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索氏体

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Pathological study revealed compression mainly caused neuron atrophy and anterior column damage in the white matter, on the other hand, excessive cervical flexion combined with compression resulted in neuron atrophy, neuron edema, chromatolysis、vacuolization and it also caused demyelination、axon degeneration、axon necrosis in all the columns of the white matter.

病理结果显示,单纯压迫时,神经元的变化以萎缩为主,白质损害主要在前索。而在过度屈曲活动组,神经元除萎缩外,肿胀、尼氏体溶解和空泡变性等病理变化也比较常见,白质各索均可有损害。

RESULTS: The mild traction group showed angulus anterior individual neuron slightly engorged, neuron and nerve fiber morphous was normal essentially; individual substantia alba demyelinates in fragmentis; medullary sheath and auxiliary fibers of nerve root were hydropsia slightly without conspicuous myelinolysis. The moderate traction group demonstrated myelinolysis change in the spinal cord, and neurofibras lined up chaos, auxiliary fibers disaggregated, neuron dropsy, tigroid body disappeared with karyopycnosis and anachromasis in neurons; nerve root showed myelinolysis. The severe traction group displayed a great demyelination region; anterior motor neurons with karyopycnosis and anachromasis; myelinated nerve fiber of nerve root shrinked, decreased in number, with severe demyelination changes.

结果:轻度牵拉组牵拉侧前角个别神经元稍肿胀,神经元及神经纤维形态基本上正常,个别白质小片状脱髓鞘样改变,神经根髓鞘和轴索轻度水肿,脱髓鞘不明显;中度牵拉组白质有脱髓鞘改变,神经纤维排列紊乱,轴索崩解、断裂;灰质神经元水肿明显加重,前角运动神经原细胞尼氏体消失,核固缩、深染,神经根髓鞘和轴索水肿,局部轻度脱髓鞘改变;重度牵拉组牵拉侧白质为不规则的大片脱髓鞘区,前角运动神经元胞体固缩、变形、核深染,神经根有髓神经纤维萎缩、数目减少,重度脱髓鞘改变。

Somatic incompatibility test showed that the density of Armillaria ostoyae genet was lower in the northeast forest of the Changbai Mountain, and it existed in rhizomorphs which was main dissmination mode.

利用体细胞非亲和性的试验方法,鉴定出东北地区林木病原菌奥氏蜜环菌在林间的遗传体密度较低,是以菌索的形式存在,并为主要传播方式。

It shows that sorbitized treat using water quench replacing lead quench is possible for Carbon steels prestressed wires.

研究表明碳素钢予应力钢丝采用水淬火取代铅浴进行索氏体化处理是可行的。

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这一模式非常关注商人的网络信用基础。

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