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红细胞破坏的

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OBJECTIVE In order to reduce the damage of negative pressure from cardiotomy suction on red blood cell and endocardium during extracorporeal circulation, we developed a cardiotomy suction relief valve and applied clinically.

目 的为了减轻体外循环心内吸引力过大,所产生的负压对红细胞和心内膜的破坏,将研制的一种心内吸引减压阀我们在临床试用。

A lot of red cells break and a lot of liver cells degeneration and necrosis were the main causes of the icterus.

结论]红细胞的大量破坏和肝细胞的大量变性、坏死是导致黄疸发生的主要原因。

OBJECTIVE In order to reduce the damage of negative pressure from cardiotomy suction on red blood cell and endocardium during extracorporeal circulation, we developed a cardiotomy suction relief valve and applied clinically.

作者:乔建民,高立兵,王委三目的为了减轻体外循环心内吸引力过大,所产生的负压对红细胞和心内膜的破坏,将研制的一种心内吸引减压阀我们在临床试用。

Multiple factors participated in the process of secondary brain edema and nerve injuries,such as the hydrostatic pressure during formation of the hematoma and clot retraction, activation of blood coagulation cascade, generation of thrombin,erythrocytolysis and the toxicity of hemoglobin, the activation of the complement system, space-occupying effects, rupture of the blood-brain barrier,secondary ischemia around the hematoma, inflammatory reaction of the neurons around hematoma and cell apoptosis.

脑出血后继发水肿及神经损害有多种因素参与,血肿形成过程中的流体静力压和血凝块的回缩、凝血瀑布的激活、凝血酶的产生、红细胞溶解和血红蛋白的毒性、补体的激活、占位效应、血脑屏障的破坏、血肿周围继发缺血、血肿周围组织神经细胞炎症反应及细胞凋亡等。

After administration of L-N〓-nitro-Arginine, a specific antagonist of NOS, it was found that the basal and stimulated NO release of blood vessels (aorta, internal carotid, renal and mensenteric artery) was decreased further. The increase of coronary blood flow stimulated by Ach was also reduced further, while the platelet aggregation degree and erythrocyte aggregation index increased significantly. Pathological examination indicated that aortic endothelium was destroyed, the blood vessels in cerebral, cardiac and renal tissues were hypertrophied further and some were almost occluded. The above fin dings resulted in SHR feeded with L-NNA having high incidence of stroke rate and high degree of renal cirrhosis when compared to control SHR. The infarct and hemorrhagic focuci in cerebral tissues, infarct focuci in cardiac tissues, atrophy and hyalinedegeneration of renal glomeruli and degeneration even necrosis of renal tubuli in renal tissues were al so found. The increase ratio of brain and heart to body weight and decrease ratio of kidney to body wei ght further demonstrated that the cerebral, cardiac and renal injuries were aggravated.

在上述结果的基础上进一步给予SHR NOS特异性拮抗剂左旋亚硝基精氨酸后,血管(胸主、颈内、肾及肠系膜动脉)NO基础释放及激动剂刺激释放进一步减少,Ach刺激冠脉流量的增加和胸主动脉内膜NOS活性也进一步减少,血小板聚集程度及红细胞聚集指数显著上升,病理切片显示胸主动脉内皮细胞破坏明显,脑心肾组织内的血管进一步增厚甚至几乎阻塞,导致饲L-NNA的SHR缺血性及出血性脑卒中发生率显著高于对照SHR,脑体重比显著升高;心肌内出现明显的缺血梗塞疤痕灶,心体重比显著升高;肾小球明显萎缩、玻璃样变性,肾小管也变性甚至坏死,使肾硬化程度显著增加呈颗粒性固缩肾,肾体重比也显著减少。

As with red cells and platelets, these transfusions should be irradiated prior to transfusion, but should not be treated with leukoreduction filters, as this would defeat the purpose of transfusing white cells.

与红细胞和血小板输注一样,粒细胞输注前也必须进行辐射处理,但不能经过减白细胞过滤,因为这样会破坏白细胞输注的目的。白细胞通过标准血液过滤器,除去其中的颗粒物和凝结的血液成分,但保留白细胞。

Haemolysis test, prothrombin time and recalcification time experiment were used to evaluate influence on erythrocyte and blood coagulation function by CLG microspheres.

通过溶血实验、凝血酶原时间实验和复钙时间实验评价交联明胶微球对红细胞的破坏作用和对凝血功能的影响。

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