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红细胞减少的

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In the experiment, an approach was made to the cause of DRP and the mechanism of it's prevention and treatmend by acupuncture. The experimental results indicated that in rat with diabetes,(1) plasma TXB2 and the ratio of TXB2 to 6-keto-PGF1α increased obviously, suggesting hyperfunction of platelet and injury to endothelial function, which tend to result in thrombosis;(2) erythrocytic filtration index increased remarkably, suggesting erythrocytic transforming and oxygen carring hypofunction, which is one of the factors leading to retinal anoxia;(3) hyperglycemia persisted, which is related to the thickening of basilar membrane of and the decrease of mural cell of retinal blood vessels;(4) serum growth hormone rose notably, which is not only one of the important factors causing basilar membrane thickening but also promotes the synthesis or Von Willebrand Factor that participates in a tendency to thrombosis.

本实验对糖尿病性视网膜病变(Diabetic Retinopathy,DRP)的成因以及针刺防治DRP的作用机理进行了探讨,实验结果表明:糖尿病大鼠(1)血浆TXB2及TXB2/6-ket0-PGF1α比值均显著升高,提示其血小板机能亢进,内皮细胞功能损伤,具有血栓形成倾向;(2)红细胞滤过指数(Filtration Index,IF)显著增高,提示其红细胞变形能力低下,携氧运行功能降低,是造成视网膜缺氧状态的因素之一;(3)呈持续高血糖状态,与视网膜血管基底膜增厚及管壁细胞减少有关;(4)血清生长激素(Growth Hormone,GH)水平明显升高,既是导致基底膜增厚的重要因素之一,又促进VWF合成,参与血栓形成倾向。

The numbers of RBC in low Se group and MCH in high Se group were apparently decreased.

低硒组大鼠的红细胞数和高硒组大鼠的平均红细胞血红蛋白含量显著减少。

This further results in low red cell counts.

这更进一步会导致红细胞的减少。

There were more fibroblasts, the collagen increased in model control group. After 30 days, the fibroblasts of model control group were hyperemic, scattered red blood cells could be found, and capillaries were proliferated. Fibrous tissue arranged in rows in the acupotomology intervention group and the blockade control group.

造模后30 d模型对照组成纤维细胞增生,可见红细胞散在分布,有增生的毛细血管;针刀干预组与封闭对照组可见纤维组织排列成行;针刀干预组较其他各组胶原纤维形成减少,瘢痕面积小。

Objective A clinical study of Zusanli(ST35)acupoint injectiontherapy with Dexamethasonum and Adenosine triphosphate coordination of routine western medicine,on leukopenia,hypoglobulia and hypochromia count in patients with cancer after chem-or radiotherapy were observed.

目的 研究用足三里穴位注射地塞米松和ATP配合药物治疗恶性肿瘤患者放化疗后引起的白细胞、红细胞和血红蛋白总数减少症。

In addition, the study mainly used methods of pharmacology and molecular biology and the results showed as:Firstly, TSSO can meliorate hypocytosis induced by radiation and chemical, improve the count of the WBC, count of the RBC and count of the PLT significantly.

结果表明: 1。地榆总皂苷对放、化疗致血细胞减少症有显著的改善作用,能显著回升白细胞计数、红细胞计数、血小板计数等血象指标。

Smear diagnostic cell measures true sex to reduce disease or verdict (aplastic anemia and hematopoiesis bring down) false positive is respectively 29.0% with 37.1%, diagnose true sex to increase a disease (lienal function hyperfunction, special disease of disease of grow in quantity of the plaque that send a gender, grow in quantity of true sex red blood cell) false negative rate is 22.6% with 21.8%, diagnostic index performance is evaluated all not beautiful.

涂片诊断细胞量真性减少疾病或结论(再生障碍性贫血和造血减低)的假阳性分别为29.0%和37.1%,诊断真性增加疾病(脾功能亢进、特发性血小板增多症、真性红细胞增多症等)假阴性率为22.6%和21.8%,诊断指标性能评价均不佳。

Presbycusis might result from the decreased SOD and RCD,and the increased MDA in some extent.

老年性耳聋病人的SOD含量下降,红细胞变形能力降低,而丙二醛水平增高,建议在临床上对老年性耳聋病人应用增加SOD含量,提高红细胞变形能力,减少MDA含量的药物。

Results:The intramembrane proteinic particles of cell membranes of erythrocyte as well as of muscular cells were diminished significantly in DMD.

结果:DMD患者红细胞与肌细胞一样,其细胞膜中的蛋白颗粒显著减少。

After administration of L-N〓-nitro-Arginine, a specific antagonist of NOS, it was found that the basal and stimulated NO release of blood vessels (aorta, internal carotid, renal and mensenteric artery) was decreased further. The increase of coronary blood flow stimulated by Ach was also reduced further, while the platelet aggregation degree and erythrocyte aggregation index increased significantly. Pathological examination indicated that aortic endothelium was destroyed, the blood vessels in cerebral, cardiac and renal tissues were hypertrophied further and some were almost occluded. The above fin dings resulted in SHR feeded with L-NNA having high incidence of stroke rate and high degree of renal cirrhosis when compared to control SHR. The infarct and hemorrhagic focuci in cerebral tissues, infarct focuci in cardiac tissues, atrophy and hyalinedegeneration of renal glomeruli and degeneration even necrosis of renal tubuli in renal tissues were al so found. The increase ratio of brain and heart to body weight and decrease ratio of kidney to body wei ght further demonstrated that the cerebral, cardiac and renal injuries were aggravated.

在上述结果的基础上进一步给予SHR NOS特异性拮抗剂左旋亚硝基精氨酸后,血管(胸主、颈内、肾及肠系膜动脉)NO基础释放及激动剂刺激释放进一步减少,Ach刺激冠脉流量的增加和胸主动脉内膜NOS活性也进一步减少,血小板聚集程度及红细胞聚集指数显著上升,病理切片显示胸主动脉内皮细胞破坏明显,脑心肾组织内的血管进一步增厚甚至几乎阻塞,导致饲L-NNA的SHR缺血性及出血性脑卒中发生率显著高于对照SHR,脑体重比显著升高;心肌内出现明显的缺血梗塞疤痕灶,心体重比显著升高;肾小球明显萎缩、玻璃样变性,肾小管也变性甚至坏死,使肾硬化程度显著增加呈颗粒性固缩肾,肾体重比也显著减少。

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