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Methodological Mao Xi is bronchitic acute period little patient 153 , check-up of the corresponding period is healthy 103 , use full automatic hematic cell analyzer undertakes outside week plaque and red blood cell (of the parameter such as RBC, HGB, HCT, MCV, MCH, MCHC) determine.

方法毛细支气管炎急性期患儿153例,同期体检健康儿103例,采用全自动血细胞分析仪进行外周血血小板(PLT、PCT、MPV、PDW)和红细胞(RBC、HGB、HCT、MCV、MCH、MCHC)等参数的测定。

Hillyer and colleagues observed an external K~+ level of 57.0mmol/L in RBCs units irradiated with 35Gy on Day 42 postirradiation.

Hillyer和他的同事发现35Gy辐照红细胞储存42天后,细胞外钾离子浓度达到了57.0 mmol/L。

Methods Red blood cell indices and reticulocyte indices of peripheral blood were determined on ADVIA 120 hematology analyzer.

方法用BayerADVIA120全自动血液分析仪对236例健康人群、101例非IDA患者和78例IDA患者的外周血红细胞和网织红细胞诸参数进行了检测,并对检验结果进行了对比分析。

2B8a was weakly reactive to neutrophils (23.72%) and negative for T cells, NK, DC, RBC and Plt. The antibody reacted to all 3 marrow CD34+ cells with an average positive rate of 39.33% while it was negative for G-CSF-mobilized CD34+ peripheral blood stem/progenitor cells (PBSC, 1.25%). Cell line analysis showed that the antibody notably reacted to three out of 4 cell lines (Raji, SMS-SB, Nalm-6 and Nall-1) with the positive rates of 98.78%, 98.61%, 94.93% respectively and weakly to one of them with 5.68% in B lineage cell lines and monoblastic cell line (U937, 67.78%) while it was only weakly positive or negative for other myeloid leukemia cell lines including Meg01 (33.40%), HL-60 (29.70%),K562 (28.19%), KG1a (16.23 %) and HEL92.1.7 (8.02%). Among 4 T lineage leukemia,5 neuroblastoma and 1 colon cancer cell lines tested, only Molt-3 was found weakly positive (31.40%) for 2B8a, while the remaining 3 T cell lines (Molt4, JM and CCRF-CEM), 5 neuroblastoma cell lines (LA-N1, KCNR, BE, SK-N-SH, SK-N-AS) and the colon cancer cell line (HR8348) tested were negative.

结果表明: 2B8a抗原在外周血B细胞上表达(3/3例,平均阳性细胞数为26.29 %),而在T淋巴细胞和NK细胞上不表达(0/3例);在粒细胞和单核细胞上阳性表达均为2/3例,平均阳性细胞数分别是23.72 %和59.84 %;在DC细胞、红细胞和血小板上均不表达(0/3例)。2B8a抗原在骨髓CD34+细胞上的阳性表达是3/3例,平均阳性细胞数39.33 %,而在G-CSF动员的外周血CD34+细胞上的阳性表达仅1/3例,平均阳性细胞数为1.25 %。2B8a抗原在B系细胞系Raji、SMS-SB、Nalm-6和Nall-1上的平均阳性细胞数分别为98.78 %、98.61 %、94.93 %和5.68 %;在T系细胞系Molt-3上的平均阳性细胞数为31.40 %,而在Molt-4、JM和CCRF-CEM 细胞上不表达;在髓系细胞系U937、Meg-01、HL-60、K562、KG1a和HEL92.1.7上的平均阳性细胞数分别为67.78 %、33.40 %、29.70 %、28.19 %、16.23 %和8.02 %;在神经母细胞瘤细胞系SK-N-SH、KCNR、BE、LAN-1和SK-N-AS细胞以及结肠癌细胞系HR8348细胞上均不表达,而在羊膜细胞系FL细胞上呈一定的阳性表达,平均阳性细胞数为45.03%。

Cell line analysis showed that the antibody notably reacted to three out of 4 cell lines (Raji, SMS-SB, Nalm-6 and Nall-1) with the positive rates of 98.78%, 98.61%, 94.93% respectively and weakly to one of them with 5.68% in B lineage cell lines and monoblastic cell line (U937, 67.78%) while it was only weakly positive or negative for other myeloid leukemia cell lines including Meg01 (33.40%), HL-60 (29.70%),K562 (28.19%), KG1a (16.23 %) and HEL92.1.7 (8.02%). Among 4 T lineage leukemia,5 neuroblastoma and 1 colon cancer cell lines tested, only Molt-3 was found weakly positive (31.40%) for 2B8a, while the remaining 3 T cell lines (Molt4, JM and CCRF-CEM), 5 neuroblastoma cell lines (LA-N1, KCNR, BE, SK-N-SH, SK-N-AS) and the colon cancer cell line (HR8348) tested were negative.

结果表明: 2B8a抗原在外周血B细胞上表达(3/3例,平均阳性细胞数为26.29 %),而在T淋巴细胞和NK细胞上不表达(0/3例);在粒细胞和单核细胞上阳性表达均为2/3例,平均阳性细胞数分别是23.72 %和59.84 %;在DC细胞、红细胞和血小板上均不表达(0/3例)。2B8a抗原在骨髓CD34+细胞上的阳性表达是3/3例,平均阳性细胞数39.33 %,而在G-CSF动员的外周血CD34 细胞上的阳性表达仅1/3例,平均阳性细胞数为1.25 %。2B8a抗原在B系细胞系Raji、SMS-SB、Nalm-6和Nall-1上的平均阳性细胞数分别为98.78 %、98.61 %、94.93 %和5.68 %;在T系细胞系Molt-3上的平均阳性细胞数为31.40 %,而在Molt-4、JM和CCRF-CEM 细胞上不表达;在髓系细胞系U937、Meg-01、HL-60、K562、KG1a和HEL92.1.7上的平均阳性细胞数分别为67.78 %、33.40 %、29.70 %、28.19 %、16.23 %和8.02 %;在神经母细胞瘤细胞系SK-N-SH、KCNR、BE、LAN-1和SK-N-AS细胞以及结肠癌细胞系HR8348细胞上均不表达,而在羊膜细胞系FL细胞上呈一定的阳性表达,平均阳性细胞数为45.03%。

In peripheral blood,〓increases, so cause 〓 balance is broken, and 〓 which can induce inflammatory reaction increases obviously too; meanwhile, there are immuno- function change in the red cells, the RBC-〓 receptor rosette promoting rate of serum decrease, indicate this uveitic animal model have both cellular immunity dysfunction induced by T cells and the changes of RBC immuno-function. The analysis of the local and systemic immuno-function changes of uveitic animal model offers objective theoretical bases for combining the local treatment and the systemic treatment.

葡萄膜炎兔房水中房水中〓及IL-2均升高,病变葡萄膜组织中T细胞表面分化抗原的免疫组化检查显示大量阳性细胞,表明兔葡萄膜组织本身存在着免疫功能亢进所导致的病理改变,而外周血中〓增高,导致〓平衡失调,介导炎症反应的〓亦明显增高,同时伴有红细胞免疫功能改变,外周血中RBC-〓受体花环率及血清中RBC-〓受体花环促进率均降低,显示葡萄膜炎兔本身既存在T细胞介导的细胞免疫反应功能紊乱,也存在红细胞免疫功能改变,这种局部和全身的免疫功能改变分析为葡萄膜炎的治疗局部和全身相结合提供了客观的理论依据。

Methods Peripheral blood was collected from 20 healthy persons and 35 patients who underwent curative gastrectomies. The expression of CCR7 in the lymphocytes was determined by flow cytometry after red cells had died and labeled with fluorescence mAb.

分别收集健康人组、胃癌患者术前和术后外周血,进行荧光标记和红细胞裂解后,流式细胞仪检测CCR7在外周血淋巴细胞各亚群的表达,并分析其与临床病理分期之间的相关性。

Some cell dropped into the cavity and became free. Thrombosis or part organization could be seen. The internal elastic layer became thin, disappear or broken. In internal and middle layer existed fibroblasts, fibrocytes and collagen. Some of the wall indicated hyaline change, soomth muscle cell decreased greatly. The massive inflammatory cells invaded the middle and external layer. There were many foam cells in the capsule tissue. Cytoplasm was filled with fatty tissue and cholesterol. some cavities were full of thrombosis. Some thrombosis was fibrosis, the bottom was organization. The surface of the thrombosis existed red blood cell and librae.(2)Elatic fibrila staining: the internal elastic menbrane almost completely disappeared, the intact internal elastic menbran could be seen in the new small vessels.

动脉瘤管壁厚薄明显不均,全层或局部区域显著变薄向外膨出,内皮细胞空泡变性或坏死脱落,部分内皮细胞剥离并突入管腔成游离状,可见血栓形成及部分血栓机化;内弹力板变薄、消失或突然中断;在内膜及中膜部位主要为纤维母细胞、纤维细胞和大片胶原;部分动脉瘤壁呈均质状玻璃样变,平滑肌细胞明显减少;中膜和外膜可见大量的炎性细胞浸润;瘤壁组织有纤维母细胞、纤维细胞、大片胶原成分及较多泡沫细胞,胞浆内充满脂类物质及胆固醇结晶;部分动脉瘤腔内充满血栓,有的血拴已经纤维化,血栓基部机化,血栓表面有红细胞和纤维素。

The general information,including primary diseases,therapeutic drugs,infection pathogens in 6 patients with pulmouary infection and unknown reason anemia were integrated and analyzed. The leukocyte and reticulocyte counts,total bilirubin,direct bilirubin,fasting blood glucose were all determined by conventional method. At the same time,the erythrocyte direct antiglobulin test,complement fixation test,cell culture and smear examination were carried out.

对2004年6例肺部感染且不明原因贫血分析的患者的基础疾病、治疗用药、感染病原体等消息进行归纳,对患者外周血进行白细胞计数、网织红细胞计数、分胆红素、间接胆红素、血糖检测,对患者的红细胞进行间接抗人球蛋白试验、补体结合实验、细胞培养与涂片镜检,对患者血浆中的抗体进行间接抗人球蛋白试验。

The general information,including primary diseases,therapeutic drugs,infection pathogens in 6 patients with pulmouary infection and unknown reason anemia were integrated and analyzed. The leukocyte and reticulocyte counts,total bilirubin,direct bilirubin,fasting blood glucose were all determined by conventional method. At the same time,the erythrocyte direct antiglobulin test,complement fixation test,cell culture and smear examination were carried out.

对2004年6例肺部感染且不明原因贫血分析的患者的基础疾病、治疗用药、感染病原体等信息进行归纳,对患者外周血进行白细胞计数、网织红细胞计数、总胆红素、直接胆红素、血糖检测,对患者的红细胞进行直接抗人球蛋白试验、补体结合实验、细胞培养与涂片镜检,对患者血浆中的抗体进行间接抗人球蛋白试验。

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