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RESULTS: The collapse was obvious at weeks 2 and 6 after modeling, with only few granulation tissues and fibrous tissues. No chondrocytes were observed. At weeks 12 and 18, fibrous tissues were found at the defect site, and bone tissue and cartilage proliferation was observed at the bottom and margin. However, it was greatly different from normal cartilage. Moreover, there was still no chondrocytes.

结果:实验兔后腿关节的股骨髁关节面的关节软骨缺损后,在制模后2,6周缺损部位凹陷明显,只有少许肉芽组织和纤维组织生长填充,未见软骨细胞爬行生长,12,18周见缺损部位以纤维组织增生为主,底部及边缘可见骨组织和软骨增生,但是镜下形态仍与正常软骨组织区别较大,表面仍未见软骨细胞爬行生长。

Fibrocartilage is found around joints such as your knees and hips, and in your spine.

纤维软骨是发现周围的关节,如您的膝盖和臀部,并在您的脊椎。

A new technique is presented here consisting of reconstruction of the radial head, leveling of the distal radioulnar joint, reconstruction of the central band of the interosseous membrane by using a pronator teres rerouting technique, and finally repair of the triangular fibrocartilage complex.

这里介绍一种新的技术,它包括:桡骨头的重建,远端桡尺关节的校准,用旋前圆肌代替技术重建骨间膜的中心联合腱,最后是三角纤维软骨联合体的修补。

Six weeks after implantation, small amount of oval lacunae appeared. PGA fibers began to be absorbed. The amount of collagen increased and Safranin O stained positively. Eight weeks after implantation, cartilage with single or occasionally double chondrocytes in round lacunae was found.

体内埋植4周后可见PGA纤维间被不成熟的软骨细胞充填。6周时出现椭圆形软骨陷窝,PGA纤维开始吸收,胶原增多,番红花—"O"有阳性染色。8周时软骨陷窝呈圆形,单个,双个对称或多个排列。

Results: Arthroscopic examination showed that there were synovitis, debris and fibrillation in the joints of ADD with reduction; synovitis, synovial hyperplasia, debris, fibrillation, chondromalacia,fibrocartilage rupture and adhision in the joints of ADD without reduction.

结果:可复性关节盘前移位的病例出现滑膜炎,关节腔内有絮状物,关节结节表面有纤维形成。不可复性关节结节表面纤维形成,关节软骨软化,软骨剥脱,同时伴有纤维粘连。

This transforms the callus into a tougher fibrocartilaginous callus, which bridges the gap between the two pieces of bone.

这个过程将血痂转变成一种更硬的纤维软骨痂,它把两块分裂的骨骼桥接在一起。

Decrease in expression of TIMP-3, possibly mediated in part by TGF-1, may cause imbalance of ADAMTS4/TIMP-3 ratio at transition period from notochordal to fibrocartilaginous NP.

可能受TGF-1调节的TIMP-3表达水平的降低,可能会导致在脊索NP向纤维软骨NP转换过程中的ADAMTS4/TIMP-3比例的失衡。

Results. Semiquantitative assessment of histologic changes indicated that 4-week-old rabbit was equivalent to fetus to 2-year-old human and 160-week-old rabbit was equivalent to 11- to 30-year-old human, particularly 11- to 16-year-old, which corresponds to transition period from notochordal to fibrocartilaginous NP.

结果:组织学改变上的半定量测定显示,4个月的兔子相当于2岁的孩子,160个月大的兔子相当于11到30岁大的成人,尤其相当于11到16个月大,相对地,这段时间正好是脊索向纤维软骨NP转换的时期。

Results. Semiquantitatie assessment of histologic changes indicated that 4-week-old rabbit was equialent to fetus to 2-year-old human and 160-week-old rabbit was equialent to 11- to 30-year-old human, particularly 11- to 16-year-old, which corresponds to transition period from notochordal to fibrocartilaginous NP.

结果:组织学改变上的半定量测定显示,4个月的兔子相当于2岁的孩子,160个月大的兔子相当于11到30岁大的**,尤其相当于11到16个月大,相对地,这段时间正好是脊索向纤维软骨NP转换的时期。

Results 1、 Generally, we can see the original blue and white, shiny, no cracks in the articular surface of the cartilage after the stress increases gradually yellow, surface roughness, cracks appear; when the pressure decreases, the yellowing, rough, the color of the fracture restore gradually and become shiny.2、the shiny smooth surface can be seen under a light microscope, formation, cell distribution, tidy, clear the level of cartilage at the articular surface stress increases, the surface roughness changes, defects, disordered cells, uneven dyeing ; when the articular surface of the pressure gradually decreased, the cartilage gradually repair and the surface of cells at the surface appear only disorder.3、immunohistochemical observation can be seen throughout the observation period, cartilage cells are type Ⅱ collagen expression and expression after 3 weeks gradually weakening, when the seventh week begin to strong gradually.4、 electron microscopy shows that when stress increases the articular surface, the cartilage cells became flat, the cytoplasm in the endoplasmic reticulum, Golgi apparatus decreased with collagen disorders; and when stress decreases the articular surface, cartilage cells gradually returned normal, cytoplasm in the endoplasmic reticulum, Golgi body gradually restore quantity; collagen fibers with a gradual rules.

结果:①大体观察可见到原本蓝白色、有光泽、无裂纹的软骨在关节面压力增大后,逐渐呈灰黄色,表面粗糙,出现裂隙;当压力逐渐减小后,变黄、粗糙、有裂隙的软骨颜色逐渐恢复,变得有光泽②光镜下可见表面光滑、平整,细胞分布均匀、整齐,层次清楚的软骨在关节面压力增大后,表面变粗糙、缺损,细胞排列紊乱、染色不均;当关节面压力逐渐减小后,软骨表面逐渐修复,细胞仅在表层排列紊乱③免疫组织化学观察可见整个观察期内软骨细胞胞浆内均有Ⅱ型胶原表达,术后3周内表达逐渐变弱,从第7周时开始逐渐变强。④电镜下可见当关节面压力增大后,软骨细胞逐渐变扁,胞质中内质网膜、高尔基体减少,胶原排列紊乱;当关节面压力减小,软骨细胞形态逐渐恢复正常,胞质中内质网膜、高尔基体数量逐渐恢复;胶原纤维排列逐渐有规则。

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