纤维化的

Hep-atic fibrosis is apart of"liver amasser"in the category of TCM.Whereas father doctors advanced that amasser can be get rid of by the way of"maintaining positive matter",to probe into the advantage and foreground of curing hepatic fi-brosis by the ways of FUZheng of TCM.

肝纤维化在中医属于"肝积"范畴，鉴于前辈医家提出"养正积自除"的理论，笔者就中医使用扶正益气培本的方法治疗肝纤维化优势及其前景展开探讨。

ResultsMacroscopic examination showed no excrescence,thrombus formation,arm fractures and corrosion.The devices were covered with collagen fibrosis and discrete endocardial cells,apparent inflammatory infiltration in the devices and around the devices 1 month after implantation.The implants were nearly endothelialized,while the inflammatory reaction relieved gradually,with myocardial cells ingrowth at the edges of the device 3 months after implantation.The devices were completely covered with endocardium and fibrous tissue.Moreover,endothelial cells could be found on the smooth microscrew adaptor.The inflammatory reaction diminished with a few chronic inflammatory cells existing.Neovascularization and lymphatic vessels ingrowth could be observed 6 months after implantation.

结果所有封堵装置表面均没有发现赘生物、血栓形成、支架发生断裂及被腐蚀；术后1个月，封堵装置表面被胶原纤维和散在内皮细胞所覆盖，大量炎症细胞浸润，封堵装置边缘有小灶性炎症细胞浸润；术后3个月，封堵装置表面几乎被内皮细胞所覆盖，炎症细胞较1个月时明显减少，封堵装置内见纤维化，封堵装置边缘心肌细胞浸入；术后6个月，封堵装置表面完全被心内膜和纤维组织所覆盖，伞尖表面光滑并有内皮细胞上爬，炎症反应明显消散，但仍有少量慢性炎症细胞存在，装置内有新生的血管、淋巴管长入。

MMP-3 proenzyme protein expression in the different stages of liver fibrosis was measured by half quantitative analysis of Western blot.

建立四氯化碳中毒性大鼠肝纤维化模型，采用半定量Western免疫印迹方法，对肝纤维化不同阶段肝内MMP - 3的酶原蛋白的表达水平进行了检测。

Abstract］ Objective To study the effect of zinc protoporphyrin,an inhibitor of heme oxygenase-1（HO-1）,on pathological and pathophysiologic changes of rat pulmonary fibrosis induced by bleomycin,and to investigate the role of Znpp in the pathogenesis of pulmonary fibrosis.

目的 观察血红素加氧酶-1（HO-1）抑制剂锌原卟啉对博莱霉素致大鼠肺纤维化模型病理和病理生理改变的影响，探讨Znpp在BLM诱导肺纤维化发病中的作用。

Results Beginning from alveolitis, PF in different diseases has similar pathological changes in a progressive and reparative process.

结果不同病种的肺间质纤维化性改变都是从肺泡炎开始，在发展和修复过程中导致的肺纤维化倾向有共同之处。

Objective To investigate the effects of Qing Gan Yin on collagen fibers and reticular fibers in hepatic fibrotic rats.

目的 研究中药清肝饮对大鼠肝纤维化模型胶原纤维和网状纤维的影响。

Here we show that senescent cells accumulate in murine livers treated to produce fibrosis, a precursor pathology to cirrhosis.

在这里我们展示在鼠类的肝脏中堆积衰老细胞用于使肝脏纤维化，肝脏纤维化是肝硬化的前驱病理改变。

Explore JIAWEISANRENTANG Decoction on liver fibrosis in rat tissue plasminogen activator inhibitor,tissue-type plasminogen activator impact.

探讨加味三仁汤对肝纤维化大鼠组织纤维酶原激活剂抑制剂、组织型纤维酶原激活剂的影响。

The alveolitis that produces fibroblast proliferation and collagen deposition is progressive over time.

肺间质发生成纤维细胞的增生，成纤维细胞可以分泌大量的胶原纤维，最终导致了肺间质纤维化。

Results The results showed that fibroblasts were the main source of extracellular matrix production in bile duct wall. The phenotype of fibroblasts in inflammatory strictured bile duct wall changed obviously, quiescent fibroblasts were activated and transformed to myofibroblasts, with massive proliferation.

结果 成纤维细胞是胆管壁中合成胶原的主要细胞；炎性狭窄胆管壁的纤维化增厚明显，其成纤维细胞表型改变，由相对静止转化为功能活跃状态，并进一步向肌成纤维细胞转化，且大量增殖。

We have no common name for a mime of Sophron or Xenarchus and a Socratic Conversation; and we should still be without one even if the imitation in the two instances were in trimeters or elegiacs or some other kind of verse--though it is the way with people to tack on 'poet' to the name of a metre, and talk of elegiac-poets and epic-poets, thinking that they call them poets not by reason of the imitative nature of their work, but indiscriminately by reason of the metre they write in.

索夫农 、森那库斯和苏格拉底式的对话采用的模仿没有一个公共的名称；三音步诗、挽歌体或其他类型的诗的模仿也没有——人们把&诗人&这一名词和格律名称结合到一起，称之为挽歌体诗人或者史诗诗人，他们被称为诗人，似乎只是因为遵守格律写作，而非他们作品的模仿本质。

The relationship between communicative competence and grammar teaching should be that of the ends and the means.

交际能力和语法的关系应该是目标与途径的关系。