粘细胞

The mechanism probably is that Pishang can inhibit the growth, movement and adheresion to extracellular matrix and endothelial cell of liver tumor cells, up-regulate the expression of nm23, down-regulate the expression of CD44, MMP-2, and inhibit the tumor neovascular by supressing the vascular endothelial cell, down-regulating the expression of VEGF, damaging the primitive mesenchymal cells and inhibiting neovascular gemmation.

砒霜具有明显的抗肝癌细胞作用，并有一定的抗侵袭与转移作用，其机制可能与其能抑制肝癌细胞增殖、迁移、阻止肝癌细胞与细胞外基质和内皮细胞粘附、上调肿瘤转移抑制基因nm23表达、下调肿瘤侵袭分子CD44、MMP-2表达以及通过抑制内皮细胞增殖、下调VEGF表达、损伤间充质细胞、阻止新生血管芽生等途径从而抑制肿瘤新生血管形成有关。

In order to investigate the theory and method in researching mechanical properties of hepatocellullar carcinoma cells with micropipette aspiration , hepatocellullar carcinoma cells.

细胞的力学特性与细胞的结构和功能密切相关，细胞粘弹性是细胞的一种本质属性，也是细胞力学特性研究的重要内容，微吸管法是一种在单个细胞水平上研究细胞力学特性的先进手段。

In this study,we firstly described anovel form of chemoresistance,which we termed sudden drug resistance,in apoptosis of human leukeamic U937cells.

进一步的研究表明这种快速耐药的发生与放线菌酮本身诱导的细胞粘附，尤其是细胞间粘附有关。

Among all adhesion molecules sereted by endotheliocyte, intercellular adhesion molecule-1(ICAM-1) and vascular cell adhesion molecule-1(VCAM-1) have considerable effect on the occurrence and development of diabetic retinopathy.

在内皮细胞分泌的粘附分子中，细胞间粘附分子-1(intercellular adhesion molecule -1，ICAM-1)对糖尿病视网膜病变的发生和发展有着重要的作用。

The results shows that the tapetal expanding abnormally occurs in dyadphase stage, microspore abortion occurs in the early of monokaryon stage, disaggregation in the end of monokaryon stage, only some spall of microspore is observed in monokaryon stage, the four cell walls also exist, the tapetal cells became completely vacuolated, almost filled the whole locule. vascular bundle of male sterile line anther vanish at mature pollen stage.

结果发现花药绒粘层在二分体时期发生异常，小孢子在单核前期开始败育，至单核后期完全解体，花药发育后期不育系药腔中只观察到少量的小孢子残体，花药壁的四层细胞依然存在，绒粘层细胞已经彻底液泡化，占据了药腔的整个空间，花粉成熟期不育系花药维管束消失。

MethodsThrough enzymelinked immunosorbent Assay,the the serum sICAM1,sVCAM1 and sC5b9 levels were compared in 28 healthy pupils、26 asthmatic children (uninhaled glucocorticoids group) and asthmatic children after inhaled glucocorticoids 3,6 and 12 months.

方法采用酶联免疫吸附法，检测了200201—200412于青岛大学医学院附属医院儿科就诊的26例哮喘患儿吸入糖皮质激素治疗前及治疗后3个月、6个月、12个月后血清细胞间粘附因子1(sICAM1)、血管内皮细胞粘附因子1(sVCAM1)和补体激活片段(sC5b9)的质量浓度变化，并与28名健康儿童比较。

Incubated with cytochalisin B for 4 hours, F-actin bunddles disrupted, confluent ECs monolayer has a complete detachment from the coverslips. So, there is a suggestion that the F-actin in ECs is essential for endothelial adherence under shear stress.

Northcrn杂交结果表明外源性纤维连接蛋白有促进EC肌动蛋白表达的作用，结合F-actin染色及FN受体染色结果，本文认为用纤维连接蛋白作为细胞种植的促粘附物质有助于内皮细胞粘附结构的形成。

Furthermore,we observed that TH1 localized to focal adhesions and filopodia in the leading edge of cells,where TH1 reduced cell migration through affecting actin and adhesion dynamics.

此外，我们还观察到TH1定位于迁移细胞前沿的粘附斑和纤维伪足上，并且通过影响肌动蛋白和粘附动力学变化干扰了细胞的迁移。

In cell adhesion experiments, adhesion of monocytes on the monolayers of PMVECs was examined.

通过粘附实验，观察单核细胞在内皮细胞单层上的粘附。

The Abnomal expression of CDlla CD123 of BMMNC and Fn IL-10 of serum can affect the bone marrow haematopoiesis and is related closely to CAA.

CAA患者粘附分子LFA-1/CD11a、Fn和细胞因子IL-10、IL-3R/CD123表达异常影响骨髓造血功能，与再障的发生密切相关，补髓生血颗粒能通过改善再障粘附分子和细胞因子的表达水平来改善CAA造血功能。

Plunder melds and run with this jewel!

掠夺melds和运行与此宝石！

My dream is to be a crazy growing tree and extend at the edge between the city and the forest.

此刻，也许正是在通往天国的路上，我体验着这白色的晕旋。