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Occasionally an unmyelinated fiber bundle contained the same kind of substance.

轴—树突触是本文中各种免疫反应纤维终末所形成突触的主要形式。

The quantitative result of EM showed also that the numbers of unmyelinated fibers in spared root of morphine spared root and spared root rats were respectively 60% and 42% higher than that of unoperated side.

提示参与Ⅱ板层突触重建的部分突触前成分是来自备用根内无髓纤维的侧支出芽。6、切除一侧腰段大部分背根节后,对侧Ⅱ板层外侧份FRAP活性降低或消失,即鱼钩尖消失。

Especially, the number of type Ⅰ complex terminals which come from unmyelinated afferent fibers recovered to the level of unoperated side. It indicates that morphine may promote the collateral sprouting and synaptic reinnervation of primary afferent fiber, unmyelinated afferent in particular. 4. Although the collateral sprouting and synaptic reinnervation were shown also in spinal cord of operated side in 34 days spared root rats, they were slower than that of morphine spared root group.

表明吗啡促进了备用根初级传入纤维,特别是无髓纤维更快地向L〓脊髓段侧支出芽,稍后在L〓平面脊髓内侧支出芽以及突触重建。4、34天备用根大鼠术侧脊髓内也有侧支出芽和突触重建,不过侧支出芽的速度要比吗啡组慢。5、电镜定量结果也显示,吗啡备用根大鼠和备用根大鼠术侧备用根的无髓纤维数比备用根大鼠非手术侧的分别增多60%和42%。

Radiolabeled pharmacological binding assay showed: 1 BmK AS bound specifically to a single class of non-interacting binding sites on both rat brain and cockroach nerve cord synaptosomes with high affinity and low low capacity. The association of BmK AS with both synaptosomes was quick to reach equilibrium, but the dissociation seemed to be only partial. The specific binding of BmK AS was independent of membrane potential, and couldn't be modified by veratridine. Moreover, the binding of BmK AS could be inhibited by native BmK AS, BmK AS1, BmK IT2 and BmK IT on both synaptosomes, but not or partially inhibited by BmK Ⅰ and suggesting BmK AS type neurotoxins might bind to a distinct receptor site on mammal and insect sodium channels with a similar manner from β-scorpion toxins.

二。蝎毒素多肽配体与哺乳动物和昆虫靶离子通道受体的药理结合特性及其靶结合位点的研究:经放射性标记结合和竞争抑制实验表明:1)BmK AS在大鼠脑和蟑螂神经索突触体上有单一非协同性高亲和力低结合容量结合位点,能快速结合并达到平衡,但仅能部分解离,其结合不依赖于膜电位,也不受藜芦碱调节,天然的BmK AS,BmK AS-1,BmK IT2和BmK IT可抑制〓AS与两种突触体的特异结合,而BmK Ⅰ无或仅有部分抑制效应。

Results Denatured necrosis occurred on the surface of hypothalamic periosteum with no cells and there was a large amount of starchy bodies and hydrops in foci with angiectasis of choroid plexus and necrosis and calcification in tunicaintima of vesicles,and mitochondria in the nerve synapse disappeared and part of anteroposterior membranes ...

结果 慢性SZ患者下丘脑部室管膜表面变性坏死、无细胞,病灶中有大量淀粉样小体和水肿,脉络丛血管扩张,管壁内膜有坏死和钙化;神经突触内大部分突触小泡和线粒体消失,部分前后膜模糊不清。

PubMed abstract: To examine the relationship between synaptic plasticity and learning and memory as directly as possible, we have developed a new simplified preparation for studying the siphon-withdrawal reflex of Aplysia in which it is relatively easy to record synaptic connections between individual identified neurons during simple forms of learning.

PubMed摘要:为了尽量直接地研究突触可塑性与学习和记忆的联系,我们做了新的一个简化制备,目的是研究海兔缩虹吸反射,其中较容易在学习的简单形式中,记录个体确定之神经元的突触联系。

The fEPSP response evoked in one pathway was small, usually 20–30% of the maximu, whereas a large response, usually 80–90% of the maximum, was evoked in the strong pathway. After recording of the baseline fEPSP evoked at 0.033 Hz, LTP of the weak pathway could be associatively induced by paired stimulation of the weak and strong pathways 100 times at 6 sec intervals, with stimulation of the weak pathway preceded 3–10 msec.

在记录以0.033Hz所引发基础的兴奋性突触后电位之后,在弱的路径上可以因配合弱与强的刺激路径而引发出相关性长期增益(每六秒一次共100次,弱的刺激路径在强的刺激路径之前 3-10 ms),然而当在两个路径之间的配合时间差大於10 ms时即无法形成相关性长期增益,此类型长期增益的引发是需要依赖活化NMDA接受器的,因为给予50 mM的D,L-AP5可以阻断相关性长期增益的引发(兴奋性突触后电位为原来的105±6﹪;n=6)。

Objective To find out the changes of synaptic density in the periaqueductalgray of the naive and kindled audiogenic seizures-prone rats.

突触素P38参与Ca2+-依赖性的神经递质释放过程,并且与其他囊泡蛋白共同参与短时程及长时程突触可塑性的调节[2]。

The in vitro effects of Gln49-PLA_2 were examined using the chick biventer cervicis nerve muscle preparation. Gln49-PLA_2 caused a dosedependent blockade of neuromuscular transmission. The time required to cause 50% blockade was about 150 min at the highest concentration 100μg/ml. This inhibition appeared to be presynaptic in origin as evidenced by the lack of effect of toxin on responses to exogenous acetylcholine in the non-stimulated BCp.

Gln49-PLA_2能够剂量依赖性地抑制颈二腹肌神经信号正常传导并影响肌肉收缩,在100μg/ml剂量下,肌肉收缩幅度在150分钟后达到50%的抑制率;同时,随着Gln49-PLA_2浓度的降低,肌肉收缩幅度的抑制效率也逐渐降低;实验证明Gln49-PLA_2对突触后膜乙酰胆碱受体敏感性无影响,对神经肌肉信号传导收缩的抑制作用位点在突触前膜。

With the advantage of being highly objective and efficient,the computer-aided analysis system for synaptic numerical density based on the Disector technique,Disector Countor 1.0,would be the developmental direction for synaptic numerical density analysis.

我们利用本文探讨应用Disector技术测量突触数密度的可行性和具体步骤,以及基于该技术的突触数密度计算机辅助分析的原理和过程。

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