糖脂

Chocolate is a mixture of roasted cocoa, cocoa butter, and very fine sugar.

巧克力是一个混合烤可可粉，可可脂，并非常细微的糖。

When you eat dark chocolate, what you are eating is cocoa liquor mixed with extra cocoa butter and some sugar.

当你吃黑巧克力时，吃进肚里的其实是可可浆、额外添加的可可脂和糖的混合物。

Cocoa paste, cocoa butter, sugar and milk are the four basic ingredients for making chocolate.

可可泥、可可脂、糖以及牛奶是制作巧克力的重要成分。

Results Three nucleoside compounds and one lignan compound were isolated from the flower buds of Panax quinquefolium L., their structure were identified as 2′-deoxyuridine, 2′-deoxythymidine,vidarabine and pinoresinol.

结果 分离得到了3个核苷类化合物和1个木脂素类化合物，分别鉴定为脱氧尿苷(2′-deoxyuridine，1)、脱氧胸苷(2′-deoxythymidine，2)、阿糖腺苷(vidarabine，3)和松脂素（pinoresinol，4）。

Observe the effects of decreasing blood sugar and kidney-enriching formula to renal structure and function of diabetic nephropathy model of rat caused by STZ, hyperlipin feed and renal hemiresection. Reveal its mechanism on the aspect of pathology, carbohydrate metabolism, renal hemodynamics and blood lipin through the contradistinctive and classified research on the medicinal constituent of the formula.

观察降糖益肾方对链脲佐菌素、饲喂高脂饲料、单侧肾切除方法诱导的糖尿病肾病大鼠模型肾脏结构与功能的影响，并就组方内滋阴益气、活血两大类药物进行对比研究，从常规、超微病理结构、糖代谢、肾脏血流动力学、血脂等方面深入探讨降糖益肾方的作用机制。

One cube of sugar and low-fat cream please.

只要一颗糖跟低脂奶精就Good。

The hepatocytes from vitamin E deficience rats were more sensitive to the toxicity of TNT, and supplement of vitamin E, treatment of desferrioxamine and dithiothreitol could prevent hepatocytes from toxicity of TNT.

明显降低TNT对肝细胞的毒性，其中尤以二硫苏糖醇的效果最为明显，这些结果从另一方面证实了，自由基反应，脂质过氧化过程和由此引起的细胞内巯基耗竭是TNT毒作用的重要环节。

Again, the high-carbohydrate diet was disasterous as it resulted in increased fasting plasma triglyceride levels and very low-density lipoprotein cholesterol levels by 24% and 23% respectively, and increased daylong plasma triglyceride, glucose, and insulin values by 10%, 12%, and 9%, respectively.

再次，高糖饮食是灾难性的，因为它导致在增加空腹血浆甘油三酯水平和极低密度脂蛋白胆固醇水平的24 ％和23 ％，以及增加daylong血浆甘油三酯，血糖，胰岛素值10 ％， 12 ％，九％，分别为。

Results: 1 The capacity of engulfment of A549 cells started enhancing at the 1h of preincubation with dexamethasone, reached the highest at the 4h of preincubation,At the 8h、24h of preincubation the capacity of engulfment evidently reduced. 2 Glucocorticoid cytosolic receptors antagonist RU38486 inhibited the improved engulfment ability by dexamethasone. 3Phagocytosing apoptotic eosinophils did not change the release of IL-6 and IL-8,but can inhibited the release of IL-6 and IL-8 from A549 cells stimulated by lipopolysaccharide. 4Increasely uptaking by dexamethasone also did not agitate the release of IL-6 and IL-8 from A549 cells. Conclusion: 1Dexamethasome improve the phagocyte capacity for apoptotic eosinophils of A549 cells by glucocorticoid cytosolic receptors, and the effect presents time-dependently. 2Phagocytosis of apoptotic eosinphils can degrade inflammatory reflection, so it is likely to be critical in the control of asthma inflammation.

结果： 1 A549细胞在地塞米松诱导后1小时吞噬能力开始升高，4小时吞噬能力达最高峰，8小时、24小时吞噬能力明显下降 2加入RU38486后抑制了地塞米松所增强的吞噬能力 3凋亡嗜酸粒细胞的吞噬并不能改变A549细胞释放IL-6和IL-8，但能抑制脂多糖刺激A549细胞后IL-6、IL-8的分泌 4地塞米松增加的吞噬也不激发A549细胞释放IL-6和IL-8 结论： 1地塞米松促进A549细胞吞噬凋亡的嗜酸粒细胞是通过A549细胞糖皮质激素胞浆受体发挥作用的，并且具有时间依赖性 2 A549细胞吞噬凋亡的嗜酸粒细胞能降低炎症反应，对哮喘炎症的控制具有重要意义

Erythrocyte membrane glycoproteins have been important factors in development of the mosaic model.

红血球膜的糖蛋白在建立类脂镶嵌模型中是一个重要因素。

The basic concept of FOP can be summarized as to further optimize effective prescription according to the standard of curative effects and with the aid of modern science and technology and theories of traditional Chinese medicine.

其基本内涵可概括为：以确有疗效的中药复方为研究对象，以现代科学技术和传统中医药理论为技术支持，以该复方所治病证的药效响应为评价标准，以优化重组疗效更优的新复方为研究目的。

Ever since our world has been a world, native forests have been indiscriminately exploited by man.

自从我们的世界一直是世界原生森林被任意剥削人。