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纤维形成

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In the wet process the web of fibers may be formed from a suspension of the fibers in water.

在湿法成网过程中,可以利用悬浮在水中的纤维形成纤维网。

2Cm bone defect was established in ulna of rabbits, the capacity of different therapeutic measures in bone defect repairment was determined in groups A , B (implanted BMP, TGF -β〓 and carrier) and C . It was found that the appearance of osteoblasts and formation of bone union were earlier and the number of osteoblasts more in group B than in group A. The amino acid analysis revealed that the volume of ossein was significantly higher in group B than in group A at the early stage.

分别用bBMP及载体bBMP、TGF-〓及载体和单纯载体修复兔尺骨1.2cm骨缺损的实验模型来比较三者间的成骨效果,结果表明B组比A组成骨细胞出现早、形成数量多、骨连接形成提前,骨胶原氨基酸分析在骨缺损修复早期胶原形成量B组均高于A组,且B组以形成Ⅰ型胶原为主,C组表现为一纤维形成过程。

The results confirmed the significant inhibitory effect of glycosylation on fibril formation, and showed that fibrils formed more easily when the residue 129 is valine.

结果发现醣化会大大的抑制自发性纤维形成,而且以含醣胜肽的129号残基为缬胺酸时,纤维的形成较容易。

Results: Arthroscopic examination showed that there were synovitis, debris and fibrillation in the joints of ADD with reduction; synovitis, synovial hyperplasia, debris, fibrillation, chondromalacia,fibrocartilage rupture and adhision in the joints of ADD without reduction.

结果:可复性关节盘前移位的病例出现滑膜炎,关节腔内有絮状物,关节结节表面有纤维形成。不可复性关节结节表面纤维形成,关节软骨软化,软骨剥脱,同时伴有纤维粘连。

Desmodium pulchellum has the effect on antihepatic fibrosis. The mechanism is possibly concerned that it can make liver cell immune against injure and deduce indirectly the forming of liver collagen protein.

排钱草具有抗肝纤维化作用,其机理可能与其能保护肝细胞免受损伤破坏,从而间接减少肝内胶原纤维形成有关。

The influence mechanism of highly fibrillated BKP on HYP performances was discussed. The bonding between the fibers is well improved by a special bridge-link network structure formed by BKP fiber whiskers and HYP fibers, which greatly raises the strength properties of HYP.

探讨了高度细纤维化的BKP影响HYP性能的机理,发现高度细纤维化的BKP与HYP纤维形成的特殊桥联网状结构能很好地改善纤维间的结合,提高了HYP的机械强度。

There were more fibroblasts, the collagen increased in model control group. After 30 days, the fibroblasts of model control group were hyperemic, scattered red blood cells could be found, and capillaries were proliferated. Fibrous tissue arranged in rows in the acupotomology intervention group and the blockade control group.

造模后30 d模型对照组成纤维细胞增生,可见红细胞散在分布,有增生的毛细血管;针刀干预组与封闭对照组可见纤维组织排列成行;针刀干预组较其他各组胶原纤维形成减少,瘢痕面积小。

Then introduced a new theory on forming super high-intensity wood-based panel with wood fiber reconsolidated micron technology, at the same time analysed its advantages, discussed the practicability of developing the new panel-MLFB.

回顾了纤维形成方法及纤维板发展历程,分析了传统纤维板形成理论的缺陷,介绍了微米长纤维高强度低密度人造板的形成理论,分析并阐述了该板种的优势,论述了开发研制这一新板种的可行性。

The research presented here is aimed at exploring the effect of DTT on the in vitro fibril formation of hen egg-white lysozymes with four disulfide bonds. In this work, we specifically addressed the effect of DTT concentration under different incubation temperatures on the hen lysozyme structure and fibril formation, the effect of addition of DTT at later time points on the amyloid fibrillization of hen lysozymes, and the effect of concentration ratio between reduced and oxidized forms of DTT on the fibril formation of lysozymes.

本研究以具有四对双硫键的母鸡蛋白溶菌酶为蛋白质模式系统,在含盐类之酸性溶液环境下诱导生成类淀粉纤维,再配合二硫代苏糖醇破坏溶菌酶中的双硫键,探讨:(1)不同温度下加入不同DTT浓度对溶菌酶类淀粉纤维形成之影响,(2)不同生长时期加入DTT对溶菌酶类淀粉纤维形成之影响,(3)不同比例之DTT和氧化态DTT浓度对溶菌酶类淀粉纤维形成之影响。

This study was conducted to examiune the fibrotic effect of Ni-Ti and 317L al loys in esophagus.The extract fluid from Ni-Ti,317L alloys was made according t o the ASTM standards of U.S.A. The Fb of esophageal scar was cultured primarily ,then incubated with alloy abstract fluid. The proliferating activity of Fb was measured by MTT at 4, 24, 48, 72 hours in the course of culturing. The esophagu s embedding test of Ni-Ti,317L alloys was made according to ASTM standards of U .S.A.The tissue around the alloys was taken at weeks 2 and 12,and the pathologi c changes were analysed.

为探讨新型支架材料Ni-Ti、317L合金在食管局部的致纤维化作用,按美国ASTM标准制备NiTi、317L合金的金属浸提液;&组织块培养法&原代培养食管壁疤痕的成纤维细胞,传代后以金属浸提液进行培养,分组后分别培养4、24、48、72 h,MTT法检测不同培养时间后Fb增殖功能的变化;按美国ASTM标准进行NiTi、317L合金试件的食管壁内包埋实验,即将金属试件经表面处理后直接置入食管壁粘膜层与肌层之间,术后2、12周取出包埋组织,分析试件周围组织的病理变化,并进行胶原纤维染色,观察纤维形成状况。

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