were abundant.At two\|nucleus and three\|nucleus stage,there were a lot of starch grains.

结果表明,85 EA、89 AR、TA 与恢复系杂交F1 代小孢子发育表现出明显的可育特征:在单核期,细胞质浓,细胞器丰富,线粒体、内质网等十分发达,二核及三核期淀粉粒数量多。

The changes of osteoblastic surface structure and ultrastructure were observed under electron microscope.

结果 (1)PDGF对培养24小时的成骨细胞S期的DNA含量明显增高(17.7%);(2)PDGF促使细胞内线粒体更丰富,粗面内质网增多。

The otological, audiological and neurological examinations of these patients were conducted, including otoscopy, pure-tone (Madsen 522) and immittance(GSI 33) audiometry. MtDNA A1555G mutation and GJB2 235delC mutation were detected with specific restriction enzyme digestion methods.

对 148 名非综合征型感音神经性耳聋患者分别进行线粒体 DNA 12SrRNA基因A1555G 点突变和 GJB2 基因235delC突变的限制性内切酶分析。

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