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METHODS: The oxidative damages of erythrocyte induced by CSE were evaluated through biochemical assays of malonaldehyde, methemoglobin and reduced glutathione levels, hemolytic rate, SDSPAGE of membrane proteins and chromatographic analysis of membrane phospholipids.

以市售香烟提取CSE,然后作用于红细胞,通过测定细胞溶血率、膜脂质过氧化水平、细胞内高铁血红蛋白以及还原谷胱甘肽含量、膜磷脂组成和膜蛋白谱变化等指标,对CSE导致的红细胞膜损害进行评估。

The transcription of PfRON4 peaks at mature schizont stage, suggesting that the PfRON4 gene may involve in erythrocyte-invasion of P.

定量分析结果显示,在恶性疟原虫红内期发育过程中, PfRON4基因的转录水平在裂殖子入侵红细胞后36~40 h达到高峰。

In addition, an oval protuberance in pars caeca retinae near pars optica retinae forms continuous lap, and hemal sinus and abundant erythrocytes exist in the protuberance. The homologous relationship may exist between the protuberance and conus papillaris and/or pectin.

视网膜盲部接近视部处有椭圆形突起,该突起呈连续分布而形成一圈,突起内可见血窦及丰富的红细胞,该突起可能与锥状突和/或栉膜具有同源关系。

In addition, an oval protuberance in pars caeca retinae near pars optica retinae forms continuous lap, and hemal sinus and abundant erythrocytes exist in the protuberance.

视网膜盲部接近视部处有椭圆形突起,该突起呈连续分布而形成一圈,突起内可见血窦及丰富的红细胞,该突起可能与锥状突和/或栉膜具有同源关系。

The partial nucleotide sequence of the gene fragment showed high homology with the essential spectrin gene of Caenorhabditis elegans and the erythrocyte surface antigen gene of Plasmodium falciparum , when the gene fragment was homologically analyzed in GenBank.

与GenBank中的基因进行同源性分析,其部分核苷酸序列与秀丽隐杆线虫红细胞膜内蛋白和恶性疟原虫红细胞表面抗原高度同源

Fibrin exudation, pulmonary interstitial incrassation, thrombosis of pulmonary artery, hemorrhage and gore were found. Conclusion: MR enhanced pulmonary perfusion can diagnose pulmonary embolism.

病理显示:梗死区可见大量的纤维素渗出,肺间质增厚,动脉腔内可见红细胞和血栓形成,其周围白细胞浸润伴肺淤血,部分可见点状出血及肺淤血。

There were no significant differences in chest tube drainage and red blood cell transfusion over the first 24 h postoperatively between patients with and without reported aspirin intake.

术后24小时内胸管引流量及输注红细胞在服用和未服用阿司匹林的患者间无显著差异。

Understand the carcinomatous metastasis of thyroid cancer curring by 131I , Polycythemia Vera curring by 32P, malignant pheochromocytoma curring by 131I—MIBG ,the indication and contraindication of β-therapy.

32P治疗真性红细胞增多症、胶体Cr32PO4腔内治疗131I—MIBG治疗恶性嗜铬细胞瘤以及β射线敷贴治疗的适应症和禁忌症。

Results LPS exposure, which had no influence on mean arterial pressure, induced a significant increase in the number of rolling and adherent leukocytes and mast cell degranulation, a progressive increase in albumin efflux, and a decrease in the velocity of red blood cells in venules.

结果 LPS在不影响血压的情况下,显著增加了细静脉内白细胞滚动和黏附数目、血浆白蛋白漏出和肥大细胞脱颗粒率,降低了细静脉红细胞流速。

Materials and methods: Vitro model of discolored teeth: Sample of fresh erythrocytes was placed in the pulp cavity of 20 extracted teeth and centrifuged twice daily for 25 minutes over a period of 3 consecutive days.

材料与方法:在20颗因正畸拔除的健康离体牙髓腔内封入新鲜红细胞,以2500rpm/min离心25分钟,每天两次,连续离心3天制备变色牙模型。

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