粘膜的

At 10 weeks, smooth muscle bundles were observed and there were no significant difference between the two groups of the number of neogenesis capillary and positive VEGFR2 expression cells. To understand the release of exogenous growth factors from small intestinal submuscosa in bladder regeneration, the release of vascular endothelial growth factor and basic fibroblast growth factor from SIS in vitro were evaluated by ELISA and MTT method.

尽管文献报道制备的小肠粘膜下层中含有少量细胞生长因子，但尚不明确其在动物体内是否仍具有生物活性，为了解小肠粘膜下层在膀胱组织修复中外源性生长因子VEGF和bFGF释放，我们用ELISA法和MTT法体外测量冻干SIS在PBS孵育液中VEGF和bFGF的含量及对上皮细胞的增殖作用；用猪小肠粘膜下层行大鼠膀胱部分修复。

Results: Symptom of allergic rhinitis, such as sneezing, and rhinorrhea were observed in sensitized groups. There were significant increases in the number of eosinophils, edema of the mucosa, proliferation of the glands.

结果：实验组均出现鼻痒、喷嚏、流清涕等变应性鼻炎的临床特征(评分 5分)，鼻粘膜和肺组织见大量的嗜酸性粒细胞浸润，粘膜水肿、腺体增生且鼻粘膜表面纤毛破坏等组织形态学变化。

Results: NKA-I cells was first found in the duodenum mucose on the 14th day of fetus, earlier than in the jejunum ( 16th day ) and ileum ( 17th day ), and its Positive reaction and the number of cells increased with further development, after birth the 30th day, it became quite identical to that of adult rat; NKA-I cells were seen in the colon mucosa on the 17th day of fetus, later on the number kept decreasing till grown up stage; No change was observed in the stomach mucosa except that few cells showed up on the 15th day of fetus; No positive cell was found on the oesphagus wall.

结果： NKA-I细胞首先见于胚胎14 d的十二指肠粘膜，早于空肠(胚胎16 d)和回肠(胚胎17 d)，其阳性反应强度及细胞量随发育而增加，30 d时与成年鼠相似；结肠粘膜内于胚胎17 d见细胞，但此后数量较少直至成年；胃粘膜于胚胎15 d见个别细胞外，以后无明显变化；食道壁始终未见阳性细胞。

Results: NKA-I cells was first found in the duodenum mucose on the 14th day of fetus, earlier than in the jejunum ( 16th day ) and ileum ( 17th day ), and its Positive reaction and the number of cells increased with further development, after birth the 30th day, it became quite identical to that of adult rat; NKA-I cells were seen in the colon mucosa on the 17th day of fetus, later on the number kept decreasing till grown up stage; No change was observed in the stomach mucosa except that few cells showed up on the 15th day of fetus; No positive cell was found on the oesphagus wall.

结果：NKA-I细胞首先见于胚胎14 d的十二指肠粘膜，早于空肠(胚胎16 d)和回肠(胚胎17 d)，其阳性反应强度及细胞数量随发育而增加，30 d时与成年鼠相似；结肠粘膜内于胚胎17 d见多个细胞，但此后数量较少直至成年；胃粘膜于胚胎15 d见个别细胞外，以后无明显变化；食道壁始终未见阳性细胞。

To minimize complications from urine reservoir constructed with an intestinal segment,reticular transplantation of bladder mucosa to an intestinal seromuscular segment was conducted in 3 randomized groups of canines.

为减少肠管膀胱术的并发症，于1994年3～7月，用犬分三组作了膀胱粘膜点状移植于浆肌层肠段重建膀胱的实验。A组：自体膀胱粘膜点状移植于浆肌层肠段形成人工膀胱，B组：在A组基础上一期将输尿管与人工膀胱吻合，C组：将同种异体膀胱粘膜移植于浆肌层肠段。

P.01 . conclusion:(1) IgA of the lamina propria mucosae would lessoning while the endotoxin in portal veinwas heightening, and the lesson of the I L-6mRNA was the main reason of the lesson of IgA in the lamina propria mucosae, so, the impairment of barrier of the intestinal mucosa was caused by the impairement of the organic immunological function descent while there was obstructive jaundice existing.

(1) 肠粘膜固有层内IgA表达减少伴随门静脉血内毒素浓度的增加，而肠粘膜内IL-6mRNA表达的减少是导致肠粘膜固有层IgA表达减少的重要原因之一，因此可推论肠粘膜免疫屏障功能的受损与梗阻性黄疸时机体的免疫功能受抑有关。

Most cases showed mucosal hyperplasia, mild crypt disarray, and variable reactive atypia. Eight cases had mucosal neuromas or other neural changes.

绝大多数病例显示阑尾粘膜增生、粘膜隐窝排列中度紊乱及程度不一的反应性不典型。8例有粘膜神经瘤或其他神经的病变。

In conclusion, our studies indicated that, by reducing the concentration of corticoid, increasing the GH concentration, dietary glutamine could maintain the N balance in the body, induce the intestinal glutamine extraction from blood, maintain the glutamine store in muscle tissue and plasma, decrease the glutamine flux from muscle tissue and protein catabolism. As the key fuel of intestinal mucosa and lymphocytes, exogenous glutamine maintained the intestinal structure, function and the CD4〓/CD8〓 ratio. In addition, glutamine normalized the SIgA containing cells'quantity and distribution through stimulating the IL-6 gene expression.

综合分析本研究的试验结果，我们认为，日粮中的外源性Gln可通过降低断奶应激后血清中皮质醇浓度，升高生长激素的浓度维持体内N的平衡，降低肠粘膜细胞从血浆中摄取的Gln，减少骨骼肌蛋白质的分解，维持骨骼肌和血浆中Gln浓度，维持淋巴细胞亚群中CD4〓/CD8〓比例；提供肠粘膜细胞能源和代谢的前体，维持肠粘膜正常的屏障结构和功能，通过刺激IL-6的表达，维持肠粘膜中SIgA分泌细胞的数量和肠道的局部免疫功能，并最终提高断奶仔猪的生产性能和抗病能力。

Different patt erns of TGFα and TGFβ1 expression were seen within the epithelium cell of rats with intestinal injury and restitution compared with untreated controls.

TGFα，TGFβ1在粘膜损伤后可能通过在粘膜上皮细胞表达部位的变化，促进细胞分化、迁移和增殖，与粘膜损伤后修复关系密切。

The two can cause ultrastructural pathogenesis widely in many organs and tissues such as hepar, pancreas, lien, ren, pulmo, cerebral, cerebellum, cor, muscular and so on, of them ,caused by i~olates YG97 and Y98 more obvious in epithelial cells of digestive tract and unobvious in epithelial cells of respiratory tract ,but ultrastructural pathogenesis caused by NDV strain F48E8 ,more obvious and more serious in glandularis and muscularis ventriaculi and in epithelial cells of respiratory tract .

将鹅源分离株YG_(97)、鸡源分离株Y_(98)和NDV强毒株F_(48)E_8感染SPF鸡，运用扫描电镜和透射电镜观察该二株病毒对机体细胞的影响，结果显示三株禽副粘病毒均引起机体肝、胰、脾、肾、肺、小脑、大脑、心和肌肉等实质器官组织细胞的超微病变，其中YG_(97)和Y_(98)引起消化道粘膜上皮细胞发生微绒毛脱落、纤毛脱落、分泌颗粒增多、细胞游离面破溃等显著的超微病变，引起气管粘膜上皮细胞的不显著超微病变；F_(48)E_8强毒株引起胃粘膜显著而严重的超微病变；还引起气管粘膜上皮严重的超微病变。

On the other hand, the more important thing is because the urban housing is a kind of heterogeneity products.

另一方面，更重要的是由于城市住房是一种异质性产品。

Climate histogram is the fall that collects place measure calm value, cent serves as cross axle for a few equal interval, the area that the frequency that the value appears according to place is accumulated and becomes will be determined inside each interval, discharge the graph that rise with post, also be called histogram.

气候直方图是将所收集的降水量测定值，分为几个相等的区间作为横轴，并将各区间内所测定值依所出现的次数累积而成的面积，用柱子排起来的图形，也叫做柱状图。