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In electricity plus thermal group, the pathologic alterations were slighter than model group; there was little crack on the surface of cartilage, with intact collagen fiber structure and less pyknotic chondrocytes. Although some chondrocytes shrank, there were intact cell organs in many chondrocytes even chondrocyte cluster in some regions.

疏密波+热软膜组在病理程度上的改变明显轻于模型对照组,软骨表面裂纹少,胶原纤维结构基本完整,固缩的软骨细胞少,虽可见部分退变的软骨细胞,但部分软骨细胞具有较多的细胞器,某些区域形成软骨细胞簇。

Methods N-vinyl pyrrolidone, the hydrophilic monomer, was grafted onto the surface of silicone breast implants by 60Co radiation.The implants were buried in the bilateral subcutaneous layers of the back of rabbits.The unchanged implants were used as the control group.

利用钴60共辐射的方法把亲水性单体 N-乙烯基吡咯烷酮接枝到硅胶乳房假体表面,改变其表面特性后植入兔的背部,以未经改性的正常假体作为对照组,隔一定时间通过组织切片及CT扫描的方法观察假体周围纤维囊壁的形成情况。

Confocal laser-scanning microscropy funther revealed that substantial number of 5-HT-immunopositive fibers and terminals distributed widely in the whole VNC, and some of 5-HT-immunopositive terminals were closely surroud the somata or dendrite of TMT/5-HT1AR double-labeling neurons and make close contact with them.

2激光扫描共聚焦显微镜下可进一步观察到大量的5-HT样阳性纤维和终末广泛地分布于VNC的各个核团内,且有部分5-HT样阳性终末紧紧地包绕在TMR/5-HT1AR双标神经元的胞体或树突的周围,并与之形成密切接触。

Results On conventional CT, pulmonary window revealed widespread intraalveolar calcifications of both lungs concentrating in the subpleural parenchyma of the middle and lower lobes.

结果常规CT表现:肺窗示肺实质内有无数细小散在的粟粒结节,以中下肺的外周部密集,其CT值为200~400HU,多合并不同程度的肺气肿及间质纤维化;纵隔窗示细结节影最密集区常呈沿胸膜的线带状或散在的点簇状钙化,形成&火焰征&及&白描征&。

Injuries to the anterior talofibular andtibiofibular ligaments and an accessory fascicle of the anteriortalofibular ligament have been implicated as causes of anterolateralimpingement syndrome. Repetitive synovial inflammation secondaryto chronic lateral ankle instability produces a soft-tissue"mass" consisting of hypertrophic synovial tissue and fibrosiswithin the lateral gutter.

前距腓韧带、胫腓韧带和前距腓韧带的副束的损伤被认为对前外侧碰撞综合症相关,继发于慢性外侧踝不稳定的反复滑膜炎在外侧沟形成软组织&肿块&,主要是肥大的滑膜组织和纤维化。

The transformed cell could be agglutinated by lower concentration of ConA and could grow in semisolid agar.

为了探讨三氧化二镍(Ni2O3)诱导人肺成纤维细胞恶性转化作用以及β-胡萝卜素和维生素C的保护作用,用不同浓度的Ni2O3在体外多次处理HLF细胞,利用刀豆凝集素A凝集实验和软琼脂集落形成实验进行转化细胞的恶性鉴定;同时添加β-胡萝卜素(5.4mg/L)和维生素C的食物(1.8mg/L),观察对Ni2O3转化作用的影响。

At carbonizing temperature between 425 ℃ and 720 ℃,the micropore volume is between 0.124-0.222 mL/g. Surface of the carbon is also observed by scanning electron microscopy. During thermogravimetric analysis,the cell wall of coconut shell disassembles to form macropore at μm level while its cellulose structure was retained.

本研究还探讨了椰壳炭的孔结构参数,炭化温度 4 2 5~ 72 0℃,其微孔容积为 0 。12 4~ 0 。2 2 2mL / g ,并用扫描电子显微镜观察了椰壳炭的表面形貌,椰壳在热分解时,细胞壁分解形成了微米级的大孔,并保留了椰壳的纤维状结构。

In carcinomatous mucosa, immunoreaction of the carinomatous cell nest was negative, or only remnants of positively were seen at the site of the former membrane by collagen type Ⅳ.

结果 正常组织的基底膜呈现很强的Ⅳ型胶原抗体阳性反应,在基底膜形成较粗的环形染色带;Ⅲ型胶原抗体阳性反应在细胞外间质呈连续的细丝纤维样。

Methodshexamethyldisiloxane polymer film was obtained by plasma polymerization method on coverslips. oxygen (o2) plasma treatment was employed to modify the surfaces with different time so that a series of surface with different wettability were produced. l929 cell were cultured on these surfaces to observe the cell attachment rate and cell spreading in 6 and 12 hours.

使用薄膜等离子体聚合法在圆形盖玻片表面生成六甲基二硅氧烷(hexamethyldisiloxane, hmdso)薄膜聚合体,然后用低温氧等离子体轰击聚合体表面,根据轰击时间的不同形成一系列的不同润湿性表面,将成纤维细胞系l929接种于不同润湿性表面,观察6h、12h时细胞的贴附率,并观察细胞的形态。

Methods Hexamethyldisiloxane polymer film was obtained by plasma polymerization method on coverslips. Oxygen (O2) plasma treatment was employed to modify the surfaces with different time so that a series of surface with different wettability were produced. L929 cell were cultured on these surfaces to observe the cell attachment rate and cell spreading in 6 and 12 hours.

使用薄膜等离子体聚合法在圆形盖玻片表面生成六甲基二硅氧烷(Hexamethyldisiloxane, HMDSO)薄膜聚合体,然后用低温氧等离子体轰击聚合体表面,根据轰击时间的不同形成一系列的不同润湿性表面,将成纤维细胞系L929接种于不同润湿性表面,观察6h、12h时细胞的贴附率,并观察细胞的形态。

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