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Conclu sion:(1)Surface densities of PCNApositive cells reached their peaks on 14th day after birth,but the number of apoptotic cells reached its peaks on 21st day p ostnatal,so the apoptotic peak was late the proliferating peak for a week,and related to the particular period in cycle of seminiferous epithelium;(2)The spe rmatogium were found move from center to the periphery of the seminiferous tubule and arrange disorder to order;(3)The spermatogonium remained replicating activ ity of DNA during the developing stage.

生精细胞凋亡的高峰要滞后其增殖峰1周左右,而且与其所处生精周期的特定阶段有关;(2)精原细胞在胚胎发育过程中向曲细精管周边迁移,其排列由无序到有序;(3)在生后各发育阶段中,精原细胞始终保持DNA复制的能力。

While under the ultrastructure, the spermatogonium can be distinguished as primary spermatogonium and secondery spermatogonium; in addition, the mature sperm has a head, a midpeice and a tail, and the "9+3" canaliculus tail has side fin in it.

超微结构下精原细胞分为初级精原细胞和次级精原细胞,成熟精子分头部、中片和尾部3个部分,尾部有侧鳍,具"9+3"式微管结构。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞,肾小管上皮细胞和血管内皮细胞表达均阴性。

In the computer vision analysis system of wood across compression,early wood cell,latewood cell,wood ray cell and resin canal cell were recognized and classified automatically byusing neural network technology,applying the function of network recognition and the learningfunction of self-organizing and self-adapting,and wood tectonics feature were taken down byinstructed-learning or uninstructed-learning.

在木材横纹压缩计算机视觉分析系统中,采用了人工神经元网络技术,并运用人工神经元网络的网络识别功能和自组织、自适应的学习功能,通过有导师或无导师学习来记忆所学习过的木材构造学特征,实现了对针叶材管胞中的早晚材细胞、木射线细胞和树脂道细胞进行自动地识别与分类。

The reason Manshen NO.1 prescription can restrain accrementition in intercapillary cells and mesangial region may be due to its adjustment of NO and ET-1's contents in vivo. It is worth of doing further researchs in the future.

1、慢肾1号方对系膜增生性肾炎有良好的治疗作用,改善症状,抑制系膜细胞及基质区的增生,从而阻止肾小管硬化的发展,防止肾功能衰竭的发生,2、肾慢1号方能够提高模型组小鼠血清NO的含量,降低ET-1的含量,使之恢复正常。3、慢肾1号方可能是通过调节体内细胞因子(NO及ET-1)的含量,从而达到抑制系膜细胞及基质增生的目的,值得深入研究。

The results showed as followed:low level NO could keep Sertoli Cells' microtubule and cell viability normally,and high level NO could increase the activity of p-38MAPK of cells, decrease antioxygen ability and cell secretion and destroy the structure and distribution of microtubule.

结果表明,低浓度NO能维持支持细胞正常的骨架和活力,而高浓度NO能提高SC内p38MAPK的活性,降低抗氧化酶的活性和破坏细胞内微管的结构和分布,甚至细胞的分泌功能。

The C3A spheroids displayed multilayer cell morphology, characterized by a large number of tight junctions, polar cells, and bile canaliculi, similar to spheroids of primary hepatocytes.

这个C3A球形体显示出多层细胞的形态学特征,包括有大量的紧密连接、极性细胞及胆小管,这与原代肝细胞球形体相似。

Methods To use Ce3+ or Pb2+ as capture agents to detect carbonic anhydrase, Na+-K+-ATPase, Mg2+-ATPase, 5' –nucleotidase and NADPH oxidase activities on endosome membrane in epithelial cells of renal proximal tubules、hepatic cell of rat and human leukocytes. with cytochemical method.

用细胞化学方法,以Ce3+或Pb2+作捕捉剂,分别检测碳酸酐酶,Mg2+-ATP酶,Na+- K+-ATP酶,5'-核等酸酶,NADPH oxidase在大鼠肾近曲小管上皮细胞、肝细胞以及人体嗜中性白细胞内吞体膜上的酶活性。

There were a few light stain MCP-1 positive cells in right cerebral ependyma at 2h ischemic.

缺血Zh 组在右侧脑室管膜区可见少量低表达MCP-1 阳性细胞;4h 组在海马区也可见散在的低染色的阳性细胞;12h、24h、48h和 72h各组均可见MCP-1 免疫阳性细胞。

Results The main pathologic changes of high dosage group were inflammatory cell infiltration into the tissues around injecting location, subarachnoid space, and ependyma.

高剂量组动物脑组织主要病理改变为给药部位周围脑组织、蛛网膜下腔、室管膜的炎细胞浸润,炎细胞主要以胶质细胞、单核、淋巴细胞为主,没有脑组织的变性坏死。

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