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红细胞增多症

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Blood disease, such as anemia, leukemia and polycythemia vera disease and various out, coagulation disorders.

血液病患者,如贫血、白血病、真性红细胞增多症及各种出、凝血性疾病。

objective:The Bloodletting treatment of polycythemia vera for observing and caring accordingly.

R554B1005-2720(2009)06-0010-02目的:阐述对真性红细胞增多症放血疗法的病情观察和相应的护理。

Methods: Fortysix patients with postrenal transplant polycythemia were randomly divided into two groups: treatment group (n=29) and control group (n=17) received low (5 mg/d) and usual dose (20 mg/d) of enalapril, respectively.

46例肾移植后红细胞增多症患者随机分为两组:小剂量组(n=29)和常规剂量组(n=17)。

A patient in shock, who had been given a transfusion in excess of the normal capacity of his circulatory system, sometimes developed polycythemia.

一个休克病人,如果得到大于他血液循环系统的正常容量的输血,有时会发展成红细胞增多症

Participants included cases of polycythemia vera from the 2001 to 2005 state cancer registry as well as self- and physician-referred cases.

参与者包括从2001年至2005年国家肿瘤登记以及自我就诊和医生转诊的真性红细胞增多症病例。

Objective To investigate the brain flood flow changes before and after exchange transfusion in neonatal polycythemia.

目的 探讨新生儿红细胞增多症换血前后脑血流变化。

The close proximity of this cluster to known areas of hazardous material exposure raises concern that such environmental factors might play a role in the origin of polycythemia vera.

这一簇群与已知危害物质暴露区域的紧密邻近,引起了关注,即这些环境因素可能在真性红细胞增多症的起病中发挥作用。

These cases included 55 cases of ITP,33 cases of HS,5 cases of MA,5 cases of PHT with splenic function accentuation,3 cases of splenic cyst,1 case of splenic abscess,3 cases of splenic angioma,2 cases of splenic angiolymphoma,2 cases of accessory spleen,and 1 case of splenic rupture.

病例构成为原发性血小板减少性紫癜(idiopathic thrombocytopenic purpura,ITP)55例,遗传性球形红细胞增多症(hereditary spherocytosis,HS)33例,地中海贫血(mediterranean anemia,MA)5例,肝硬化门脉高压脾亢5例,脾囊肿3例,脾脓肿1例,脾血管瘤3例,脾淋巴管瘤2例,副脾2例,脾破裂1例。

RESULTS: The Bcl-x deamidation pathway was inhibited in myeloid cells, but not T cells, in patients with CML or polycythemia vera.

结果:CML患者或真性红细胞增多症患者的Bcl -×脱酰胺途径受抑制是在髓系细胞中,而不是T细胞。

BCR-ABL and mutant JAK2 inhibit the Bcl-x deamidation pathway and the apoptotic response to DNA damage in primary cells from patients with CML or polycythemia vera.

BCR-ABL和突变的JAK2可抑制CML或真性红细胞增多症患者Bcl -×的脱酰胺途径和原代细胞DNA损伤诱导的凋亡反应。

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