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精神分裂症

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Objective To study of clinical features of family and sporadic schizophrenia.

目的探讨家族性与散发性精神分裂症的异同。

The results hereinbefore indicated that MK-801 at 0.6mg/kg could induce hyperlocomotor, stereotypy and ataxia, and was suitable for the further mechanical experiment.

以上结果表明,MK-801 0.6 mg/kg既可以诱导小鼠的运动亢进,又表现有明显的定型行为和共济失调,适合进行下面的一些神经生化学实验研究,便于深入探讨精神分裂症的发病机理。

In our previous report of stepwise fine mapping of this region, the MEGF10 gene was one of the genes showing consistent associations in our screening subsample.

他们发现,在家族基因研究和病例-对照基因研究中,MEGF10基因决定了精神分裂症的遗传风险。

Objective To explore the clinical features of different subtype of treatment-resistant schizophrenia.

目的探讨难治性精神分裂症不同亚型的临床特征。

objective to explore the clinical features of different subtype of treatment resistant schizophrenia.

目的 探讨难治性精神分裂症不同亚型的临床特征。

Objective To explore the clinical features of different subtype of treatmentresistant schizophrenia.

目的 探讨难治性精神分裂症不同亚型的临床特征。

There are many similar on symptomatology between affective disorders and schizophrenia. It may be an important factor for misdiagnosis.

结论儿童及少年期的情感障碍在症状学上与精神分裂症有许多相似之处,是导致误诊的重要原因。

But the core symptoms of schizophrenia are important symptomatology indictions for differential diagnosis between affective disorders and schizophrenia.

但儿童少年期精神分裂症存在的一些核心症状可能是两者鉴别诊断的症状学指标。

Xitelopram may be used as a synergist of Haloperidol in the treatment of schizophrenia with negative symptoms.

西肽普兰可作为氟哌啶醇治疗以阴性症状为主的精神分裂症的增效剂。

ObjectiveTo investigate the possible association of CYP2D6 gene C188T polymorphism with tardive dyskinesiain Chinese patients with schizophrenia.

目的观察广州地区汉族人群中有或无迟发性运动障碍精神分裂症患者中CYP2D6C188T基因多态性的分布,并探讨该多态与TD发生的关系。

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。

There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.

双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。