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Neither nail change nor mucosal involvement was noted. The biopsy specimen taken from the thigh showed characteristic acantholysis and dyskeratosis. The clinical and histological differential diagnoses are presented and discussed.

患者无家族史,亦无指甲与粘膜病变,大腿皮肤病灶切片,病理显示典型棘层分解与角化不全,关於临床及病理下类似的鉴别诊断,将在本文中加以讨论。

In hydrogen peroxide group, 3% hydrogen peroxide、anti-virus and acesodyne were used, while in combined therapy group, additional 2% Lidocaine was used. Then the cure time and the effective rate were observed.

过氧化氢组采用3% H2O2加口服抗病毒、止痛药,而综合组在对照组的基础上加用2%利多卡因湿敷口内粘膜,观察各组的治愈时间和有效治愈率。

This product even to used for diminish inflammation of perineum, used for acesodyne and restored the trauma of mucosa, sprayed of the underpants or sanitary towel in the using period can be taking a best effect.

本品还可用于会阴的消炎、止痛和修复受损粘膜,在使用期间同时喷洒于内裤或卫生巾上效果更佳。

However, there is no difference for NAT2 rapid and slow acetylator between two groups.

吃红烧鱼因素在NAT2的快速酶中,特别是在野生纯合型中直肠腺瘤复发的相对危险度有增高趋势,提示了NAT2〓4等位基因编码快酶型NAT,可能在红烧鱼产生的杂环胺类化合物中代谢中加速其N-乙酰基化代谢过程,从而加速其代谢为终致癌物而与肠粘膜上皮细胞的DNA产生加成物,损伤其遗传物质。

Nevertheless, Smoking could interact with wild type of NAT2 and increase the relative risk of rectal adenoma recurrence significantly. It is suggested that NAT2 might involve the metabolism of pre-carcinogen generating from tobacco and alter the susceptibility of cancer. Meanwhile, the NAT2〓4 alleles encode rapid acetylator of NAT2 would increase the risk of adenoma recurrence among those high-risk people with habit of fried fish intake.

吃红烧鱼因素在NAT2的快速酶中,特别是在野生纯合型中直肠腺瘤复发的相对危险度有增高趋势,提示了NAT2〓4等位基因编码快酶型NAT,可能在红烧鱼产生的杂环胺类化合物中代谢中加速其N-乙酰基化代谢过程,从而加速其代谢为终致癌物而与肠粘膜上皮细胞的DNA产生加成物,损伤其遗传物质。

A portion of ductal cells and individual serous acinic cells of the minor salivary gland also expressed Fas not only in membranous but also in cytoplasmic pattern.

结果发现,在正常鼻咽粘膜表面及隐窝的假复层纤毛柱状上皮细胞胞膜、部分小涎腺的导管上皮和个别浆液性腺泡上皮细胞的胞膜和胞浆Fas阳性表达;鳞状化生上皮的表层细胞胞膜有Fas表达,但基底细胞未见Fas表达;浸润的淋巴细胞也不表达Fas.45.9%(17/37)的鼻咽非角化性癌Fas阳性,但只有不足10%的癌细胞胞膜微弱表达Fas。

Previous studies have revealed that aganglionic or morphological and numerical abnormalities of the intestinal intramural ganglia may contribute to the pathogenesis of the intestine motility obstacle of them.

目前认为HD和HAD肠动力障碍的发病机制主要是病变肠段粘膜下神经丛和肌间神经丛的神经节细胞的缺失和减少,病变肠壁内的副交感、交感及第三类神经系统均有改变。

It was original report to find the developmental delay of the ENS, absence of submucosal plexus and the aganglionic segment of colon ending in trisomy 16 mice, an animal model for Down syndrome.

Down综合征的动物模型常染色体16三体胎鼠ENS发育迟缓,粘膜下神经丛发育障碍出现后肠末端无神经节区。

Incidence Department patch albuginea surrounding mucosal hyperemia.

发病处有斑片白膜,周围粘膜充血。

FLD at high dose appears to be better than cimetidine for hydrochloric acid- induced model and ab-solute ethvl alco- hol- induced model.

茯苓饮500,1000,2000mg/Kg灌胃,对大鼠胃粘膜损伤有良好的保护作用,其中对盐酸和无水乙醇模型,茯苓饮高剂量优于cimetidine(P<0.05,或P<0.01)。

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