糖血症

Poorly controlled gestational diabetes is associated with an increase in the incidence of preeclampsia, polyhydramnios, fetal macrosomia, birth trauma, operative delivery, and neonatal hypoglycemia.

控制不好的妊娠期糖尿病与先兆子痫，羊水过多，巨大儿，产伤，手术分娩，和新生儿低糖血症的发生率升高有关。

Poorly controlled gestational diabetes is associated with an increase in the incidence of preeclampsia,polyhydramnios,fetal macrosomia,birth trauma,operative delivery,and neonatal hypoglycemia.

控制不好的糖尿病期糖尿病与先兆子痫，羊水过多，巨大儿，产伤，手术分娩，和干细胞糖尿病足低糖血症的发生率升高有关。

Poorly controlled gestational diabetes is associated with anincrease in the incidence of preeclampsia, polyhydramnios, fetalmacrosomia, birth trauma, operative delivery, and neonatalhypoglycemia.

控制不好的妊娠期糖尿病与先兆子痫，羊水过多，巨大儿，产伤，手术分娩，和新生儿低糖血症的发生率升高有关。

The improved puberty PCOS-IR rat model has following characteristic:Retained the high androgen syndrome and/or the high LH syndrome as the endocrine characteristics;Simultaneously it had sugar fat metabolism unusual, such as the insulin resistance,the redemptive high insulin blood sickness as well as cholesterol ascension.Has the abdomen obese characteristic----In particular fat pad which around reproductive organ obvious increased;The ovary increased obviously,Ovarian cortex accumulated,the number in ovary pouch expansion graafian follicle were remarkable increased, ovulation suppression.

一、改良青春期PCOS-IR大鼠模型具有以下特征：1、保留了高雄激素血症和/或高LH血症等内分泌特征；同时有胰岛素抵抗，代偿性高胰岛素血症以及总胆固醇升高的糖脂代谢异常。2、具有腹部肥胖的特征——尤其是生殖器周围脂肪垫明显增加；3、卵巢明显增大，皮质增厚，卵巢内囊性扩张卵泡数目显著增加，排卵抑制。

AIM: To investigate the effects of insulin on aortic constriction caused by endothelin-1(ET-1) in normal and hyperglycemic rats.

目的： 研究高糖血症时胰岛素对内皮素-1（ET-1）缩血管作用的影响。

Hyperuricemia is closely related with glycometabolism, lipid metabolism and hypertension, which is a risk factor responsible for occurrence and development of diabetes, hyperlipoidemia and hypertension.

高尿酸血症与糖、脂代谢、高血压关系密切，是糖尿病、高脂血症、高血压发生发展的重要危险因素。

ConclusionHyperuricemia is closely related with glycometabolism, lipid metabolism and hypertension, which is a risk factor responsible for occurrence and development of diabetes, hyperlipoidemia and hypertension.

高尿酸血症与糖、脂代谢、高血压关系密切，是糖尿病、高脂血症、高血压发生发展的重要危险因素。

Shai pointed out that ethanol metabolism is believed to inhibit gluconeogenesis, which could increase the risk of hypoglycemia.

Shai指出，酒精代谢可以抑制糖异生，因此能够增加发生低糖血症的风险。

So hyperinsulinemia may step up blood pressure.These explain IR have some invariable effect in pathogenesis of eclampsism. Sex hormone binding globulin is a species of sterin haptoglobulin which is form from liver.The level of SHBG is an indirect index of balanate between androgen and estrogen.Not only hyperinsulinemia but also IR have intimate relationship with blood serum SHBG level.

高胰岛素血症、胰岛素抵抗与血清SHBG水平均有着密切关系，胰岛素对调节SHBG的代谢具有重要意义，胰岛素水平升高使SHBG生成减少，造成体内性激素水平紊乱，导致糖、脂肪、胰岛素代谢障碍，提示SHBG是高胰岛素血症、胰岛素抵抗的一个特有标志。

Objective To observe the effect of PGE 1 on diabetic blood hyperviscosity.Methods 59type2diaˉbetes mellitus with blood hyperviscosity were invotved in a randmoized control clinical trial.30cases were treated with PGE 1 200μg solved in500ml inj dextran lowmolecular intravenously everyday for3weeks.29cases treated with the same basic hypolycemic therapy and blood-lipid lowering drugs as the PGE 1 group except500ml inj dextran lowmolecular inˉtravenously everyday for3weeks as controls.

目的 观察列腺素E 1 治疗糖尿病高粘血症的临床效果方法 59例糖尿病合并高粘血症患者随机分组对照观察，前列腺素E 1 治疗组30例给予前列腺素E 1 注射液200μg溶于低分子右旋糖酐注射液500ml中静滴，每日1次，共3周；对照组29例降血糖、降血脂等基本治疗与前列腺素E 1 组相同，另外只给予低分子右旋糖酐注射液500ml静滴，每日1次，共3周。

Do you know, i need you to come back

你知道吗，我需要你回来

Yang yinshu、Wang xiangsheng、Li decang,The first discovery of haemaphysalis conicinna.

1〕 杨银书，王祥生，李德昌。安徽省首次发现嗜群血蜱。