纤维化的

Con2clusion : The serum TNF -αand IL - 6 levels might be related closely with liver fibrosis and participate in fibrosis formation and development.

血清TNF -α和IL - 6 水平与肝纤维化程度密切相关，TNF -α和IL - 6 参与了肝纤维化的形成和发展。

Demonstrative green tea can reduce gland purine to be damaged to the qualitative pathology between bandicoot kidney, green tea has fight the action that qualitative fiber changes between kidney, and can restrain hyperplasia of fibrous of I,Ⅲ collagen, this may be green tea qualitative fiber is changed between kidney of prevention and cure, one of mechanisms that defer chronic kidney failure to develop a process.

说明绿茶能够减轻腺嘌呤对大鼠肾间质的病理损害，绿茶有抗肾间质纤维化的功能，并且能抑制I、Ⅲ型胶原纤维的增生，这可能是绿茶防治肾间质纤维化、延缓慢性肾衰竭发展进程的机制之一。

The pathological changes of liver were also improved in the treatment group.conclusionit implies that the extract of retinervus luffae fructus and carapax trionyei is beneficial for the prevention and treatment of hepatic fibrosis in balb/c rats.

肝组织病理观察结果显示复方针剂能缓解肝纤维化的发生和发展，并可加快降解增生的纤维组织。结论鳖甲复方针剂对con a在balb/c雄性小鼠中诱导肝纤维化有一定预防和治疗作用。

Then we discuss the equivariant fibration and several results about the ordinary fibration are generalized to the equivariant category.

此后，我们对等变纤维化进行了研究并将拓扑空间范畴中纤维化的一些结果推广到了等变拓扑空间范畴。

The unniquess result on homogeneous special Lagrangian fibration with singular central fibers is proven, which says that special lagrangian fibration with homogeneous isolated singularity is essentially the model of Harvey-Lawson.

证明了当特殊拉格朗日纤维化在孤立奇点附近是代数的，则奇异性是唯一的。这个纤维化在孤立奇点的结构本质上是Harvey-Laweon在1982年给出的模型。这个结果在纤维化的奇异结构的研究中是十分重要的。

The influence mechanism of highly fibrillated BKP on HYP performances was discussed. The bonding between the fibers is well improved by a special bridge-link network structure formed by BKP fiber whiskers and HYP fibers, which greatly raises the strength properties of HYP.

探讨了高度细纤维化的BKP影响HYP性能的机理，发现高度细纤维化的BKP与HYP纤维形成的特殊桥联网状结构能很好地改善纤维间的结合，提高了HYP的机械强度。

2TGF-β1 and PDGF-B mRNA in liver tissue of liver-fibrotic rats can express increasedly, TGF-β1 and PDGF-B relate with the happening and developing of liver fibrosis closely.

肝纤维化大鼠肝组织TGF一p，和PDGF一B mRNA表达增加，TGF一pl和PDGF一B与肝纤维化的发生发展关系密切。

On experimental day , the rats were intratracheally instilled with bleomycin(5mg/kg body weight)or sterile saline, and then treated with the Fluvastain and saline until they were killed .5 rats of every group were executed in 7,14,28,42 day. Immunohistochemistry and Westernblotting were used to measure the expression of TGF-β, ERK_(1/2) p-ERK protein.

本实验利用博莱酶素诱导大鼠肺间质纤维化，以氟伐他汀为代表研究他汀类药物对肺纤维化大鼠模型的干预作用，并通过观察氟伐他汀对ERK信号通路中重要调节酶ERK_(1/2)的表达及活化的影响，探索肺纤维化的信号传导机制，并为临床提供新的治疗思路。

The mechanism of action maybe include the depression in content of collagen, inhibition to proliferation of lung fibroblast, action of anti free redical and anti lipid peroxidation, and elevation of content of GSH-PX.So we think phenol total base of plumula nelumbinis is a sort of promising drug to healing PF.

综上所述，我们认为莲子心酚性总碱可能对肺纤维化的病理过程有多靶点作用：前期通过抗自由基作用，从而减轻肺泡炎损伤：后期直接抑制肺成纤维细胞的增殖及蛋白沉积，减轻纤维化病变程度。

Objective: To observe the effect of Zhenggan Huangu recipe on the expressions of MMPs and TIMPs in the hepatic tissues of rats with hepatic fibrosis and probe its mechanism of anti-hepatic fibrosis.

目的：观察正肝化瘀方对肝纤维化大鼠肝组织MMPs和TIMPs蛋白表达的影响，探讨其抗肝纤维化的作用机制。

Lugalbanda was a god and shepherd king of Uruk where he was worshipped for over a thousand years.

Lugalbanda 是神和被崇拜了一千年多 Uruk古埃及喜克索王朝国王。

I am coming just now,' and went on perfuming himself with Hunut, then he came and sat.

我来只是现在，'歼灭战perfuming自己与胡努特，那麼，他来到和SAT 。