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In this family,deafness is maternally inherited and all the sufferers have the mitochondrial DNA 12s RNA A1555G mutation. Four methods are used in the experiments for establishing immortal lymphoblastoid cell lines of the family with non-syndromic deafness.

该家系患者呈典型的母系遗传特征,且研究发现患者中均具有线粒体DNA 12s RNA A1555G突变,是迄今世界上最大的非综合征耳聋家系之一,在该家系的建系过程中使用了4种不同的方法。

After three days, the ultrastructure of mesophyll cells of wheat seedlings were observated.

线粒体结构普遍受到损害,表现在外膜、内膜和嵴膨胀,结构模糊。

Under normal temperature,both Ca2+ and oxalate treatments could induce theexpression of sHSP in mesophyll and root cells.

常温下,在对照辣椒叶肉细胞和根尖分生细胞中,小分子量热激蛋白的表达量非常低或很难观察到;热胁迫下在细胞核、线粒体以及细胞质中增加。

When the pollen tube comes into the micropyle and contact one of the synergids, both synergids do not show any sign of degeneration.

在二细胞原胚的顶细胞和基细胞中,均可观察到与合子中形态相似的双亲质体和线粒体

Objective To explore the mitochondria and microsome of testis cells caused by nickel sulfate in rate.

目的从氧化应激角度探讨硫酸镍(NiSO4)对大鼠睾丸细胞线粒体及微粒体的毒性作用。

Abstract摘要: Objective To explore the mitochondria and microsome of testis cells caused by nickel sulfate in rate.

摘要摘要:目的从氧化应激角度探索硫酸镍(NiSO4)对大鼠睾丸细胞线粒体及微粒体的毒性功能。

The rat's testis damage is related to the enhancement of oxidative stress of mitochondria and microsome caused by nickel sulfate.

镍对大鼠睾丸细胞的损伤可能和其所致的睾丸细胞线粒体及微粒体氧化应激效应增强有关。

The generative cell contains a few plastids immediately after the microspore mitosis in L.

在生殖细胞的发育过程中,线粒体的DNA也很快降解。

In L. davidii, the male plastids were stripped off during the division of microspore.

在生殖细胞的发育过程中,质体发生解体,到成熟花粉的生殖细胞只含线粒体,没有质体。

These mimetic molecules do their damage without the need for the Smac signal from the mitochondria.

这些模拟分子不需要线粒体释放Smac信号就可以执行破坏功能。

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。

There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.

双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。