线粒体

Membrane alterations (membrane vacuoles, invagination into adjacent organelles, and myelin figure-like changes) occur in a subpopulation of mitochondria in sepsis, but other hepatocyte organelles showed no consistent ultrastructural injury.

败血症中膜的改变（膜泡，向邻近的细胞内陷和髓磷脂形态样的变化）发生在线粒体的亚群，但是其他肝细胞器官未见一致的超微结构损伤。

These cell factors will be released to cytosol under stimulating factor by involucrum penetrability changes or involucrum breaking. This will render amplifying of apoptosis signals.

在凋亡刺激因素作用下，线粒体渗透性发生变化或外膜破裂，这些细胞因子将释放入胞浆内使凋亡信号得以放大，最终导致细胞凋亡。

The isolated working heart model was set up, high-performance liquid chromatography were used to measure myocardial adenosine triphosphate,total adenine nucleotides, while left ventricular end-diastolic pressure,left ventricular developed pressure, and rate of change of left ventricular pressure were determined before and after preservation.

对照组用St Thomas停搏液停搏并保存大鼠心脏6小时；实验组在St Thomas液中加入前列腺素E1(5 μg/L)停搏并冷保存6小时，利用离体鼠心非循环式Langendorff灌流功能测定模型，测定左心室舒张期末压、左心室发展压和左心室压力变化率，并检测心肌细胞线粒体三磷酸腺苷、腺嘌呤核苷酸总量。

Objective To observe the change of mitochondrial injury and c-Jun N-terminal kinase activity in kainite acid induced epilepsy in rat.

目的 观察海人酸致颞叶癫癎大鼠海马神经元线粒体损伤及c-Jun氨基末端激酶活性的改变。

As the ischemic time was prolonged, amorphous dense bodies were found in swellen mitochondria, and the nucleus progressed gradually from early pyknosis to karyswelling, karyorhexis and karyolysis. The myocardium eosin staining became pale. Cell membrane was disrupted. This indicates that it entered an irreversible swelling stage.

随着缺血时间的延长，缺血1小时后，肿胀的心肌细胞，线粒体可见不定形颗粒，细胞核逐渐由早期固缩到肿胀、破裂及溶解，细胞伊红染色变淡，细胞膜破裂，表明此时的细胞已进入不可逆性肿胀阶段。

We also have observed some organs pathological changes of juvenile Jian Carp, such as pancreas acinous gland and zymogen granules of acinous gland cell disappearing, intercellular substance hyperplasia and inflammatory cells soakage, hepatatrophia, liver cell granular or vacuolar degeneration and necrosis, karyolysis or pyknosis, glycogen granules decreasing, metanephros atrophy, metanephric canaliculus epithelium granular or vacuolar degeneration and necrosis, mitochondrion swelling and mitochondrion cristae disappearing, karyolysis, distal convolutal tubule microvilli desquamating, spleen marrow cell degeneration and necrosis, intercellular substance of spleen hyperplasia, spleen atresia, blood corpuscle disappearing.

后肾土黄色、淡褐色或苍白色，肾小管上皮细胞肿胀、颗粒或水泡变性、坏死，细胞内有大量血细胞流出，线粒体肿胀，嵴结构消失，细胞核溶解，肾间质甲状腺滤泡和拟淋巴细胞增生，远曲小管微绒毛脱落、管道细胞界限不清。心脏肌纤维肿胀、颗粒或空泡变性，严重的肌纤维溶解、变细或断裂，肌纤维间水肿、炎性细胞浸润，部分心肌细胞核浓缩。脾脏暗褐色，脾髓质细胞变性、坏死，拟淋巴细胞明显减少，淋巴细胞岛少见、岛中细胞成份减少，黑素巨噬细胞中心减少、体积缩小，脾脏网状基质水肿，脾窦闭锁，血细胞减少。

An abnormal distribution of Ca〓 in mitochondria of myocardial cell from patients with KD was also observed.

与此同时，我们还发现克山病患者的心肌细胞和心肌线粒体的Ca〓平衡有异常现象。

Methods Rat survival rate, hepatocytic mitochondrial respiratory function, hepatic ATP content and ketone body ratio in arterial blood were investigated at different time after portal vein ligation under portal vein bypass.

在门静脉转流下阻断门静脉不同时间后，观察大鼠存活率、肝细胞线粒体呼吸活性、肝组织ATP含量及动脉血酮比值。

Krause said at a news conference that the lab was sequencing mitochondrial DNA from several samples when he discovered one that was very unusual.

克劳斯在记者招待会上说，实验室是测序时，他发现这就是一个非常不寻常的几个样本线粒体

Nucleus of endothelial cell was often pushed to laterality and its shape was flat.

内皮细胞的细胞核常被挤到一侧，成扁形，细胞质中多含有线粒体。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。