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Objective: To observe varying aerobics interferential effects on blood glucose and blood lipids of type 2 diabetes patients in community.

目的:观察糖尿病不同有氧运动方式的护理干预对社区2型糖尿病患者体重、血糖、血脂的影响。

intramyocardial microvasculopathy on the early stage of diabetic patients may affect the flow dynamics of intramyocardial microvascular, and lead to myocardial ischemia impairments when no any coronary pathological changes exist in diabetic patients.

糖尿病早期心肌微血管的病变使心肌内冠脉血流动力学发生了变化,造成糖尿病心肌在无冠脉病变时的缺血损伤。

Methods Fasting plasma insulin,fasting plasma glucose,glycoslated hemoglobin (HbA1c),plasma lipids and lipoprotein,insulinase activity of erythrocytes and EIG were determined in 30 patients with type 2 diabetes mellitus laking chloroquine for 14 day in dosage of 250mg twice daily,27 diabetic patients laking placebo and 20 normal subjects.

随机选择服用磷酸氯喹(0.5g/d×14d)的2型糖尿病患者30例,服用安慰剂的2型糖尿病患者27例以及正常对照组20例。

There is a close relation between glucose lipide metabolism disturbance and DM complications.

结论本实验证实糖尿病时伴发的糖、脂代谢紊乱与糖尿病血管并发症的发生发展有密切关系。

The mice with alloxan-induced diabetes mellitus were orally administrated with the crude extracts and the crystal phloridzin from Lithocarpus polystachyus Rehd, and their fasting blood level as well as no-fasting level were reduced significantly, and their glucose tolerance curves also were improved.

降血糖机制研究结果表明,CA和PHL对正常和糖尿病小鼠的降血糖机制与阿卡波糖不同;CA和PHL有效降低了血清MDA水平,提高了血清SOD和GSH-Px活性,说明糖尿病小鼠体内的抗氧化能力增强。

This paper reviewed the effect of some major neurotrophic factors on diabetic neuropathy and their application in clinical treatment.

本文对一些主要的神经营养因子对糖尿病性神经病理变性及再生作用,以及神经营养因子在临床糖尿病性神经变性的应用研究作-综述。

Compared with nondiabetic patients, diabetic patients had higher rates of infectious and noninfectious complications, as well as higher lengths of stay overall and in the intensive care unit.

相比于非糖尿病患者,糖尿病患者有较高的感染和非感染的并发症,且总体住院时间增加。

Methods Diabetic rat models were prepared by giving alloxan. After administration of sodium orthovanadate for seven days, the blood glucose levels of rats were determined. The glucose tolerances of diabetic rats were evaluated by the oxidation-enzyme method, and the insulin levels were determined by radioimmunoassay.

建立四氧嘧啶诱导的糖尿病大鼠模型,对正常大鼠及糖尿病大鼠给予原钒酸钠治疗7天后,采用葡萄糖氧化酶法测定大鼠的血糖值、糖耐量,并以放射免疫法测定血清胰岛素值。

Purpose To observe the expressions of calpainⅡ in sciatic nerves of diabetic rats, and its implication in the pathogenesis of diabetic polyneuropathy.

目的 观察早期糖尿病大鼠坐骨神经calpainⅡ的动态表达,探讨其在糖尿病多发性神经病发病中的作用。

There has been increasing support for the contribution of immunologic mechanisms in the pathogenesis of diabetic neuropathy, especially in diabetic patients with severe lumbosacral radiculoplexus neuropa-thy, mononeuropathy and distal symmetric polyneuropathy.

越来越多的研究证实,免疫因素参与了糖尿病神经病变,尤其是糖尿病腰骶神经根病、神经病和远端对称性多神经病的发病过程。

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The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应,而且减少跟风的可能性。

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