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红细胞减少

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Effect of dilute autotransfusion is better than hemodilution in minizing blood loss and preserving RBC.

稀释式自体输血能更有效地减少血液流失,保存红细胞。

The precipitates of alpha-globin chains in major beta-thalassemia erythroid cells were lessened after treatment with ASON,particularly in early erythroblast stage.There were no changes in those of control group.

此外,透射电镜结果也证实ASON组中红系细胞内过剩α-珠蛋白肽链沉积有所减少,特别是在早期幼红细胞中;在空白对照组中沉积物无任何变化。

Results The megakaryocyte and platelet of 112 cases hemopathy marrow smear with acetal phospholipids staining were all positive, the plasmas cell in anaemia were positive, the erythroblast of aplastic anemia positive, the lipocyte in thrombocytopenic purpura positive, and the other cells all negative.

结果]112例血液病骨髓涂片缩醛磷脂染色中的巨核细胞和血小板均为阳性,贫血中的浆细胞为阳性,再生障碍性贫血中的幼红细胞为阳性,血小板减少性紫癜中的脂肪细胞为阳性,其余各种细胞均呈阴性反应。

Drink plenty of water to reduce Hematocrit and Hemoglobin levels from raising too high from the Testosterone.

多喝水 ,以减少红细胞和血红蛋白水平的提高太高的睾酮。

In the experiment, an approach was made to the cause of DRP and the mechanism of it's prevention and treatmend by acupuncture. The experimental results indicated that in rat with diabetes,(1) plasma TXB2 and the ratio of TXB2 to 6-keto-PGF1α increased obviously, suggesting hyperfunction of platelet and injury to endothelial function, which tend to result in thrombosis;(2) erythrocytic filtration index increased remarkably, suggesting erythrocytic transforming and oxygen carring hypofunction, which is one of the factors leading to retinal anoxia;(3) hyperglycemia persisted, which is related to the thickening of basilar membrane of and the decrease of mural cell of retinal blood vessels;(4) serum growth hormone rose notably, which is not only one of the important factors causing basilar membrane thickening but also promotes the synthesis or Von Willebrand Factor that participates in a tendency to thrombosis.

本实验对糖尿病性视网膜病变(Diabetic Retinopathy,DRP)的成因以及针刺防治DRP的作用机理进行了探讨,实验结果表明:糖尿病大鼠(1)血浆TXB2及TXB2/6-ket0-PGF1α比值均显著升高,提示其血小板机能亢进,内皮细胞功能损伤,具有血栓形成倾向;(2)红细胞滤过指数(Filtration Index,IF)显著增高,提示其红细胞变形能力低下,携氧运行功能降低,是造成视网膜缺氧状态的因素之一;(3)呈持续高血糖状态,与视网膜血管基底膜增厚及管壁细胞减少有关;(4)血清生长激素(Growth Hormone,GH)水平明显升高,既是导致基底膜增厚的重要因素之一,又促进VWF合成,参与血栓形成倾向。

Vitamin C deficiency also resulted in anaemia, basically due to hypocythaemiam and hypochromia, and leukocytosis.

维生素C缺乏幼建鲤出现贫血和白细胞增多症,主要表现为红细胞数量和血红蛋白含量减少,白细胞数量增多。

The numbers of RBC in low Se group and MCH in high Se group were apparently decreased.

低硒组大鼠的红细胞数和高硒组大鼠的平均红细胞血红蛋白含量显著减少。

It suggested that the level of CD35、CD44 and CD62P on thrombocyte in cancer patients is increased,however,the decrease of CD35 and CD44 on erythrocyte in patients demonstrates immunity reduction of erythrocyte.

提示恶性肿瘤患者血小板CD35、CD44和CD62P分子处于高表达状态,而红细胞CD35、CD44分子表达减少,和红细胞免疫功能下降有关,这些变化可能对肿瘤的生长和转移具有重要意义。

Presbycusis might result from the decreased SOD and RCD,and the increased MDA in some extent.

老年性耳聋病人的SOD含量下降,红细胞变形能力降低,而丙二醛水平增高,建议在临床上对老年性耳聋病人应用增加SOD含量,提高红细胞变形能力,减少MDA含量的药物。

After administration of L-N〓-nitro-Arginine, a specific antagonist of NOS, it was found that the basal and stimulated NO release of blood vessels (aorta, internal carotid, renal and mensenteric artery) was decreased further. The increase of coronary blood flow stimulated by Ach was also reduced further, while the platelet aggregation degree and erythrocyte aggregation index increased significantly. Pathological examination indicated that aortic endothelium was destroyed, the blood vessels in cerebral, cardiac and renal tissues were hypertrophied further and some were almost occluded. The above fin dings resulted in SHR feeded with L-NNA having high incidence of stroke rate and high degree of renal cirrhosis when compared to control SHR. The infarct and hemorrhagic focuci in cerebral tissues, infarct focuci in cardiac tissues, atrophy and hyalinedegeneration of renal glomeruli and degeneration even necrosis of renal tubuli in renal tissues were al so found. The increase ratio of brain and heart to body weight and decrease ratio of kidney to body wei ght further demonstrated that the cerebral, cardiac and renal injuries were aggravated.

在上述结果的基础上进一步给予SHR NOS特异性拮抗剂左旋亚硝基精氨酸后,血管(胸主、颈内、肾及肠系膜动脉)NO基础释放及激动剂刺激释放进一步减少,Ach刺激冠脉流量的增加和胸主动脉内膜NOS活性也进一步减少,血小板聚集程度及红细胞聚集指数显著上升,病理切片显示胸主动脉内皮细胞破坏明显,脑心肾组织内的血管进一步增厚甚至几乎阻塞,导致饲L-NNA的SHR缺血性及出血性脑卒中发生率显著高于对照SHR,脑体重比显著升高;心肌内出现明显的缺血梗塞疤痕灶,心体重比显著升高;肾小球明显萎缩、玻璃样变性,肾小管也变性甚至坏死,使肾硬化程度显著增加呈颗粒性固缩肾,肾体重比也显著减少。

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