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红细胞

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From ultrastructure of RBC which co-cultured with HAP|,HAP4,HAP5,we have observed that HAPi and HAP4 can induce RBC membrane to deform and decrease stability of RBC membrane, so the particles can enter intramembrane, as a result, intracellular hydroxyapatite nanoparticles may heighten the concentration of Ca2+ and effect structure of membrane protein, membrane lipid and hemoglobin, subsequently,the deformability of RBC decreased all the better and leaded to erythrocyte aggregation. However,HAP.s uniformly dispersed around cell and didn't adhere to it,also, cytomorphosis cannot be found.

观察HAP_1,HAP_4,HAP_5与细胞作用后红细胞的超微结构中,HAP_1与HAP_4引起红细胞膜变形和膜稳定性下降,粒子进入胞内,进入胞内的HAP纳米粒子可能升高胞内Ca~(2+)浓度,从而影响膜蛋白、膜脂、血红蛋白的结构,使红细胞变形性进一步下降,最终导致聚集的发生。

In this paper,the effect of ginsenosides on hemolysis in human red cells in vitro and in dog red cells in vivo was investigated.

本实验研究了人参总皂甙在体外对人红细胞及在体内对狗红细胞的溶血作用。

Methods The Hb, Hct, Plt, and Ret were examined in patients before and after hemospasia, after transfusion and before discharge, respectively.

于采血前、采血后、回输血后、出院前检测病人红细胞、血红蛋白、白细胞、血小板、网织红细胞、血细胞比容的变化。

The animal tests indicated that: the levels of plasma and hepatic LPO were elevated in the rats with hyperlipidemia and hypercholesterolemia and at the same time, the red blood cell osmotic fragility and the concentration of SGPT were also in- creased; when the α-Toc was given, the LPO levels of serum and hepar could be de- pressed, the RBCOF and the level of SGPT were reduced too, which suggessted that the membranes of RBC and hepatic cells might be damaged by LPO.

动物实验表明,具有高脂、高胆固醇血症的大鼠血清、肝组织的LPO亦增高,同时红细胞渗透脆性,SGPT也增高,给予α-Toc可以降低血清和肝组织LPO的浓度,RBCOF,SGPT也下降,提示LPO可能引起了红细胞和肝细胞膜的损害。

In the experiment, an approach was made to the cause of DRP and the mechanism of it's prevention and treatmend by acupuncture. The experimental results indicated that in rat with diabetes,(1) plasma TXB2 and the ratio of TXB2 to 6-keto-PGF1α increased obviously, suggesting hyperfunction of platelet and injury to endothelial function, which tend to result in thrombosis;(2) erythrocytic filtration index increased remarkably, suggesting erythrocytic transforming and oxygen carring hypofunction, which is one of the factors leading to retinal anoxia;(3) hyperglycemia persisted, which is related to the thickening of basilar membrane of and the decrease of mural cell of retinal blood vessels;(4) serum growth hormone rose notably, which is not only one of the important factors causing basilar membrane thickening but also promotes the synthesis or Von Willebrand Factor that participates in a tendency to thrombosis.

本实验对糖尿病性视网膜病变(Diabetic Retinopathy,DRP)的成因以及针刺防治DRP的作用机理进行了探讨,实验结果表明:糖尿病大鼠(1)血浆TXB2及TXB2/6-ket0-PGF1α比值均显著升高,提示其血小板机能亢进,内皮细胞功能损伤,具有血栓形成倾向;(2)红细胞滤过指数(Filtration Index,IF)显著增高,提示其红细胞变形能力低下,携氧运行功能降低,是造成视网膜缺氧状态的因素之一;(3)呈持续高血糖状态,与视网膜血管基底膜增厚及管壁细胞减少有关;(4)血清生长激素(Growth Hormone,GH)水平明显升高,既是导致基底膜增厚的重要因素之一,又促进VWF合成,参与血栓形成倾向。

Methods: This paper has collected 40323 normal reference values of Middle-aged Male's red blood cell count in 353 departments in China, using the method of microscope technique, and research the geographic distribution of normal reference value of Middle-aged Male's red blood cell count, the incliner 、complexity relationship characteristic between the normal reference value of Middle-aged Male's red blood cell count and geographic factors , including height above sea level , sunshine hours yearly , mean relative humidity yearly, mean temperature yearly , precipitation yearly, etc.

收集了中国353个单位用显微镜技术法测定的40323例中年男性红细胞计数正常参考值,运用BP神经网络和GIS空间分析,研究了其与海拔高度、年日照时数、年平均相对湿度、年平均气温、年降水量等地理因素间的非线性、复杂性关系特征及红细胞计数正常参考值的地理分布。

The velocity and flux of red blood cell in villus tip arteriole and capillaries were measured by FITC-labeled erythrocytes and intravital microscopy. The effect of iNOS was determined by targeted disruption of mice iNOS-gene and administration of S-methylthiourea sulfate, a selective inhibitor of iNOS, before LPS injection.

利用FITC标记红细胞和活体显微镜方法直接观察并计算小鼠小肠绒毛尖端小动脉和毛细血管内红细胞的流速和流量,并观察敲除小鼠iNOS基因和选择性iNOS抑制剂S-methylthiourea sulfate对实验过程中小肠微循环的影响。

MAIN OUTCOME MEASURES: Variables as followings were measures: blood routine examination indexes, blood sedimentation, lactic acid dehydrogenase, C reactive protein, lymphocyte proportion, reticulocyte, mean corpuscular volume, febrile, hemorrhage, arthralgia, intumescence, edema and erythra. RESULTS: A total of 92 patients (86%) developed diseases of the hematopoietic system.

主要观察指标:考察变量为血常规3项、血沉、乳酸脱氢酶、C反应蛋白、淋巴细胞比例、网织红细胞、平均红细胞体积、发热、出血、骨关节痛、肿大、水肿及皮疹等15项指标。

The numbers of RBC in low Se group and MCH in high Se group were apparently decreased.

低硒组大鼠的红细胞数和高硒组大鼠的平均红细胞血红蛋白含量显著减少。

Methods Analyzed the clinical observation and nursing from 2004 to 2008 116 examples of monge disease patients,introduced the clinical manifestation,the prevention and the remedial measure with emphasis,as well as psychology and treatment and so on nursing.

回顾性分析2004年至2008年我院116例高原性红细胞增多症患者患病期间的临床观察和护理,重点介绍高原性红细胞增多症的临床表现、预防和治疗措施,以及心理及治疗等的护理。

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