端细胞

The in vivo adjuvant effect of PEI for DNA vaccines was examined in EL4/EG7-OVA syngeneic mouse lymphoma model.

在动物实验中，我们於小鼠脚掌经皮下/皮间的途径接种疫苗，发现接种PEI/DNA复合物疫苗的小鼠，其接种位置的树突状细胞会呈现OVA抗原并移行到近端淋巴结。

Under the same conditions pET28a-△SIL-2 expression was higher. RNA structure software analysis showed that mRNA5'end sequences of pET28a-IL-2 formed a complex and relatively stable secondary structure, which testified that the existence of the signal peptide sequence had influence on the ChIL-2 gene in prokaryon expression.

RNAstructure软件分析表明，pET28a-IL-2的mRNA5'端序列形成了复杂且较为稳定的二级结构，不利于翻译的起始，证明信号肽序列的存在对ChIL-2基因在原核细胞中的表达有一定的影响。

Oncolysis and suppression of tumor growth by a GFPexpressing oncolytic adenovirus controlled by an hTERT and CMV hybrid promoter［J］.

人端粒酶催化亚单位启动子驱动EGFP真核表达载体的构建及在肺癌细胞的表达［J］。

The stably transfected cells were selected by neomycin and expanded life span which were designated hTERT-MSCs was observed.

检测转染前后hTERT在mRNA水平的表达，端粒酶活性的变化，hTERT转入后对细胞增殖的影响。

Morphology and cytohistological zonation appeared different between cone apex and the vegetative shoot apex.

雌球果原基与营养茎端在外部形态及内部细胞组织学分区结构有明显差异。

They focused on telomeres, areas of genetic material on the ends of a cell's chromosomes.

他们侧重于端粒，不同地区的遗传物质，对两端的细胞的染色体。

Dr. Kaszubowska at the University of Gdansk, Poland, who showed that centenarians have long telomeres in their cells.

波兰格丹斯克大学的Kaszubowska博士证实，百龄老人体内细胞的端粒都较长。

To investigate the function of Nogo-A in neurons, human embryonic kidney cell line 293FT was used as a model, along with the pathway profiling reporter system to check the involvement of Nogo-A in regulating the intracellular signaling pathway. It was found that over-expression of Nogo-A could activate NF-κB signaling specifically, then followed by using different Nogo-A alternative splicing isoforms and a serial of truncations, it was confirmed that the activation of NF-κB by Nogo-A relied on its N-terminal proline rich domain. Furthermore, by co-expressing the dominant negative mutants of some NF-κB pathway associated proteins, it revealed that the IκBα,TRAF6, Rac/Cdc42 were involved in Nogo-A inducing activation of NF-κB.

为探讨神经元内Nogo-A的功能，以HEK293FT细胞为模型，利用信号途径报告基因系统筛选过表达Nogo-A对多种信号途径的调控作用，发现过表达Nogo-A能特异激活NF-κB信号，利用不同的Nogo-A剪接体和截断体形式研究，证明Nogo-A激活NF-κB信号依赖于其氨基端的proline rich结构域，进一步使用NF-κB信号途径相关分子显性突变体揭示IκBα、TRAF6、 Rac/Cdc42 参与Nogo-A激活NF-κB信号。

To investigate the function of Nogo-A in neurons, human embryonic kidney cell line 293FT was used as a model, along with the pathway profiling reporter system to check the involvement of Nogo-A in regulating the intracellular signaling pathway. It was found that over-expression of Nogo-A could activate NF-KB signaling specifically, then followed by using different Nogo-A alternative splicing isoforms and a serial of truncations, it was confirmed that the activation of NF-kB by Nogo-A relied on its N-terminal proline rich domain. Furthermore, by co-expressing the dominant negative mutants of some NF-kB pathway associated proteins, it revealed that the IkBα,TRAF6, Rac/Cdc42 were involved in Nogo-A inducing activation of NF-kB.

为探讨神经元内Nogo-A的功能，以HEK293FT细胞为模型，利用信号途径报告基因系统筛选过表达Nogo-A对多种信号途径的调控作用，发现过表达Nogo-A能特异激活NF-kB信号，利用不同的Nogo-A剪接体和截断体形式研究，证明Nogo-A激活NF-kB信号依赖于其氨基端的proline rich结构域，进一步使用NF-KB信号途径相关分子显性突变体揭示IkBα、TRAF6、Rac/Cdc42参与Nogo-A激活NF-kB信号。

According to the clear water experiment, aeration performance of the new equipment is good with high total oxygen transfer coefficient and oxygen utilization ratio.

曝气设备的动力效率在叶轮转速为120rpm～150rpm时取得最大值，此时氧利用率和充氧能力也具有较高值。

The environmental stability of that world - including its crushing pressures and icy darkness - means that some of its most famous inhabitants have survived for eons as evolutionary throwbacks, their bodies undergoing little change.

稳定的海底环境─包括能把人压扁的压力和冰冷的黑暗─意谓海底某些最知名的栖居生物已以演化返祖的样态活了万世，形体几无变化。