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Study on effects of differentiation inducer on adhesion, movement and invasion of chordoma cell line in vitro

结果: 分化诱导剂HMBA诱导的CM319细胞在30 min和90 min粘附实验中的粘附率,分别下降了27%和31%。

That of Bbr was 33. 20%, that of Bi and Bl were 18. 40% and 11. 78% respectively by comparison of the adhesion of radioactively labeled Bifidobacterium strains to the mucus.

采用放射性代谢标记方法研究双歧杆菌与仔猪肠粘膜粘液糖蛋白的粘附,发现Ba的粘附能力最强,为44.61%;其次是Bbr,为33.20%;Bi和Bl分别是18.40%和11.78%。

Adhered intension of passivation film: scrub film 2 to 3 second by soft rubber, then check scratch mark on the film.

2.2钝化膜的粘附强度:检验有色钝化膜的粘附强度时,可用一块软质橡皮在镀层表面摩擦2至3秒钟,镀层膜上应无刮伤痕迹。

The experimental results show that the spectral width caused by using overall affix is 0.28 nm, and the linearity of using two-spot affix increases by 1.89%.

由实验结果得出,在压力为0~4N测量范围内,采用全粘附耦合引起的FBG反射光谱的展宽约为0.28nm;采用两点粘附耦合后,谱宽不随压力而变,而线性度较之全粘贴耦合时提高了1.89%。

Objective To study the mechanism of monocyte recruitment in atherogenesis and to clarify the effect of monocyte chemotactic protein-1 (MCP-1) in this process.

目的 了解动脉粥样硬化发病过程中单核细胞粘附并进入动脉内膜的机制,分析单核细胞趋化蛋白-1(MCP-1)基因表达对单核细胞粘附动脉内膜的作用。

Defective expression of CR1 in liver cancer is mostly acquired through central peripheral mechanisms.The changes of CR1 quantitative expression and adhering activity are consanguineously related to the severity and the activity of the development in patients with liver cancer.

结论提示肝癌患者红细胞CR1分子数量及粘附活性的变化主要是后天因素引起,肝癌患者红细胞CR1分子数量及粘附活性的变化与病情发展及严重程度密切相关。

He results showed that prestimulation of BCMEC with TNF(100 kU·L 1 ) enhanced monocytes adhesion to and migration...

NF(100kU·L-1)可促进单核细胞与BCMEC的粘附及迁移,其促粘附与迁移作用具明显的时效关系。

These critical spermatozoal functions are acquired in the epididymis where a specific luminal environment is created by the blood-epididymal barrier; proteins secreted by epididymal principal cells bind to maturing spermatozoa and regulate the maturational process of the spermatozoa.

精子功能在附睾特殊的管腔环境中获得,这一环境是由血-附睾屏障来维护。附睾主细胞分泌的蛋白与精子结合并调控精子的成熟。附睾中细胞-细胞间的相互作用通过粘附连接和紧密连接形成。

MethodsThrough enzymelinked immunosorbent Assay,the the serum sICAM1,sVCAM1 and sC5b9 levels were compared in 28 healthy pupils、26 asthmatic children (uninhaled glucocorticoids group) and asthmatic children after inhaled glucocorticoids 3,6 and 12 months.

方法采用酶联免疫吸附法,检测了200201—200412于青岛大学医学院附属医院儿科就诊的26例哮喘患儿吸入糖皮质激素治疗前及治疗后3个月、6个月、12个月后血清细胞间粘附因子1(sICAM1)、血管内皮细胞粘附因子1(sVCAM1)和补体激活片段(sC5b9)的质量浓度变化,并与28名健康儿童比较。

The study ~s conducted to elucidate the mechanism by which the expression of adhesion molecules on human endothelial cells ~s up-regulatecL Methods Human endothelial cells derived fium umbilical venisQ-IUVEC were coincubated in viny whh native human sennn albtuiiin, HSA modified v~ith advanced glycation end products or RPMI-1640 culture medium. The expression of adhesion molecule intereellular adhesion molecule-l(7ICAM-l), vascular cell adhesion molecule-l and E-selectin was determined by irnrnunofluorescen staining and flow cytometer analysis.

分离正常人脐静脉内皮细胞,将晚期糖基化终产物修饰的人血清白蛋白、人血清白蛋白以及RPMI-1640完全培养基与HUVEC在体外共同培养,用荧光单克隆抗体染色,流式细胞仪定量检测内皮细胞表面细胞间粘附分子-1(ICAM-1)、血管细胞间粘附分子-1(VCAM-1)、E-选择素的表达。

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