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纤维形成

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One of the proteins into which actomyosin can be split; can exist in either a globular or a fibrous form.

肌动球蛋白分解形成的一种蛋白质,呈球状或纤维状。

BACKGROUND: Fibration and scar formation are major factors for surgical failure following glaucoma filtration. Although there are so many drugs and methods to inhibit scar formation, they are not ideal.

背景:青光眼滤过后,滤过道的纤维化与瘢痕组织形成,是手术失败的主要原因,虽然抑制青光眼滤过术后瘢痕形成的药物和给药方法很多,但多不理想。

The corneal fibroblast of rabbit formed a monolayer and had a fibriform arragement after two weeks culture in culture flasks.

眼角膜成纤维细胞在培养瓶中培养 2周后可形成单层;2 4孔板培养 3d后细胞贴壁生长旺盛,10d形成单层。

4Histological examination of the capsular bags: It could be seen in group 4 that a small amount of LECs at the capsular equator proliferated and migrated to the posterior capsule, forming a thin soemmering ring in the periphery of the capsular bags, forming thin fibromembrane which embodied monolay metaplastic fibroblast-like cells in the center of the capsular bags, or with no cells or cortical material in the center of the capsular bags.

囊袋组织学检查:HSV-TK+GCV组可见少量赤道部晶状体上皮细胞(lens epithelial cells, LECs)增殖并向后囊膜迁移,在囊袋外周部形成稀薄的soemmering环,在中央部无细胞和皮质,或仅形成单细胞层纤维膜。

Results: No fiber layer and inflammatory tissues around any inserted implants were founded. New-grown osteoid tissue appeared faster in Bio-Ti implants than pure titanium and matured bone tissues comprise bone trabecula and Haversian canals appeared in 6 weeks, while at least 12 weeks needed to form matured bone tissue in control groups.

结果:所有种植体周围没有发现明显炎症组织或形成纤维层,Bio-Ti种植体组新生类骨组织生长较快,并于6w时已形成含有骨小梁及哈弗氏小管结构的较成熟骨组织,对照组12w时新生骨组织基本成熟。

The human cartilages are composed of chondrocyte and extracellular matrix,the form of chondrocytes are hypertrophy and the quantity are less;the ECM of cartilage are compised of type II collagen and proteoglycan. Articular cartilages are all hyaline with little fibers. Trauma and arthritis are the main cause of cartilage injury,the ommilayer injury ofcartilage can be recovered by marrow,but because of without stimulation mechanism,the new tissues are merely fibrocartilages,they can not be coincide with hyaline cartilage in menchanics;the purely damage of articular cartilage can not stimulate chondrocyte to regenerate because of without blood circulation,thus,the plerosis of articular catilage can not depend on the proliferation of local chondrocyte.Ever since,people tried their best to find a way to reconstruct articular cartilage.

造成人体关节软骨损伤的原因主要为创伤和关节炎,关节软骨全层损伤可由于骨髓中间充质干细胞的高速增殖修复,但这种修复由于缺乏相应的刺激机制,只能形成纤维软骨,而不能形成符合关节生理、力学要求的透明软骨;单纯软骨部分损伤软骨组织内无血管,软骨细胞迁移迟缓,无法使损伤区域软骨细胞再生,因此,关节炎及关节创伤后的软骨修复不能依赖于软骨细胞的增殖和迁移。

The human cartilages are composed of chondrocyte and extracellular matrix , the form of chondrocytes are hypertrophy and the quantity are less; the ECM of cartilage are compised of type Ⅱ collagen and proteoglycan. Articular cartilages are all hyaline with little fibers. Trauma and arthritis are the main cause of cartilage injury, the ommilayer injury ofcartilage can be recovered by marrow, but because of without stimulation mechanism, the new tissues are merely fibrocartilages, they can not be coincide with hyaline cartilage in menchanics; the purely damage of articular cartilage can not stimulate chondrocyte to regenerate because of without blood circulation, thus, the plerosis of articular catilage can not depend on the proliferation of local chondrocyte.

造成人体关节软骨损伤的原因主要为创伤和关节炎,关节软骨全层损伤可由于骨髓中间充质干细胞的高速增殖修复,但这种修复由于缺乏相应的刺激机制,只能形成纤维软骨,而不能形成符合关节生理、力学要求的透明软骨;单纯软骨部分损伤软骨组织内无血管,软骨细胞迁移迟缓,无法使损伤区域软骨细胞再生,因此,关节炎及关节创伤后的软骨修复不能依赖于软骨细胞的增殖和迁移。

The human cartilages are composed of chondrocyte and extracellular matrix , the form of chondrocytes are hypertrophy and the quantity are less; the ECM of cartilage are compised of type Ⅱ collagen and proteoglycan. Articular cartilages are all hyaline with little fibers. Trauma and arthritis are the main cause of cartilage injury, the ommilayer injury ofcartilage can be recovered by marrow, but because of without stimulation mechanism, the new tissues are merely fibrocartilages, they can not be coincide with hyaline cartilage in menchanics; the purely damage of articular cartilage can not stimulate chondrocyte to regenerate because of without blood circulation, thus, the plerosis of articular catilage can not depend on the proliferation of local chondrocyte. Ever since, people tried their best to find a way to reconstruct articular cartilage.

中文题名人骨髓基质干细胞成软骨诱导及多孔复合材料作为细胞载体的体外实验研究副题名外文题名 Cartilage induction of human mesenchymal stem cells and experiment on compound porous materials as cells' scaffold in vitro 论文作者刘晓岚导师周江南学科专业外科学研究领域\研究方向学位级别博士学位授予单位中南大学学位授予日期2003 论文页码总数68页关键词骨组织工程软骨细胞骨髓基质干细胞壳聚糖高分子外消旋聚乳酸馆藏号BSLW /2003 /R68 /10 造成人体关节软骨损伤的原因主要为创伤和关节炎,关节软骨全层损伤可由于骨髓中间充质干细胞的高速增殖修复,但这种修复由于缺乏相应的刺激机制,只能形成纤维软骨,而不能形成符合关节生理、力学要求的透明软骨;单纯软骨部分损伤软骨组织内无血管,软骨细胞迁移迟缓,无法使损伤区域软骨细胞再生,因此,关节炎及关节创伤后的软骨修复不能依赖于软骨细胞的增殖和迁移。

The lateral tendinous fascicle formed mainly functional anatomic mechanism of the proxinmal interphalangeal joint was extended, and it had close relation to the lateral fascicle and had a coordinated action with the lateral tendon.The lateral tendinous fascicle had also association with the flexor tendinous sheath, and made the flexor tendinous sheath play a role in balancing muscular strength of flexor and extensor.

外侧腱束主要形成伸近节指间关节的功能解剖机制;其与外侧束形成的密切纤维联系而具有协同外侧腱的功能;该腱束与屈肌腱鞘有联系又使其具有平衡屈肌和伸肌肌力的作用。

Results show that chemical reaction between additives Al2O3 and Y2O3 accelerated the liquid phase sintering and the formation of the YAG non-crystalline,which was beneficial to improving fracture toughness of Cf/SiC composites.Higher sintering temperature resulted in the strongly bonded fiber/matrix interface.Fiber push-off,crack deflection and grains bridging are main toughening mechanisms of silicon carbide cera...

结果表明:烧结过程中,烧结助剂Al2O3、Y2O3之间发生化学反应,促进液相烧结,形成晶界间的次晶相YAG(3 Y2O3.5Al2O3),有利于提高复合材料的断裂韧性;在较高烧结温度下,碳纤维、烧结助剂与基体间发生反应,形成较强结合界面;纤维拔出、裂纹偏转和晶粒桥联是碳化硅陶瓷的主要增韧机制。

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