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Results: The tumor cell-platelet adhesion was significant in SACC-LM than in SACC-83; aspirin, RD, magnesium acetylsalicylate inhibitet the adhesion of SACC-LM in the final concentration of 1, 5 ,25 μ g/ml respectively.

结果:SACC-LM细胞与SACC-83细胞相比和血小板的粘附增高,阿斯匹林、阿斯匹林镁、RD在1,5,25μg/ml时抑制血小板与SACC-LM细胞的粘附,RD能抑制SACC-LM实验性肺转移的形成。

Results: The tumor cell-platelet adhesion was significant in SACC-LM than in SACC-83; aspirin, RD, magnesium acetylsalicylate inhibitet the adhesion of SACC-LM in the final concentration of 1, 5 ,25 μ g/ml respectively. RD inhibited experimental metastasis of SACC.

结果:SACC-LM细胞与SACC-83细胞相比和血小板的粘附增高,阿斯匹林、阿斯匹林镁、RD在1,5,25μg/ml时抑制血小板与SACC-LM细胞的粘附,RD能抑制SACC-LM实验性肺转移的形成。

METHODS AND RESULTS: Adherent platelets express substantial amounts of SDF-1 and recruit CD34+ cells in vitro and in vivo. A monoclonal antibody to SDF-1 or to its counterreceptor, CXCR4, inhibits stem cell adhesion on adherent platelets under high arterial shear in vitro and after carotid ligation in mice, as determined by intravital fluorescence microscopy.

方法和结果:在体和离体的粘附血小板表达大量SDF-1、募集CD34+细胞,在体荧光镜下可见,在小鼠颈动脉结扎后、离体条件下的动脉内高剪切应力时,SDF-1或其相应受体的单克隆抗体-CXCR4抑制干细胞粘附于血小板上。

First, we made a model of exhaustive exercise in rats, and used sucessfully the model in this study. We studied the effects of exhaustive exercise on adhesive behaviour of leukocytes in mesenteric venules by using an intravital microscope video system Its results indicated that the adhesiveness of circulating leukocytes to mesenteric venules enhanced after exhaustive exercise in rats.

首先,本文以游泳方法建立了大鼠模拟运动力竭模型,并成功地将此模型运用到本研究中;设计制作了肠系膜固定有机玻璃板,组成了一套活体显微观察实验系统;研究了大鼠运动力竭前后肠系膜微循环中白细胞粘附行为,结果表明:大鼠运动力竭后肠系膜微循环血流流态发生了明显的改变,大鼠运动力竭后肠系膜微循环中白细胞粘附性增强、白细胞滚动速率降低、在体白细胞变形性减小,将影响微循环正常的灌注,是导致微循环障碍发生发展的一个重要原因,进而最终表现为机体运动能力和运动质量下降。

To begin with, the adhesive experimental model between SMMC-7721 cells and HUVECs was established in this paper, then expression of integrin β〓 in SMMC-7721 cells was determined by flow cytometer, finally, adhesive mechanical properties of different cell cycle SMMC-7721 cells to collagen Ⅳ were studied by micropipette aspiration technique.

本文首先建立了肝癌细胞与内皮细胞粘附力分析的实验模型,接着应用流式细胞技术检测肝癌细胞上integrin β〓的表达,最后研究了不同细胞周期肝癌细胞与内皮细胞的粘附力学特性。

Methods: THP-1 cells were induced by oxLDL in vitro. The CD11b, CD54 expressions and adhesion to human umbilic al vein endothelial cells were measured after treatment with probucol at different concentrations by flow cytometry and β-nitrophenyl N-acetyl-β-D -glucosminide test.

分别应用流式细胞仪和β-N-乙酰氨基己糖苷酶比色法,观察不同浓度普罗布考对oxLDL诱导的THP-1对人脐静脉内皮细胞的粘附及粘附分子CD 11b和CD54表达水平的影响。

The ability of erythrocytes to adhere tumor cell was completely interdicted by monoclonal antibody of CR1 or without complement.

红细胞的免疫粘附作用属补体依赖性CR1分子介导的天然免疫粘附。

Furthermore,we observed that TH1 localized to focal adhesions and filopodia in the leading edge of cells,where TH1 reduced cell migration through affecting actin and adhesion dynamics.

此外,我们还观察到TH1定位于迁移细胞前沿的粘附斑和纤维伪足上,并且通过影响肌动蛋白和粘附动力学变化干扰了细胞的迁移。

Two types of SAM coatings, such as FDTS and OTS were applied to the micromachining, the results have revealed that the SAM coatings not only prevent released stiction but also in use stiction.

并对典型的FDTS和OTS分子膜进行了详细的讨论,研究表明,自组装分子膜不仅能有效防止释放粘附,更能防止工作粘附。

The Abnomal expression of CDlla CD123 of BMMNC and Fn IL-10 of serum can affect the bone marrow haematopoiesis and is related closely to CAA.

CAA患者粘附分子LFA-1/CD11a、Fn和细胞因子IL-10、IL-3R/CD123表达异常影响骨髓造血功能,与再障的发生密切相关,补髓生血颗粒能通过改善再障粘附分子和细胞因子的表达水平来改善CAA造血功能。

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On the other hand, the more important thing is because the urban housing is a kind of heterogeneity products.

另一方面,更重要的是由于城市住房是一种异质性产品。

Climate histogram is the fall that collects place measure calm value, cent serves as cross axle for a few equal interval, the area that the frequency that the value appears according to place is accumulated and becomes will be determined inside each interval, discharge the graph that rise with post, also be called histogram.

气候直方图是将所收集的降水量测定值,分为几个相等的区间作为横轴,并将各区间内所测定值依所出现的次数累积而成的面积,用柱子排起来的图形,也叫做柱状图。

You rap, you know we are not so good at rapping, huh?

你唱吧,你也知道我们并不那么擅长说唱,对吧?